Depletion of hematogenous macrophages promotes partial hindlimb recovery and neuroanatomical repair after experimental spinal cord injury.
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Characterization of early and terminal complement proteins associated with polymorphonuclear leukocytes in vitro and in vivo after spinal cord injuryChondroitin sulfate proteoglycans in the nervous system: inhibitors to repairQuantitative analysis of cellular inflammation after traumatic spinal cord injury: evidence for a multiphasic inflammatory response in the acute to chronic environmentDeficiency in complement C1q improves histological and functional locomotor outcome after spinal cord injuryMacrophages promote axon regeneration with concurrent neurotoxicityCytokine and Growth Factor Activation In Vivo and In Vitro after Spinal Cord InjuryInduced Pluripotent Stem Cell Therapies for Cervical Spinal Cord InjuryNeurotrauma and inflammation: CNS and PNS responsesA novel antagonist of p75NTR reduces peripheral expansion and CNS trafficking of pro-inflammatory monocytes and spares function after traumatic brain injury.Azithromycin drives alternative macrophage activation and improves recovery and tissue sparing in contusion spinal cord injuryA novel inhibitor of p75-neurotrophin receptor improves functional outcomes in two models of traumatic brain injury.Acute leptin treatment enhances functional recovery after spinal cord injuryIntravenous multipotent adult progenitor cell treatment decreases inflammation leading to functional recovery following spinal cord injury.Infiltrating blood-derived macrophages are vital cells playing an anti-inflammatory role in recovery from spinal cord injury in miceHematogenous macrophage depletion reduces the fibrotic scar and increases axonal growth after spinal cord injuryThe glial scar-monocyte interplay: a pivotal resolution phase in spinal cord repairArylsulfatase B improves locomotor function after mouse spinal cord injuryDerivation of multivariate syndromic outcome metrics for consistent testing across multiple models of cervical spinal cord injury in ratsThe LTB4-BLT1 axis mediates neutrophil infiltration and secondary injury in experimental spinal cord injury.CD47 knockout mice exhibit improved recovery from spinal cord injury.Prevention of both neutrophil and monocyte recruitment promotes recovery after spinal cord injury.Role of CCL2 (MCP-1) in traumatic brain injury (TBI): evidence from severe TBI patients and CCL2-/- miceLiposomal clodronate selectively eliminates microglia from primary astrocyte culturesReciprocal modulation between microglia and astrocyte in reactive gliosis following the CNS injuryBlockade of P2 nucleotide receptors after spinal cord injury reduced the gliotic response and spared tissueImatinib enhances functional outcome after spinal cord injuryInhibition of EGFR/MAPK signaling reduces microglial inflammatory response and the associated secondary damage in rats after spinal cord injuryActivated microglia inhibit axonal growth through RGMaTwo faces of chondroitin sulfate proteoglycan in spinal cord repair: a role in microglia/macrophage activationIdentification of two distinct macrophage subsets with divergent effects causing either neurotoxicity or regeneration in the injured mouse spinal cordDealing with Danger in the CNS: The Response of the Immune System to Injury.Chronic mild stress eliminates the neuroprotective effect of Copaxone after CNS injury.Intravital imaging of axonal interactions with microglia and macrophages in a mouse dorsal column crush injuryAnother barrier to regeneration in the CNS: activated macrophages induce extensive retraction of dystrophic axons through direct physical interactions.B cells produce pathogenic antibodies and impair recovery after spinal cord injury in mice.Glycan-dependent binding of galectin-1 to neuropilin-1 promotes axonal regeneration after spinal cord injury.Monocyte recruitment and myelin removal are delayed following spinal cord injury in mice with CCR2 chemokine receptor deletion.Characterization and modeling of monocyte-derived macrophages after spinal cord injury.Low-level laser facilitates alternatively activated macrophage/microglia polarization and promotes functional recovery after crush spinal cord injury in rats.Comparison of immunopathology and locomotor recovery in C57BL/6, BUB/BnJ, and NOD-SCID mice after contusion spinal cord injury
P2860
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P2860
Depletion of hematogenous macrophages promotes partial hindlimb recovery and neuroanatomical repair after experimental spinal cord injury.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Depletion of hematogenous macr ...... perimental spinal cord injury.
@en
Depletion of hematogenous macr ...... perimental spinal cord injury.
@nl
type
label
Depletion of hematogenous macr ...... perimental spinal cord injury.
@en
Depletion of hematogenous macr ...... perimental spinal cord injury.
@nl
prefLabel
Depletion of hematogenous macr ...... perimental spinal cord injury.
@en
Depletion of hematogenous macr ...... perimental spinal cord injury.
@nl
P2093
P356
P1476
Depletion of hematogenous macr ...... perimental spinal cord injury.
@en
P2093
P304
P356
10.1006/EXNR.1999.7118
P407
P577
1999-08-01T00:00:00Z