Heterogeneous expression of repolarizing, voltage-gated K+ currents in adult mouse ventricles.
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Cardiovascular Action of Insulin in Health and Disease: Endothelial L-Arginine Transport and Cardiac Voltage-Dependent Potassium ChannelsSex differences in the mechanisms underlying long QT syndromeTrpm4 gene invalidation leads to cardiac hypertrophy and electrophysiological alterationsMarkov models of use-dependence and reverse use-dependence during the mouse cardiac action potentialLocalization of Kv1.5 channels in rat and canine myocyte sarcolemmaThe homeodomain transcription factor Irx5 establishes the mouse cardiac ventricular repolarization gradientAugmentation of Kv4.2-encoded currents by accessory dipeptidyl peptidase 6 and 10 subunits reflects selective cell surface Kv4.2 protein stabilizationInterdependent roles for accessory KChIP2, KChIP3, and KChIP4 subunits in the generation of Kv4-encoded IA channels in cortical pyramidal neuronsSpatial distributions of Kv4 channels and KChip2 isoforms in the murine heart based on laser capture microdissection.Molecular and physiological characterization of RV remodeling in a murine model of pulmonary stenosis.Regulation of excitation-contraction coupling in mouse cardiac myocytes: integrative analysis with mathematical modellingMicroRNA-133a protects against myocardial fibrosis and modulates electrical repolarization without affecting hypertrophy in pressure-overloaded adult heartsMolecular determinants of cardiac transient outward potassium current (I(to)) expression and regulationAutocrine A2 in the T-system of ventricular myocytes creates transmural gradients in ion transport: a mechanism to match contraction with load?Molecular dissection of I(A) in cortical pyramidal neurons reveals three distinct components encoded by Kv4.2, Kv4.3, and Kv1.4 alpha-subunitsHeterogeneous ventricular sympathetic innervation, altered beta-adrenergic receptor expression, and rhythm instability in mice lacking the p75 neurotrophin receptorEstrogen contributes to gender differences in mouse ventricular repolarizationTransmural heterogeneity of repolarization and Ca2+ handling in a model of mouse ventricular tissue.Multistep ion channel remodeling and lethal arrhythmia precede heart failure in a mouse model of inherited dilated cardiomyopathy.Remodeling of early-phase repolarization: a mechanism of abnormal impulse conduction in heart failureA mathematical model of the mouse ventricular myocyte contraction.Reduced sialylation impacts ventricular repolarization by modulating specific K+ channel isoforms distinctly.Fatty acid synthase modulates homeostatic responses to myocardial stress.Mice with cardiac overexpression of peroxisome proliferator-activated receptor γ have impaired repolarization and spontaneous fatal ventricular arrhythmiasArrhythmogenic mechanisms in the isolated perfused hypokalaemic murine heart.Effects of C-reactive protein on K(+) channel interaction protein 2 in cardiomyocytes.Alk7 Depleted Mice Exhibit Prolonged Cardiac Repolarization and Are Predisposed to Ventricular Arrhythmia.Impact of KChIP2 on Cardiac Electrophysiology and the Progression of Heart Failure.Mathematical modeling mechanisms of arrhythmias in transgenic mouse heart overexpressing TNF-αCan the electrophysiological action of rosiglitazone explain its cardiac side effects?Reduced Na(+) and higher K(+) channel expression and function contribute to right ventricular origin of arrhythmias in Scn5a+/- mice.Transient outward potassium current, 'Ito', phenotypes in the mammalian left ventricle: underlying molecular, cellular and biophysical mechanisms.KChIP2 genotype dependence of transient outward current (Ito) properties in cardiomyocytes isolated from male and female mice.Kv4.3 is not required for the generation of functional Ito,f channels in adult mouse ventricles.Increased intracellular magnesium attenuates β-adrenergic stimulation of the cardiac Ca(V)1.2 channel.Molecular Mechanisms of Sympathetic Remodeling and Arrhythmias.Intraventricular and interventricular cellular heterogeneity of inotropic responses to α(1)-adrenergic stimulation.Ventricular arrhythmia risk after subarachnoid hemorrhage.Hyperoxia-induced hypertrophy and ion channel remodeling in left ventriclePPARalpha-mediated remodeling of repolarizing voltage-gated K+ (Kv) channels in a mouse model of metabolic cardiomyopathy.
P2860
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P2860
Heterogeneous expression of repolarizing, voltage-gated K+ currents in adult mouse ventricles.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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@zh-hans
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name
Heterogeneous expression of re ...... nts in adult mouse ventricles.
@en
Heterogeneous expression of re ...... nts in adult mouse ventricles.
@nl
type
label
Heterogeneous expression of re ...... nts in adult mouse ventricles.
@en
Heterogeneous expression of re ...... nts in adult mouse ventricles.
@nl
prefLabel
Heterogeneous expression of re ...... nts in adult mouse ventricles.
@en
Heterogeneous expression of re ...... nts in adult mouse ventricles.
@nl
P2093
P2860
P1476
Heterogeneous expression of re ...... nts in adult mouse ventricles.
@en
P2093
David Fedida
Franck Aimond
Jeanne M Nerbonne
Jodene Eldstrom
Kathryn A Yamada
Sylvain Brunet
Weinong Guo
P2860
P304
P356
10.1113/JPHYSIOL.2004.063347
P407
P577
2004-06-11T00:00:00Z