Frequent co-alterations of TP53, p16/CDKN2A, p14ARF, PTEN tumor suppressor genes in human glioma cell lines.
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Combined RNAi-mediated suppression of Rictor and EGFR resulted in complete tumor regression in an orthotopic glioblastoma tumor modelLoss of PTEN binding adapter protein NHERF1 from plasma membrane in glioblastoma contributes to PTEN inactivationPTEN tumor suppressor associates with NHERF proteins to attenuate PDGF receptor signaling.MicroRNA 21 promotes glioma invasion by targeting matrix metalloproteinase regulatorsActivation of the human PAX6 gene through the exon 1 enhancer by transcription factors SEF and Sp1Human glioma growth is controlled by microRNA-10bBrain angiogenesis inhibitor 1 is differentially expressed in normal brain and glioblastoma independently of p53 expressionSui generis: gene therapy and delivery systems for the treatment of glioblastomaKeep-ING balance: tumor suppression by epigenetic regulationA kinome-wide RNAi screen in Drosophila Glia reveals that the RIO kinases mediate cell proliferation and survival through TORC2-Akt signaling in glioblastomaTargeted polypharmacology: discovery of dual inhibitors of tyrosine and phosphoinositide kinasesAn improved monomeric infrared fluorescent protein for neuronal and tumour brain imagingPharmacologic inhibition of cyclin-dependent kinases 4 and 6 arrests the growth of glioblastoma multiforme intracranial xenografts.The future role of personalized medicine in the treatment of glioblastoma multiformeGSK3beta regulates differentiation and growth arrest in glioblastomaMERTK Inhibition Induces Polyploidy and Promotes Cell Death and Cellular Senescence in Glioblastoma MultiformeDifferential SELEX in human glioma cell linesStructure-activity relationship of 2,2-dimethyl-2H-chromene based arylsulfonamide analogs of 3,4-dimethoxy-N-[(2,2-dimethyl-2H-chromen-6-yl)methyl]-N-phenylbenzenesulfonamide, a novel small molecule hypoxia inducible factor-1 (HIF-1) pathway inhibitHIF-1α inhibition by siRNA or chetomin in human malignant glioma cells: effects on hypoxic radioresistance and monitoring via CA9 expression.Structural mutation analysis of PTEN and its genotype-phenotype correlations in endometriosis and cancerPTEN has tumor-promoting properties in the setting of gain-of-function p53 mutations.Alpha-secretase inhibition reduces human glioblastoma stem cell growth in vitro and in vivo by inhibiting Notch.A chalcone-related small molecule that induces methuosis, a novel form of non-apoptotic cell death, in glioblastoma cells.Vasculostatin inhibits intracranial glioma growth and negatively regulates in vivo angiogenesis through a CD36-dependent mechanism.A proprotein convertase/MMP-14 proteolytic cascade releases a novel 40 kDa vasculostatin from tumor suppressor BAI1.GAPDH is not regulated in human glioblastoma under hypoxic conditions.Glioma cells on the run - the migratory transcriptome of 10 human glioma cell linesGlioma stem cells are more aggressive in recurrent tumors with malignant progression than in the primary tumor, and both can be maintained long-term in vitro.Immunocompetent murine models for the study of glioblastoma immunotherapyBioenergetic metabolites regulate base excision repair-dependent cell death in response to DNA damage.PAX6 suppression of glioma angiogenesis and the expression of vascular endothelial growth factor ADifferent molecular patterns in glioblastoma multiforme subtypes upon recurrenceRestoration of endogenous wild-type p53 activity in a glioblastoma cell line with intrinsic temperature-sensitive p53 induces growth arrest but not apoptosis.Calcium-activated potassium channels BK and IK1 are functionally expressed in human gliomas but do not regulate cell proliferation.Multiparameter computational modeling of tumor invasion.Integrative genome analysis reveals an oncomir/oncogene cluster regulating glioblastoma survivorship.Targeting human glioblastoma cells: comparison of nine viruses with oncolytic potential.PTEN loss compromises homologous recombination repair in astrocytes: implications for glioblastoma therapy with temozolomide or poly(ADP-ribose) polymerase inhibitors.Sulfonamides as a new scaffold for hypoxia inducible factor pathway inhibitors.Clinically relevant doses of chemotherapy agents reversibly block formation of glioblastoma neurospheres.
P2860
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P1343
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P2860
Frequent co-alterations of TP53, p16/CDKN2A, p14ARF, PTEN tumor suppressor genes in human glioma cell lines.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh
1999年學術文章
@zh-hant
name
Frequent co-alterations of TP5 ...... es in human glioma cell lines.
@en
Frequent co-alterations of TP5 ...... es in human glioma cell lines.
@nl
type
label
Frequent co-alterations of TP5 ...... es in human glioma cell lines.
@en
Frequent co-alterations of TP5 ...... es in human glioma cell lines.
@nl
prefLabel
Frequent co-alterations of TP5 ...... es in human glioma cell lines.
@en
Frequent co-alterations of TP5 ...... es in human glioma cell lines.
@nl
P2093
P2860
P4510
P1433
P1476
Frequent co-alterations of TP5 ...... es in human glioma cell lines.
@en
P2093
P2860
P304
P356
10.1111/J.1750-3639.1999.TB00536.X
P577
1999-07-01T00:00:00Z