PED/PEA-15 controls fibroblast motility and wound closure by ERK1/2-dependent mechanisms. - Wikidata - awgldk - TriplyDB

PED/PEA-15 controls fibroblast motility and wound closure by ERK1/2-dependent mechanisms.

PED/PEA-15 controls fibroblast motility and wound closure by ERK1/2-dependent mechanisms.

http://www.wikidata.org/entity/Q42272320

about

On the Quest of Cellular Functions of PEA-15 and the Therapeutic Opportunities A novel chemotaxis assay in 3-D collagen gels by time-lapse microscopy Hyaluronidase modulates inflammatory response and accelerates the cutaneous wound healing The Wound Healing and Antibacterial Activity of Five Ethnomedical Calophyllum inophyllum Oils: An Alternative Therapeutic Strategy to Treat Infected Wounds Extracellular matrix proteins and displacement of cultured fibroblasts from duodenal biopsies in celiac patients and controls NGF accelerates cutaneous wound healing by promoting the migration of dermal fibroblasts via the PI3K/Akt-Rac1-JNK and ERK pathways.Phosphoprotein enriched in astrocytes (PEA)-15: a potential therapeutic target in multiple disease states.Adipose microenvironment promotes triple negative breast cancer cell invasiveness and dissemination by producing CCL5.Inhibition of miR-155 Protects Against LPS-induced Cardiac Dysfunction and Apoptosis in Mice.How calcite and modified hydroxyapatite influence physicochemical properties and cytocompatibility of alpha-TCP based bone cements.

P2860

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P2860

PED/PEA-15 controls fibroblast motility and wound closure by ERK1/2-dependent mechanisms.

http://www.wikidata.org/entity/Q42272320

description

2012 nî lūn-bûn

@nan

2012年の論文

@ja

2012年論文

@yue

2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

@zh-mo

2012年論文

@zh-tw

2012年论文

@wuu

2012年论文

@zh

2012年论文

@zh-cn

name

PED/PEA-15 controls fibroblast motility and wound closure by ERK1/2-dependent mechanisms.

@en

PED/PEA-15 controls fibroblast motility and wound closure by ERK1/2-dependent mechanisms.

@nl

type

label

PED/PEA-15 controls fibroblast motility and wound closure by ERK1/2-dependent mechanisms.

@en

PED/PEA-15 controls fibroblast motility and wound closure by ERK1/2-dependent mechanisms.

@nl

prefLabel

PED/PEA-15 controls fibroblast motility and wound closure by ERK1/2-dependent mechanisms.

@en

PED/PEA-15 controls fibroblast motility and wound closure by ERK1/2-dependent mechanisms.

@nl

P1433

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P1433

Journal of Cellular Physiology

P1476

PED/PEA-15 controls fibroblast motility and wound closure by ERK1/2-dependent mechanisms.

@en

P2093

Angela Cassese

http://www.w3.org/2001/XMLSchema#string

Claudia Miele

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Corrado Garbi

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Ferdinando Giacco

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Francesco Beguinot

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Francesco Oriente

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Giuseppe Perruolo

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Roberta Buonomo

http://www.w3.org/2001/XMLSchema#string

Sergio Caserta

http://www.w3.org/2001/XMLSchema#string

Valentina Pagliara

http://www.w3.org/2001/XMLSchema#string

P2860

PED/PEA-15 gene controls glucose transport and is overexpressed in type 2 diabetes mellitus.

On the Theory of the Brownian Motion

Cell migration: integrating signals from front to back

Regulation of expression of phospholipase D1 and D2 by PEA-15, a novel protein that interacts with them

Knock-out of the neural death effector domain protein PEA-15 demonstrates that its expression protects astrocytes from TNFalpha-induced apoptosis

RSK2 activity is regulated by its interaction with PEA-15

Peripheral arterial disease in people with diabetes

PEA-15 mediates cytoplasmic sequestration of ERK MAP kinase

Multiple members of the mitogen-activated protein kinase family are necessary for PED/PEA-15 anti-apoptotic function

PEA-15 is inhibited by adenovirus E1A and plays a role in ERK nuclear export and Ras-induced senescence

P304

2106-2116

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P31

scholarly article

P356

10.1002/JCP.22944

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P433

5

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P478

227

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P50

Pietro Formisano

Gelsomina Mansueto

Angela Vasaturo

P577

2012-05-01T00:00:00Z

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P5875

51512526

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21780113

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P932

3306794

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