Role of CXCR2 in cigarette smoke-induced lung inflammation. - Wikidata - awgldk - TriplyDB

Role of CXCR2 in cigarette smoke-induced lung inflammation.

Role of CXCR2 in cigarette smoke-induced lung inflammation.

http://www.wikidata.org/entity/Q36801487

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Chemokine receptors and their therapeutic opportunities in diseased lung: far beyond leukocyte trafficking Targeting CXCR1/CXCR2 receptor antagonism in malignant melanoma Neu-164 and Neu-107, two novel antioxidant and anti-myeloperoxidase compounds, inhibit acute cigarette smoke-induced lung inflammation Genetic Ablation of CXCR2 Protects against Cigarette Smoke-Induced Lung Inflammation and Injury Inhibition of human neutrophil elastase by pentacyclic triterpenes Bronchiolar chemokine expression is different after single versus repeated cigarette smoke exposure Effect of cigarette smoke extract on dendritic cells and their impact on T-cell proliferation.Aging enhances susceptibility to cigarette smoke-induced inflammation through bronchiolar chemokines.Chemokine receptor CXCR3 is important for lung tissue damage and airway remodeling induced by short-term exposure to cigarette smoking in mice Protein kinase C zeta mediates cigarette smoke/aldehyde- and lipopolysaccharide-induced lung inflammation and histone modifications.Inflammatory changes in the airways of mice caused by cigarette smoke exposure are only partially reversed after smoking cessation.The role of the CCR1 receptor in the inflammatory response to tobacco smoke in a mouse model Small molecule antagonists for CXCR2 and CXCR1 inhibit human colon cancer liver metastases.An aberrant leukotriene A4 hydrolase-proline-glycine-proline pathway in the pathogenesis of chronic obstructive pulmonary disease Small-molecule antagonists for CXCR2 and CXCR1 inhibit human melanoma growth by decreasing tumor cell proliferation, survival, and angiogenesis A novel anti-inflammatory and pro-resolving role for resolvin D1 in acute cigarette smoke-induced lung inflammation Lung-targeted overexpression of the NF-κB member RelB inhibits cigarette smoke-induced inflammation Neonatal hyperoxia contributes additively to cigarette smoke-induced chronic obstructive pulmonary disease changes in adult mice.Ocular fibroblast diversity: implications for inflammation and ocular wound healing Aryl hydrocarbon receptor-deficient mice develop heightened inflammatory responses to cigarette smoke and endotoxin associated with rapid loss of the nuclear factor-kappaB component RelB.The effect of a selective CXCR2 antagonist (AZD5069) on human blood neutrophil count and innate immune functions VEGFR-2 inhibition augments cigarette smoke-induced oxidative stress and inflammatory responses leading to endothelial dysfunction Genetic ablation of NADPH oxidase enhances susceptibility to cigarette smoke-induced lung inflammation and emphysema in mice.Exposure to neonatal cigarette smoke causes durable lung changes but does not potentiate cigarette smoke-induced chronic obstructive pulmonary disease in adult mice.High-dose but not low-dose mainstream cigarette smoke suppresses allergic airway inflammation by inhibiting T cell function Attenuation of inflammatory mediator production by the NF-κB member RelB is mediated by microRNA-146a in lung fibroblasts.The immunopathogenesis of chronic obstructive pulmonary disease: insights from recent research.Mechanisms of cigarette smoke-induced COPD: insights from animal models.Identifying targets for COPD treatment through gene expression analyses.The composition of cigarette smoke determines inflammatory cell recruitment to the lung in COPD mouse models.The functional significance behind expressing two IL-8 receptor types on PMN.The possible role of granzyme B in the pathogenesis of chronic obstructive pulmonary disease.Promoting effect of neutrophils on lung tumorigenesis is mediated by CXCR2 and neutrophil elastase.CXCL8 and its cognate receptors in melanoma progression and metastasis.Preclinical animal models of asthma and chronic obstructive pulmonary disease.Current concepts on oxidative/carbonyl stress, inflammation and epigenetics in pathogenesis of chronic obstructive pulmonary disease.CXCR2 in acute lung injury.Role of histone deacetylase 2 in epigenetics and cellular senescence: implications in lung inflammaging and COPD Blockade of CXCR2 signalling: a potential therapeutic target for preventing neutrophil-mediated inflammatory diseases.IKK alpha causes chromatin modification on pro-inflammatory genes by cigarette smoke in mouse lung.

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P2860

Role of CXCR2 in cigarette smoke-induced lung inflammation.

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Role of CXCR2 in cigarette smoke-induced lung inflammation.

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Role of CXCR2 in cigarette smoke-induced lung inflammation.

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American Journal of Physiology - Lung Cellular and Molecular Physiology

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Role of CXCR2 in cigarette smoke-induced lung inflammation.

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Neutrophil and B cell expansion in mice that lack the murine IL-8 receptor homolog

CXC chemokine redundancy ensures local neutrophil recruitment during acute inflammation

CXC chemokine receptor CXCR2 is essential for protective innate host response in murine Pseudomonas aeruginosa pneumonia.

The many faces of airway inflammation. Asthma and chronic obstructive pulmonary disease. Asthma Research Group.

Mechanisms of leukocyte sequestration in inflamed lungs.

Cigarette smoke induces cyclooxygenase-2 and microsomal prostaglandin E2 synthase in human lung fibroblasts: implications for lung inflammation and cancer.

Acute lung injury: the role of cytokines in the elicitation of neutrophils.

Cytokine modulators as novel therapies for airway disease.

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Chronic obstructive pulmonary disease: definition and epidemiology.

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