The mdx-amplification-resistant mutation system assay, a simple and rapid polymerase chain reaction-based detection of the mdx allele.
about
Osteopontin promotes fibrosis in dystrophic mouse muscle by modulating immune cell subsets and intramuscular TGF-betaGalectin-1 Protein Therapy Prevents Pathology and Improves Muscle Function in the mdx Mouse Model of Duchenne Muscular DystrophyAbsence of alpha 7 integrin in dystrophin-deficient mice causes a myopathy similar to Duchenne muscular dystrophyP2RX7 purinoceptor: a therapeutic target for ameliorating the symptoms of duchenne muscular dystrophyTargeted nucleotide exchange in bovine myostatin gene.CXCL12 and osteopontin from bone marrow-derived mesenchymal stromal cells improve muscle regeneration.Myogenic stem cell function is impaired in mice lacking the forkhead/winged helix protein MNFGenetic background affects properties of satellite cells and mdx phenotypes.Excitation-contraction coupling alterations in mdx and utrophin/dystrophin double knockout mice: a comparative study.MAP kinase phosphatase-1 deficiency impairs skeletal muscle regeneration and exacerbates muscular dystrophyNeuronal nitric oxide synthase-rescue of dystrophin/utrophin double knockout mice does not require nNOS localization to the cell membraneRestoration of dystrophin expression using the Sleeping Beauty transposon.Interleukin-10 reduces the pathology of mdx muscular dystrophy by deactivating M1 macrophages and modulating macrophage phenotype.Inflammatory monocytes promote progression of Duchenne muscular dystrophy and can be therapeutically targeted via CCR2.Myogenic Akt signaling attenuates muscular degeneration, promotes myofiber regeneration and improves muscle function in dystrophin-deficient mdx mice.Genotyping mdx, mdx3cv, and mdx4cv mice by primer competition polymerase chain reaction.IFN-γ promotes muscle damage in the mdx mouse model of Duchenne muscular dystrophy by suppressing M2 macrophage activation and inhibiting muscle cell proliferationExpression of the dystrophin isoform Dp116 preserves functional muscle mass and extends lifespan without preventing dystrophy in severely dystrophic miceA novel mechanism of autophagic cell death in dystrophic muscle regulated by P2RX7 receptor large-pore formation and HSP90.Dysferlin-mediated membrane repair protects the heart from stress-induced left ventricular injury.A nitric oxide synthase transgene ameliorates muscular dystrophy in mdx miceEnhanced expression of the alpha 7 beta 1 integrin reduces muscular dystrophy and restores viability in dystrophic miceMajor basic protein-1 promotes fibrosis of dystrophic muscle and attenuates the cellular immune response in muscular dystrophy.Rescue of dystrophin expression in mdx mouse muscle by RNA/DNA oligonucleotides.Shifts in macrophage phenotypes and macrophage competition for arginine metabolism affect the severity of muscle pathology in muscular dystrophy.Sub-physiological sarcoglycan expression contributes to compensatory muscle protection in mdx miceMatrix metalloproteinase-9 inhibition ameliorates pathogenesis and improves skeletal muscle regeneration in muscular dystrophy.Loss of positive allosteric interactions between neuronal nitric oxide synthase and phosphofructokinase contributes to defects in glycolysis and increased fatigability in muscular dystrophy.Osteopontin ablation ameliorates muscular dystrophy by shifting macrophages to a pro-regenerative phenotypeImpaired regenerative capacity and lower revertant fibre expansion in dystrophin-deficient mdx muscles on DBA/2 background.Klotho gene silencing promotes pathology in the mdx mouse model of Duchenne muscular dystrophy.Myogenic reprogramming of bone marrow derived cells in a W⁴¹Dmd(mdx) deficient mouse modelA deficit of brain dystrophin impairs specific amygdala GABAergic transmission and enhances defensive behaviour in mice.Dystrophin rescue by trans-splicing: a strategy for DMD genotypes not eligible for exon skipping approachescDNA sequence of five mouse guanine deaminase (Gda) alleles and mapping to mouse chromosome 19.Increased vulnerability to kainate-induced seizures in utrophin-knockout mice.Macrophages escape Klotho gene silencing in the mdx mouse model of Duchenne muscular dystrophy and promote muscle growth and increase satellite cell numbers through a Klotho-mediated pathway.A new method of genotyping MDX4CV mice by PCR-RFLP analysis.Acute pathophysiological effects of muscle-expressed Dp71 transgene on normal and dystrophic mouse muscle.Low Six4 and Six5 gene dosage improves dystrophic phenotype and prolongs life span of mdx mice.
P2860
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P2860
The mdx-amplification-resistant mutation system assay, a simple and rapid polymerase chain reaction-based detection of the mdx allele.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
@zh
1996年學術文章
@zh-hant
name
The mdx-amplification-resistan ...... d detection of the mdx allele.
@en
The mdx-amplification-resistan ...... d detection of the mdx allele.
@nl
type
label
The mdx-amplification-resistan ...... d detection of the mdx allele.
@en
The mdx-amplification-resistan ...... d detection of the mdx allele.
@nl
prefLabel
The mdx-amplification-resistan ...... d detection of the mdx allele.
@en
The mdx-amplification-resistan ...... d detection of the mdx allele.
@nl
P2860
P1433
P1476
The mdx-amplification-resistan ...... d detection of the mdx allele.
@en
P2093
Amalfitano A
Chamberlain JS
P2860
P304
P356
10.1002/(SICI)1097-4598(199612)19:12<1549::AID-MUS4>3.0.CO;2-A
P577
1996-12-01T00:00:00Z