The electrophysiological properties of spontaneously beating pacemaker cells isolated from mouse sinoatrial node. - Wikidata - awgldk - TriplyDB

The electrophysiological properties of spontaneously beating pacemaker cells isolated from mouse sinoatrial node.

The electrophysiological properties of spontaneously beating pacemaker cells isolated from mouse sinoatrial node.

http://www.wikidata.org/entity/Q44524951

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CaMKII regulation of cardiac K channels Role of sodium and calcium dysregulation in tachyarrhythmias in sudden cardiac death.Sinus node dysfunction following targeted disruption of the murine cardiac sodium channel gene Scn5a Age-dependent changes in Na current magnitude and TTX-sensitivity in the canine sinoatrial node.PP2 prevents β-adrenergic stimulation of cardiac pacemaker activity.Phosphorylation and modulation of hyperpolarization-activated HCN4 channels by protein kinase A in the mouse sinoatrial node Specific pattern of ionic channel gene expression associated with pacemaker activity in the mouse heart.BK channels regulate sinoatrial node firing rate and cardiac pacing in vivo The mitochondrial uniporter controls fight or flight heart rate increases.Pacemaker activity and ionic currents in mouse atrioventricular node cells.Enhancement of Spontaneous Activity by HCN4 Overexpression in Mouse Embryonic Stem Cell-Derived Cardiomyocytes - A Possible Biological Pacemaker.Modeling the heart.New insights into the molecular and cellular workings of the cardiac Na+/Ca2+ exchanger.What keeps us ticking: a funny current, a calcium clock, or both?Dynamic Support Culture of Murine Skeletal Muscle-Derived Stem Cells Improves Their Cardiogenic Potential In Vitro Distinct populations of HCN pacemaker channels produce voltage-dependent and voltage-independent currents.Expression and roles of Cav1.3 (α1D) L-type Ca²+ channel in atrioventricular node automaticity.Electrophysiology and pacemaker function of the developing sinoatrial node.Genesis and regulation of the heart automaticity.Methods for the Isolation, Culture, and Functional Characterization of Sinoatrial Node Myocytes from Adult Mice.Cardiac strong inward rectifier potassium channels.Small functional If current in sinoatrial pacemaker cells of the brown trout (Salmo trutta fario) heart despite strong expression of HCN channel transcripts.A Singular Role of IK1 Promoting the Development of Cardiac Automaticity during Cardiomyocyte Differentiation by IK1 -Induced Activation of Pacemaker Current.A mathematical model of action potentials of mouse sinoatrial node cells with molecular bases.Murine Electrophysiological Models of Cardiac Arrhythmogenesis.Neuregulin/ErbB signaling regulates cardiac subtype specification in differentiating human embryonic stem cells.CaV1.3 L-type Ca2+ channel contributes to the heartbeat by generating a dihydropyridine-sensitive persistent Na+ current.A rapidly activating delayed rectifier K+ current regulates pacemaker activity in adult mouse sinoatrial node cells.Effects of C-type natriuretic peptide on ionic currents in mouse sinoatrial node: a role for the NPR-C receptor.Identification and detection of the isolated sinus venosus from the Asian toad.The mechanism of increased postnatal heart rate and sinoatrial node pacemaker activity in mice.Role of sarcolemmal ATP-sensitive K+ channels in the regulation of sinoatrial node automaticity: an evaluation using Kir6.2-deficient mice.Chronic heart rate reduction remodels ion channel transcripts in the mouse sinoatrial node but not in the ventricle.Heterogeneous functional expression of the sustained inward Na+ current in guinea pig sinoatrial node cells.L-type Cav1.3 channels regulate ryanodine receptor-dependent Ca2+ release during sino-atrial node pacemaker activity.Kir2 inward rectification-controlled precise and dynamic balances between Kir2 and HCN currents initiate pacemaking activity.

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P2860

The electrophysiological properties of spontaneously beating pacemaker cells isolated from mouse sinoatrial node.

http://www.wikidata.org/entity/Q44524951

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

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2003年學術文章

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name

The electrophysiological prope ...... ed from mouse sinoatrial node.

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The electrophysiological prope ...... ed from mouse sinoatrial node.

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JPHYSIOL.2003.040501

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The Journal of Physiology

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The electrophysiological prope ...... ed from mouse sinoatrial node.

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Akinori Noma

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Hyun-Sung Cho

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Makoto Takano

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P2860

Transgenic mice overexpressing human KvLQT1 dominant-negative isoform. Part I: Phenotypic characterisation

Identification of a gene encoding a hyperpolarization-activated pacemaker channel of brain

Molecular characterization of the hyperpolarization-activated cation channel in rabbit heart sinoatrial node.

Functional embryonic cardiomyocytes after disruption of the L-type alpha1C (Cav1.2) calcium channel gene in the mouse.

Molecular basis of functional voltage-gated K+ channel diversity in the mammalian myocardium.

The sinoatrial node, a heterogeneous pacemaker structure.

Molecular and cellular mechanisms of cardiac arrhythmias.

Genetic manipulation of cardiac K(+) channel function in mice: what have we learned, and where do we go from here?

A family of hyperpolarization-activated mammalian cation channels.

The consequences of disrupting cardiac inwardly rectifying K(+) current (I(K1)) as revealed by the targeted deletion of the murine Kir2.1 and Kir2.2 genes.

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169-180

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10.1113/JPHYSIOL.2003.040501

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Pt 1

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550

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2003-07-01T00:00:00Z

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12879867

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electrophysiology

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2343002

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