Modulation of the JNK pathway in liver affects insulin resistance status. - Wikidata - awgldk - TriplyDB

Modulation of the JNK pathway in liver affects insulin resistance status.

Modulation of the JNK pathway in liver affects insulin resistance status.

http://www.wikidata.org/entity/Q45032503

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Inflammation, stress, and diabetes The protein kinase IKKepsilon regulates energy balance in obese mice Stress kinases in the modulation of metabolism and energy balance Choline supplementation promotes hepatic insulin resistance in phosphatidylethanolamine N-methyltransferase-deficient mice via increased glucagon action Activation of the Liver X Receptor by Agonist TO901317 Improves Hepatic Insulin Resistance via Suppressing Reactive Oxygen Species and JNK Pathway Palmitate induces insulin resistance in H4IIEC3 hepatocytes through reactive oxygen species produced by mitochondria Inhibition of gluconeogenesis through transcriptional activation of EGR1 and DUSP4 by AMP-activated kinase A predominant role for parenchymal c-Jun amino terminal kinase (JNK) in the regulation of systemic insulin sensitivity.Prevention of steatosis by hepatic JNK1 Nutrient sensing and inflammation in metabolic diseases.Metaflammasome components in the human brain: a role in dementia with Alzheimer's pathology?Myeloid differentiation factor 88 (MyD88)-deficiency increases risk of diabetes in mice Dietary Lycium barbarum polysaccharide induces Nrf2/ARE pathway and ameliorates insulin resistance induced by high-fat via activation of PI3K/AKT signaling.Leukocyte Cell-Derived Chemotaxin-2: It's Role in Pathophysiology and Future in Clinical Medicine Adenovirus infection results in alterations of insulin signaling and glucose homeostasis.Inflammation and insulin resistance: an old story with new ideas.The effects of palmitate on hepatic insulin resistance are mediated by NADPH Oxidase 3-derived reactive oxygen species through JNK and p38MAPK pathways Role of reactive oxygen species in injury-induced insulin resistance.cJun NH2-terminal kinase 1 (JNK1): roles in metabolic regulation of insulin resistance JNK regulation of hepatic manifestations of the metabolic syndrome.Selective inactivation of c-Jun NH2-terminal kinase in adipose tissue protects against diet-induced obesity and improves insulin sensitivity in both liver and skeletal muscle in mice.Anti-Inflammatory Action of Insulin via Induction of Gadd45-β Transcription by the mTOR Signaling Pathway.p38 mitogen-activated protein kinase-dependent transactivation of ErbB receptor family: a novel common mechanism for stress-induced IRS-1 serine phosphorylation and insulin resistance.Cell lysis-free quantum dot multicolor cellular imaging-based mechanism study for TNF-α-induced insulin resistance.Sirtuin 1 (SIRT1) protein degradation in response to persistent c-Jun N-terminal kinase 1 (JNK1) activation contributes to hepatic steatosis in obesity.Role of inhibitory κB kinase and c-Jun NH2-terminal kinase in the development of hepatic insulin resistance in critical illness diabetes Uses for JNK: the many and varied substrates of the c-Jun N-terminal kinases.Hepatitis B virus X protein regulates hepatic glucose homeostasis via activation of inducible nitric oxide synthase.Neuroendocrine and metabolic components of dopamine agonist amelioration of metabolic syndrome in SHR rats Gender-dimorphic regulation of DJ1 and its interactions with metabolic proteins in streptozotocin-induced diabetic rats.Reproductive tissues maintain insulin sensitivity in diet-induced obesity.Mechanisms Linking Inflammation to Insulin Resistance The JNK pathway as a therapeutic target for diabetes.Inhibition of apolipoprotein B100 secretion by lipid-induced hepatic endoplasmic reticulum stress in rodents.Cudrania tricuspidata water extract improved obesity-induced hepatic insulin resistance in db/db mice by suppressing ER stress and inflammation Pathways leading to muscle insulin resistance--the muscle--fat connection.JNK pathway: diseases and therapeutic potential.Diabetes associated cell stress and dysfunction: role of mitochondrial and non-mitochondrial ROS production and activity.Central melanin-concentrating hormone influences liver and adipose metabolism via specific hypothalamic nuclei and efferent autonomic/JNK1 pathways.Selective Insulin Resistance and the Development of Cardiovascular Diseases in Diabetes: The 2015 Edwin Bierman Award Lecture

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P2860

Modulation of the JNK pathway in liver affects insulin resistance status.

http://www.wikidata.org/entity/Q45032503

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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2004年學術文章

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name

Modulation of the JNK pathway in liver affects insulin resistance status.

@en

Modulation of the JNK pathway in liver affects insulin resistance status.

@nl

type

label

Modulation of the JNK pathway in liver affects insulin resistance status.

@en

Modulation of the JNK pathway in liver affects insulin resistance status.

@nl

prefLabel

Modulation of the JNK pathway in liver affects insulin resistance status.

@en

Modulation of the JNK pathway in liver affects insulin resistance status.

@nl

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Journal of Biological Chemistry

P1476

Modulation of the JNK pathway in liver affects insulin resistance status.

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P2093

Dan Kawamori

http://www.w3.org/2001/XMLSchema#string

Hideaki Kaneto

http://www.w3.org/2001/XMLSchema#string

Masahiro Hatazaki

http://www.w3.org/2001/XMLSchema#string

Masatsugu Hori

http://www.w3.org/2001/XMLSchema#string

Munehide Matsuhisa

http://www.w3.org/2001/XMLSchema#string

Taka-Aki Matsuoka

http://www.w3.org/2001/XMLSchema#string

Takeshi Miyatsuka

http://www.w3.org/2001/XMLSchema#string

Yoshihisa Nakatani

http://www.w3.org/2001/XMLSchema#string

Yoshimitsu Yamasaki

http://www.w3.org/2001/XMLSchema#string

Yoshitaka Kajimoto

http://www.w3.org/2001/XMLSchema#string

P2860

Insulin signalling and the regulation of glucose and lipid metabolism

JNK1: a protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain

A simplified system for generating recombinant adenoviruses

Oxidants, oxidative stress and the biology of ageing

The c-Jun NH(2)-terminal kinase promotes insulin resistance during association with insulin receptor substrate-1 and phosphorylation of Ser(307)

Signal transduction by the JNK group of MAP kinases

Reversal of obesity- and diet-induced insulin resistance with salicylates or targeted disruption of Ikkbeta

Mammalian MAP kinase signalling cascades

Involvement of c-Jun N-terminal kinase in oxidative stress-mediated suppression of insulin gene expression

Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage

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45803-45809

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10.1074/JBC.M406963200

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44

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279

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2004-08-24T00:00:00Z

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15331609

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