PFKFB4 controls embryonic patterning via Akt signalling independently of glycolysis.
about
Loss of PFKFB4 induces cell death in mitotically arrested ovarian cancer cellsA chemical screen in zebrafish embryonic cells establishes that Akt activation is required for neural crest development.A molecular atlas of the developing ectoderm defines neural, neural crest, placode, and nonneural progenitor identity in vertebrates.PFKFB4 control of AKT signaling is essential for premigratory and migratory neural crest formation.FGF mediated MAPK and PI3K/Akt Signals make distinct contributions to pluripotency and the establishment of Neural Crest.Copy number variation as a genetic basis for heterotaxy and heterotaxy-spectrum congenital heart defects.Derivation of neural crest stem cells from human epidermal keratinocytes requires FGF-2, IGF-1, and inhibition of TGF-β1Alpha-enolase regulates the malignant phenotype of pulmonary artery smooth muscle cells via the AMPK-Akt pathway
P2860
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P2860
PFKFB4 controls embryonic patterning via Akt signalling independently of glycolysis.
description
2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年學術文章
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2015年學術文章
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2015年學術文章
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name
PFKFB4 controls embryonic patterning via Akt signalling independently of glycolysis.
@en
PFKFB4 controls embryonic patterning via Akt signalling independently of glycolysis.
@nl
type
label
PFKFB4 controls embryonic patterning via Akt signalling independently of glycolysis.
@en
PFKFB4 controls embryonic patterning via Akt signalling independently of glycolysis.
@nl
prefLabel
PFKFB4 controls embryonic patterning via Akt signalling independently of glycolysis.
@en
PFKFB4 controls embryonic patterning via Akt signalling independently of glycolysis.
@nl
P2093
P2860
P356
P1476
PFKFB4 controls embryonic patterning via Akt signalling independently of glycolysis
@en
P2093
Anne H Monsoro-Burq
Caterina Pegoraro
Celio Pouponnot
Frédérique Maczkowiak
P2860
P2888
P356
10.1038/NCOMMS6953
P407
P577
2015-01-20T00:00:00Z