Motor learning in Ts65Dn mice, a model for Down syndrome.
about
Deletion of the triplet repeat encoding polyglutamine within the mouse Huntington's disease gene results in subtle behavioral/motor phenotypes in vivo and elevated levels of ATP with cellular senescence in vitroComprehensive behavioral phenotyping of Ts65Dn mouse model of Down syndrome: activation of β1-adrenergic receptor by xamoterol as a potential cognitive enhancer.LRRK2 knockout mice have an intact dopaminergic system but display alterations in exploratory and motor co-ordination behaviors.Impairments in motor coordination without major changes in cerebellar plasticity in the Tc1 mouse model of Down syndromeHedgehog agonist therapy corrects structural and cognitive deficits in a Down syndrome mouse modelChronic up-regulation of the SHH pathway normalizes some developmental effects of trisomy in Ts65Dn mice.Timing of muscle response to a sudden leg perturbation: comparison between adolescents and adults with Down syndromeOpposite phenotypes of muscle strength and locomotor function in mouse models of partial trisomy and monosomy 21 for the proximal Hspa13-App region.Vaccination of Alzheimer's model mice with Abeta derivative in alum adjuvant reduces Abeta burden without microhemorrhagesTrisomy 21 and early brain developmentIncreased excitability and altered action potential waveform in cerebellar granule neurons of the Ts65Dn mouse model of Down syndromeWeaker control of the electrical properties of cerebellar granule cells by tonically active GABAA receptors in the Ts65Dn mouse model of Down's syndrome.Size does not always matter: Ts65Dn Down syndrome mice show cerebellum-dependent motor learning deficits that cannot be rescued by postnatal SAG treatmentCognitive and sensorimotor tasks for assessing functional impairments in mouse models of Alzheimer's disease and related disordersA new mouse model for the trisomy of the Abcg1-U2af1 region reveals the complexity of the combinatorial genetic code of down syndromeAneuploidy: from a physiological mechanism of variance to Down syndrome.Abnormal expression of the G-protein-activated inwardly rectifying potassium channel 2 (GIRK2) in hippocampus, frontal cortex, and substantia nigra of Ts65Dn mouse: a model of Down syndrome.Effects of aneuploidy on skull growth in a mouse model of Down syndrome.Effect of practice on a novel task--walking on a treadmill: preadolescents with and without Down syndrome.Rapid forgetting of social learning in the Ts65Dn mouse model of Down syndrome: New evidence for hippocampal dysfunction.
P2860
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P2860
Motor learning in Ts65Dn mice, a model for Down syndrome.
description
2001 nî lūn-bûn
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2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
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2001年學術文章
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name
Motor learning in Ts65Dn mice, a model for Down syndrome.
@en
Motor learning in Ts65Dn mice, a model for Down syndrome.
@nl
type
label
Motor learning in Ts65Dn mice, a model for Down syndrome.
@en
Motor learning in Ts65Dn mice, a model for Down syndrome.
@nl
prefLabel
Motor learning in Ts65Dn mice, a model for Down syndrome.
@en
Motor learning in Ts65Dn mice, a model for Down syndrome.
@nl
P2093
P2860
P1476
Motor learning in Ts65Dn mice, a model for Down syndrome.
@en
P2093
P2860
P356
10.1002/1098-2302(2001)38:1<33::AID-DEV3>3.0.CO;2-0
P577
2001-01-01T00:00:00Z