FTY720 suppresses CD4+CD44highCD62L- effector memory T cell-mediated colitis.
about
A functional role for CCR6 on proallergic T cells in the gastrointestinal tractShining a light on intestinal trafficCTLA4-Ig in combination with FTY720 promotes allograft survival in sensitized recipients.Pharmacological intervention studies using mouse models of the inflammatory bowel diseases: translating preclinical data into new drug therapies.Lactobacillus reuteri DSM 17938 differentially modulates effector memory T cells and Foxp3+ regulatory T cells in a mouse model of necrotizing enterocolitis.Voltage-gated potassium channel Kv1.3 blocker as a potential treatment for rat anti-glomerular basement membrane glomerulonephritis.Ozanimod (RPC1063) is a potent sphingosine-1-phosphate receptor-1 (S1P1 ) and receptor-5 (S1P5 ) agonist with autoimmune disease-modifying activity.Compared benefit of approved and experimental immunosuppressive therapeutic approaches in multiple sclerosis.Hematopoietic sphingosine 1-phosphate lyase deficiency decreases atherosclerotic lesion development in LDL-receptor deficient miceThe sphingosine-1-phosphate analogue FTY720 impairs mucosal immunity and clearance of the enteric pathogen Citrobacter rodentium.Sphingosine-1-phosphate in inflammatory bowel disease and colitis-associated colon cancer: the fat's in the fireSphingosine-1-phosphate receptor-1 (S1P1) is expressed by lymphocytes, dendritic cells, and endothelium and modulated during inflammatory bowel diseasePersistent retention of colitogenic CD4+ memory T cells causes inflammatory bowel diseases to become intractable.Immunological function of sphingosine 1-phosphate in the intestine.Targeting T-cell migration in inflammatory bowel disease.Nutritional components regulate the gut immune system and its association with intestinal immune disease development.Recurrence of autoimmunity in pancreas transplant patients: research update.Sphingolipids in neutrophil function and inflammatory responses: Mechanisms and implications for intestinal immunity and inflammation in ulcerative colitis.Frontiers in Drug Research and Development for Inflammatory Bowel Disease.Members of the novel UBASH3/STS/TULA family of cellular regulators suppress T-cell-driven inflammatory responses in vivo.FTY720 suppresses the development of colitis in lymphoid-null mice by modulating the trafficking of colitogenic CD4+ T cells in bone marrow.Colitogenic CD4+ effector-memory T cells actively recirculate in chronic colitic mice.FTY720 mediates activation suppression and G(0)/G (1) cell cycle arrest in a concanavalin A-induced mouse lymphocyte pan-activation model.Sphingosine-1-Phosphate Signaling and Metabolism Gene Signature in Pediatric Inflammatory Bowel Disease: A Matched-case Control Pilot Study.
P2860
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P2860
FTY720 suppresses CD4+CD44highCD62L- effector memory T cell-mediated colitis.
description
2006 nî lūn-bûn
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2006年の論文
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2006年学术文章
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2006年学术文章
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name
FTY720 suppresses CD4+CD44highCD62L- effector memory T cell-mediated colitis.
@en
FTY720 suppresses CD4+CD44highCD62L- effector memory T cell-mediated colitis.
@nl
type
label
FTY720 suppresses CD4+CD44highCD62L- effector memory T cell-mediated colitis.
@en
FTY720 suppresses CD4+CD44highCD62L- effector memory T cell-mediated colitis.
@nl
prefLabel
FTY720 suppresses CD4+CD44highCD62L- effector memory T cell-mediated colitis.
@en
FTY720 suppresses CD4+CD44highCD62L- effector memory T cell-mediated colitis.
@nl
P2093
P2860
P356
P1476
FTY720 suppresses CD4+CD44highCD62L- effector memory T cell-mediated colitis.
@en
P2093
Tsuchiya K
Watanabe M
P2860
P304
P356
10.1152/AJPGI.00496.2005
P577
2006-03-30T00:00:00Z