Bcl-2 expression delays mammary tumor development in dimethylbenz(a)anthracene-treated transgenic mice.
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BAD/BCL-[X(L)] heterodimerization leads to bypass of G0/G1 arrestDeletion of Bid impedes cell proliferation and hepatic carcinogenesisp27 deficiency cooperates with Bcl-2 but not Bax to promote T-cell lymphoma.WAP-TAg transgenic mice and the study of dysregulated cell survival, proliferation, and mutation during breast carcinogenesis.Bcl-2 retards cell cycle entry through p27(Kip1), pRB relative p130, and altered E2F regulation.BCL2 interaction with actin in vitro may inhibit cell motility by enhancing actin polymerization.MTA1--a stress response protein: a master regulator of gene expression and cancer cell behavior.Bcl-2 expression inhibits liver carcinogenesis and delays the development of proliferating foci.Apoptosis in normal and diseased oral tissues.c-Myc induction of programmed cell death may contribute to carcinogenesis: a perspective inspired by several concepts of chemical carcinogenesis.Loss of BCL-2 in the progression of oral cancer is not attributable to mutations.Targeting BCL-2 to enhance vulnerability to therapy in estrogen receptor-positive breast cancer.Upregulation of SRF Is Associated With Hypoxic Pulmonary Hypertension by Promoting Viability of Smooth Muscle Cells via Increasing Expression of Bcl-2.Bcl-xL/Bcl-2 coordinately regulates apoptosis, cell cycle arrest and cell cycle entry.G0 function of BCL2 and BCL-xL requires BAX, BAK, and p27 phosphorylation by Mirk, revealing a novel role of BAX and BAK in quiescence regulation.Bcl-2 activates a programme of premature senescence in human carcinoma cells.Resistance to apoptosis should not be taken as a hallmark of cancer.Bcl-2 delays cell cycle through mitochondrial ATP and ROS.Apoptosis drives cancer cells proliferate and metastasize.Lymphoma development in Bax transgenic mice is inhibited by Bcl-2 and associated with chromosomal instability.PGC-1α coordinates with Bcl-2 to control the cell cycle in U251 cells through reducing ROS* #.
P2860
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P2860
Bcl-2 expression delays mammary tumor development in dimethylbenz(a)anthracene-treated transgenic mice.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh
1999年學術文章
@zh-hant
name
Bcl-2 expression delays mammary tumor development in dimethylbenz
@nl
Bcl-2 expression delays mammar ...... acene-treated transgenic mice.
@en
type
label
Bcl-2 expression delays mammary tumor development in dimethylbenz
@nl
Bcl-2 expression delays mammar ...... acene-treated transgenic mice.
@en
prefLabel
Bcl-2 expression delays mammary tumor development in dimethylbenz
@nl
Bcl-2 expression delays mammar ...... acene-treated transgenic mice.
@en
P2093
P2860
P356
P1433
P1476
Bcl-2 expression delays mammar ...... acene-treated transgenic mice.
@en
P2093
P2860
P2888
P304
P356
10.1038/SJ.ONC.1203099
P407
P577
1999-11-01T00:00:00Z