Differences on the inhibitory specificities of H-Ras, K-Ras, and N-Ras (N17) dominant negative mutants are related to their membrane microlocalization.
about
Activation of H-Ras in the endoplasmic reticulum by the RasGRF family guanine nucleotide exchange factorsRole of TC21/R-Ras2 in enhanced migration of neurofibromin-deficient Schwann cellsMASL1 induces erythroid differentiation in human erythropoietin-dependent CD34+ cells through the Raf/MEK/ERK pathwayRole of Rac1-dependent NADPH oxidase in the growth of pancreatic cancer.Ras, an actor on many stages: posttranslational modifications, localization, and site-specified events.PP2A-mediated regulation of Ras signaling in G2 is essential for stable quiescence and normal G1 length.Spatial cycles in G-protein crowd controlMutations in the c-Kit gene disrupt mitogen-activated protein kinase signaling during tumor development in adenoid cystic carcinoma of the salivary glandsAlteration of EGFR spatiotemporal dynamics suppresses signal transduction.Transforming growth factor beta receptor 2 (TGFBR2) changes sialylation in the microsatellite unstable (MSI) Colorectal cancer cell line HCT116SOS1 and Ras regulate epithelial tight junction formation in the human airway through EMP1.Differential Regulation of RasGAPs in Cancer.Classic Ras Proteins Promote Proliferation and Survival via Distinct Phosphoproteome Alterations in Neurofibromin-Null Malignant Peripheral Nerve Sheath Tumor Cells.JAK/STAT, Raf/MEK/ERK, PI3K/Akt and BCR-ABL in cell cycle progression and leukemogenesis.Synthesis and evaluation of antitumor activity of novel N-acyllavendamycin analogues and quinoline-5,8-dionesOncogenic Ras in tumour progression and metastasis.Transcriptomal profiling of site-specific Ras signals.Continuous treatment with FTS confers resistance to apoptosis and affects autophagy.Kallikrein-related peptidase (KLK) family mRNA variants and protein isoforms in hormone-related cancers: do they have a function?Genetics of Bladder Malignant Tumors in ChildhoodContextualizing the Genes Altered in Bladder Neoplasms in Pediatric andTeen Patients Allows Identifying Two Main Classes of Biological ProcessesInvolved and New Potential Therapeutic TargetsThe disordered hypervariable region and the folded catalytic domain of oncogenic K-Ras4B partner in phospholipid binding.Effect of Ras inhibition in hematopoiesis and BCR/ABL leukemogenesisActivated k-ras, but not h-ras or N-ras, regulates brain neural stem cell proliferation in a raf/rb-dependent manner.Regulation of H-Ras-driven MAPK signaling, transformation and tumorigenesis, but not PI3K signaling and tumor progression, by plasma membrane microdomains.Ras activation in Jurkat T cells following low-grade stimulation of the T-cell receptor is specific to N-Ras and occurs only on the Golgi apparatus.Structural and spatial determinants regulating TC21 activation by RasGRF family nucleotide exchange factorsLocalized diacylglycerol-dependent stimulation of Ras and Rap1 during phagocytosis.The adaptor protein AMOT promotes the proliferation of mammary epithelial cells via the prolonged activation of the extracellular signal-regulated kinases.H-Ras-specific activation of Rac-MKK3/6-p38 pathway: its critical role in invasion and migration of breast epithelial cells.H-ras distribution and signaling in plasma membrane microdomains are regulated by acylation and deacylation events.Ras palmitoylation is necessary for N-Ras activation and signal propagation in growth factor signalling.The differential effects of wild-type and mutated K-Ras on MST2 signaling are determined by K-Ras activation kinetics.The MEK inhibitor PD184352 enhances BMS-214662-induced apoptosis in CD34+ CML stem/progenitor cellsProto-oncogenic H-Ras, K-Ras, and N-Ras are involved in muscle differentiation via phosphatidylinositol 3-kinase.Ras subcellular localization defines extracellular signal-regulated kinase 1 and 2 substrate specificity through distinct utilization of scaffold proteinsAbsence of K-Ras Reduces Proliferation and Migration But Increases Extracellular Matrix Synthesis in Fibroblasts.Activated Kras, but not Hras or Nras, may initiate tumors of endodermal origin via stem cell expansionMyc antagonizes Ras-mediated growth arrest in leukemia cells through the inhibition of the Ras-ERK-p21Cip1 pathway.RasGRP1 sensitizes an immature B cell line to antigen receptor-induced apoptosis.
P2860
Q24601002-48639A5F-D05A-4CC9-820E-88F96334D567Q28511967-AC455541-B1A1-452B-8FCE-D412672721F0Q30009940-8B740D73-E4F1-418D-954C-B2AD3C8DAD56Q30498776-C21E1606-1C2B-4CEF-BE89-3B21DF381F05Q33351823-33C1347D-0874-49B6-B9B2-29BBEB664FE8Q33985022-F0D0EB00-5BA9-4801-A6B1-4DC58C56119AQ34076266-91727DBC-7FB6-4FD4-A210-BD43D86CEA01Q34105349-4A4DE0BE-809E-4889-8F39-1F18272E7799Q34326437-2B8436DC-9A64-48FD-B20F-85E8B0A76FFEQ34611732-A8151ABF-5F4F-46C1-9AD6-640A46975E49Q35006945-0ED67E7A-6DFC-4B5A-B153-A426F9033AA3Q35083732-EE970ACB-0ABD-4786-B0D5-321D7B56BFA3Q35612406-867F9937-D2AA-4F25-A4B2-A2225E592BC9Q35635353-A79E9FAF-AB1F-40EF-B3DA-87C27ADF2570Q35857145-48C8B155-D350-4E2B-A82F-1FA0F869B35BQ36103287-B80ACB14-5D31-4BC2-9277-A6C7FA027080Q36158417-2673A1E6-3479-4A1F-994D-C6C9E4E3D4A9Q36267411-903F0949-B574-4D15-A48C-C0E2D8C857E3Q36519378-532AC179-ED8D-41EB-9710-D32D92BDC867Q36658484-47A064C8-B012-4ABB-8E73-948900285A31Q36658489-44764AA9-CBAA-4128-9CC6-D1C1C0D2645BQ36669604-3510CD58-3E99-4206-99D4-4302255CB915Q36735335-54E90A02-B49A-4C14-B7E3-F68D62CA89FCQ36956839-6B2EE777-EE6F-43EB-AD9C-200FBE0D04F2Q37096248-B289C08D-BD8E-426E-8DC9-7F93E19472DAQ37274925-D4BB77C7-0055-41CB-A263-18D16A978365Q37387979-9B8014F8-83F7-4458-A69D-EFD71B9C4557Q37431056-86557807-EC66-42C0-BB06-CBA25374ABF3Q37718727-0D7D62D5-E4BB-4B5C-BA7A-E4B229406EFAQ38515287-E68FF12B-8741-4A22-B555-7A29ECA15C04Q38899159-D712B93D-86AE-41D9-8562-E9EE80611CC9Q39140690-1A4432AF-E994-434F-8EAD-0AA4FA2DDB49Q39185488-DFCCC3BE-C7BF-4194-85C9-4AC7C5CFABC7Q39560403-38917306-4C52-4CBF-8DCB-69B4E1374130Q39684105-9FFBFAA2-074E-4BF9-90E7-547DC8C2D5AAQ39900720-00F9A53D-DFF4-465D-96D6-A126161D17ABQ39999843-822BACFA-AABE-4041-A7D5-35E254860D59Q40014102-383B6880-9CAE-40AE-9585-8845E71A3BCEQ40495521-9B5AFA9B-FEFE-4D8A-9115-0545CB881899Q40588937-B07C39EA-2563-4E6B-80E1-3C8A41E8FF7F
P2860
Differences on the inhibitory specificities of H-Ras, K-Ras, and N-Ras (N17) dominant negative mutants are related to their membrane microlocalization.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
2002年论文
@zh
2002年论文
@zh-cn
name
Differences on the inhibitory ...... ir membrane microlocalization.
@en
Differences on the inhibitory ...... ir membrane microlocalization.
@nl
type
label
Differences on the inhibitory ...... ir membrane microlocalization.
@en
Differences on the inhibitory ...... ir membrane microlocalization.
@nl
prefLabel
Differences on the inhibitory ...... ir membrane microlocalization.
@en
Differences on the inhibitory ...... ir membrane microlocalization.
@nl
P2093
P2860
P356
P1476
Differences on the inhibitory ...... ir membrane microlocalization.
@en
P2093
Angel Pellicer
David S Aaronson
Imanol Arozarena
Maria T Berciano
Miguel Lafarga
Piero Crespo
P2860
P304
P356
10.1074/JBC.M209807200
P407
P577
2002-11-27T00:00:00Z