Restriction of transient receptor potential vanilloid-1 to the peptidergic subset of primary afferent neurons follows its developmental downregulation in nonpeptidergic neurons.
about
TRPV1: A Target for Rational Drug DesignCapsaicin, Nociception and PainThe functional and anatomical dissection of somatosensory subpopulations using mouse geneticsCT-guided injection of a TRPV1 agonist around dorsal root ganglia decreases pain transmission in swine.TMEM16C facilitates Na(+)-activated K+ currents in rat sensory neurons and regulates pain processingSelective spider toxins reveal a role for the Nav1.1 channel in mechanical painSubtype-Selective Small Molecule Inhibitors Reveal a Fundamental Role for Nav1.7 in Nociceptor Electrogenesis, Axonal Conduction and Presynaptic Release.Direct reticular projections of trigeminal sensory fibers immunoreactive to CGRP: potential monosynaptic somatoautonomic projections.Acute heat-evoked temperature sensation is impaired but not abolished in mice lacking TRPV1 and TRPV3 channels.Physiological temperatures drive glutamate release onto trigeminal superficial dorsal horn neurons.Threshold-free population analysis identifies larger DRG neurons to respond stronger to NGF stimulation.Selective distribution of GABA(A) receptor subtypes in mouse spinal dorsal horn neurons and primary afferents.Activation of brainstem neurons by underwater diving in the ratCGRPα-expressing sensory neurons respond to stimuli that evoke sensations of pain and itch.Parcellation of cerebellins 1, 2, and 4 among different subpopulations of dorsal horn neurons in mouse spinal cordThe mammalian diving response: an enigmatic reflex to preserve life?Cholinergic neurotransmission links solitary chemosensory cells to nasal inflammationRole of peptidergic nerve terminals in the skin: reversal of thermal sensation by calcitonin gene-related peptide in TRPV1-depleted neuropathy.The modality-specific contribution of peptidergic and non-peptidergic nociceptors is manifest at the level of dorsal horn nociresponsive neurons.External QX-314 inhibits evoked cranial primary afferent synaptic transmission independent of TRPV1.αCGRP is essential for algesic exocytotic mobilization of TRPV1 channels in peptidergic nociceptors.Acupuncture for visceral pain: neural substrates and potential mechanisms.VGLUT2 controls heat and punctuate hyperalgesia associated with nerve injury via TRPV1-Cre primary afferentsNovel endogenous N-acyl amides activate TRPV1-4 receptors, BV-2 microglia, and are regulated in brain in an acute model of inflammation.Inhibitory Interneurons That Express GFP in the PrP-GFP Mouse Spinal Cord Are Morphologically Heterogeneous, Innervated by Several Classes of Primary Afferent and Include Lamina I Projection Neurons among Their Postsynaptic TargetsModeling pain in vitro using nociceptor neurons reprogrammed from fibroblasts.Robo2 determines subtype-specific axonal projections of trigeminal sensory neurons.Social stress in mice induces urinary bladder overactivity and increases TRPV1 channel-dependent afferent nerve activity.Dynasore blocks evoked release while augmenting spontaneous synaptic transmission from primary visceral afferents.The unsilent majority-TRPV1 drives "spontaneous" transmission of unmyelinated primary afferents within cardiorespiratory NTSNociceptor Sensitization Depends on Age and Pain Chronicity(1,2,3).A combined electrophysiological and morphological study of neuropeptide Y-expressing inhibitory interneurons in the spinal dorsal horn of the mouse.Ablation of sensory neurons in a genetic model of pancreatic ductal adenocarcinoma slows initiation and progression of cancer.Peptidergic CGRPα primary sensory neurons encode heat and itch and tonically suppress sensitivity to cold.Bradykinin Induces TRPV1 Exocytotic Recruitment in Peptidergic Nociceptors.Distinct Calcium Sources Support Multiple Modes of Synaptic Release from Cranial Sensory Afferents.Epidermal keratinocyte polarity and motility require Ca²⁺ influx through TRPV1.Low-cost functional plasticity of TRPV1 supports heat tolerance in squirrels and camels.Targeting Pain-evoking Transient Receptor Potential Channels for the Treatment of Pain.Gi-DREADD Expression in Peripheral Nerves Produces Ligand-Dependent Analgesia, as well as Ligand-Independent Functional Changes in Sensory Neurons
P2860
Q26738621-73316853-B457-494E-9C3E-9B1F34203495Q26744369-12720E91-2221-4469-96DC-BC0D1044DF88Q27014891-573AFA32-E84D-4862-835E-0F3654F2EDB1Q27324714-2087A438-A4E6-4FC3-B1C8-B95D62E04134Q28581384-240645D8-5D4C-441B-B85A-F1A2C4114395Q28594552-F5B1672A-BBB3-4EC5-B87B-BD7CED9FF6A5Q28601095-856782B7-549F-4172-B49C-E2EF51702E02Q33714098-EE1B0478-E54B-4CCD-B3CB-EC1E71D166ABQ33748817-8F9BC5F2-59CB-4520-8CB4-C0ABC7AC43A0Q33900700-B3B47B0A-DBFB-41AD-ABEB-2DC80CF3EF97Q34222320-7ABF82CF-DEC7-4DA9-BCEF-B1F6A051FEA1Q34241326-132E2CAE-386F-4F0C-82D0-A66622A56CE1Q34259589-5004ADC7-8406-44FE-8E26-92ECCE4B1DA9Q34260129-223B1E3F-CF72-457B-817E-DCC26E20F75DQ34357507-C1BA9FD4-82A8-4AB1-A04F-A623FCCCEB5AQ34368106-BB827379-6EF1-4067-8383-CD47D42D1485Q34414185-82208EF1-DBA4-446E-8A5F-733B0494AAD6Q34500724-58E657B1-9D72-4828-A1C7-98E4BB899A8EQ34522611-A322AC64-7620-44E4-B72F-B00A64FB621CQ34627127-59EF215D-D4B0-4146-A8D5-64482737CCDFQ34793162-F071F3F1-198E-436C-A1B4-A8C49F155B06Q34966570-3A5104C5-3F71-4FFE-A903-34099E789DAEQ35007197-520892CE-76A6-4465-9A5A-15E738A5C52CQ35227731-B6175110-B06F-485F-A2F7-6725EAE246AAQ35598747-4FC8019D-5B7F-408A-BAF0-B1B54661AA31Q35600035-0B343BD4-1DF6-468A-97F7-0FA60039B23BQ35655632-53796D07-4853-48C6-9356-5E9C735FBF63Q36114103-4C2C8F06-81BE-41A4-8866-078AF9475B9EQ36328379-F31CA89E-C04C-4631-B418-400DB42D42C4Q36494873-2444411E-9BC9-4F60-8A22-370EEBB897D1Q36555867-B3A24573-A34F-4191-93EB-A8DE7B2F0A07Q36574960-C63A73AC-1781-48D5-8430-75F9D5689A30Q36710531-98D3EFC2-843C-4F74-98F2-38D8F83DAC80Q36772210-591E7C70-7FB1-40CD-8058-024684464A29Q37030016-50E55380-1390-407B-9DDD-9BECC6C2132CQ37198425-F20EED05-30BB-4826-8B9D-A37A75AE50F4Q37280498-ED5C6803-94E5-4D22-9EF5-7CDAE3D82A5AQ37322990-153DF305-4D8B-473C-A529-F7EB18C8125FQ37362053-C8821437-7F94-42E0-9E2D-D71B390117FCQ37372025-A8F02E7A-72F8-43C8-9407-987BAF192EDB
P2860
Restriction of transient receptor potential vanilloid-1 to the peptidergic subset of primary afferent neurons follows its developmental downregulation in nonpeptidergic neurons.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Restriction of transient recep ...... ion in nonpeptidergic neurons.
@en
Restriction of transient recep ...... ion in nonpeptidergic neurons.
@nl
type
label
Restriction of transient recep ...... ion in nonpeptidergic neurons.
@en
Restriction of transient recep ...... ion in nonpeptidergic neurons.
@nl
prefLabel
Restriction of transient recep ...... ion in nonpeptidergic neurons.
@en
Restriction of transient recep ...... ion in nonpeptidergic neurons.
@nl
P2093
P2860
P1476
Restriction of transient recep ...... ion in nonpeptidergic neurons.
@en
P2093
Alexander T Chesler
Allan I Basbaum
Daniel J Cavanaugh
David Julius
Joao M Bráz
Nirao M Shah
P2860
P304
10119-10127
P356
10.1523/JNEUROSCI.1299-11.2011
P407
P577
2011-07-01T00:00:00Z