Novel blocker of NFAT activation inhibits IL-6 production in human myometrial arteries and reduces vascular smooth muscle cell proliferation - Wikidata - awgldk - TriplyDB

Novel blocker of NFAT activation inhibits IL-6 production in human myometrial arteries and reduces vascular smooth muscle cell proliferation

Novel blocker of NFAT activation inhibits IL-6 production in human myometrial arteries and reduces vascular smooth muscle cell proliferation

http://www.wikidata.org/entity/Q55919537

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NFAT regulates the expression of AIF-1 and IRT-1: yin and yang splice variants of neointima formation and atherosclerosis Nuclear factor of activated T cells - a transcription factor family as critical regulator in lung and colon cancer Temporal network based analysis of cell specific vein graft transcriptome defines key pathways and hub genes in implantation injury Inhibition of nuclear factor of activated T-cells (NFAT) suppresses accelerated atherosclerosis in diabetic mice The role of calcineurin/NFAT in SFRP2 induced angiogenesis--a rationale for breast cancer treatment with the calcineurin inhibitor tacrolimus Secreted frizzle-related protein 2 stimulates angiogenesis via a calcineurin/NFAT signaling pathway NFATc3 mediates chronic hypoxia-induced pulmonary arterial remodeling with alpha-actin up-regulation Genome-wide microarray analyses identify the protein C receptor as a novel calcineurin/nuclear factor of activated T cells-dependent gene in vascular smooth muscle cell phenotypic modulation.Cyclosporine up-regulates Krüppel-like factor-4 (KLF4) in vascular smooth muscle cells and drives phenotypic modulation in vivo.Nuclear factor of activated T cells regulates osteopontin expression in arterial smooth muscle in response to diabetes-induced hyperglycemia.Integrative genomics identifies DSCR1 (RCAN1) as a novel NFAT-dependent mediator of phenotypic modulation in vascular smooth muscle cells.Complex regulation and function of the inflammatory smooth muscle cell phenotype in atherosclerosis Molecular mechanisms of collagen isotype-specific modulation of smooth muscle cell phenotype Calcineurin/nuclear factor of activated T cells-coupled vanilliod transient receptor potential channel 4 ca2+ sparklets stimulate airway smooth muscle cell proliferation NFATc3 contributes to intermittent hypoxia-induced arterial remodeling in mice Simvastatin stimulates production of the antiapoptotic protein Bcl-2 via endothelin-1 and NFATc3 in SH-SY5Y cells.Systematic identification of core transcription factors mediating dysregulated links bridging inflammatory bowel diseases and colorectal cancer Endothelin-1 contributes to increased NFATc3 activation by chronic hypoxia in pulmonary arteries.RNA-Seq reveals a role for NFAT-signaling in human retinal microvascular endothelial cells treated with TNFα.The role of calcineurin signaling in microcystin-LR triggered neuronal toxicity.Nuclear factor of activated T-cell isoform expression and regulation in human myometrium Band-pass processing in a GPCR signaling pathway selects for NFAT transcription factor activation Radiation damage and radioprotectants: new concepts in the era of molecular medicine Oxytocin-stimulated NFAT transcriptional activation in human myometrial cells.Calcineurin in a Crowded World.TRPC6 mutations associated with focal segmental glomerulosclerosis cause constitutive activation of NFAT-dependent transcription.Gax regulates human vascular smooth muscle cell phenotypic modulation and vascular remodeling Mechanisms of intermittent hypoxia induced hypertension.Novel inhibitors of the calcineurin/NFATc hub - alternatives to CsA and FK506?Potential cross-talk between (pro)renin receptors and Wnt/frizzled receptors in cardiovascular and renal disorders.Intracellular Ca²⁺ signalling and phenotype of vascular smooth muscle cells.Vascular smooth muscle cell phenotype is defined by Ca2+-dependent transcription factors.Targeting angiogenesis and the tumor microenvironment.RNA interference targeting STIM1 suppresses vascular smooth muscle cell proliferation and neointima formation in the rat.Nuclear factor of activated T cells is activated in the endothelium of retinal microvessels in diabetic mice.Inhibition of SOC/Ca2+/NFAT pathway is involved in the anti-proliferative effect of sildenafil on pulmonary artery smooth muscle cells.Streptococcal M1 protein triggers chemokine formation, neutrophil infiltration, and lung injury in an NFAT-dependent manner.Nuclear factor of activated T cells regulates neutrophil recruitment, systemic inflammation, and T-cell dysfunction in abdominal sepsis.RAGE-dependent activation of the oncoprotein Pim1 plays a critical role in systemic vascular remodeling processes.NFATc3 is required for intermittent hypoxia-induced hypertension.

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Novel blocker of NFAT activation inhibits IL-6 production in human myometrial arteries and reduces vascular smooth muscle cell proliferation

http://www.wikidata.org/entity/Q55919537

description

article

@en

im November 2006 veröffentlichter wissenschaftlicher Artikel

@de

wetenschappelijk artikel

@nl

наукова стаття, опублікована в березні 2007

@uk

ലേഖനം

@ml

name

Novel blocker of NFAT activati ...... ooth muscle cell proliferation

@en

Novel blocker of NFAT activati ...... ooth muscle cell proliferation

@nl

type

label

Novel blocker of NFAT activati ...... ooth muscle cell proliferation

@en

Novel blocker of NFAT activati ...... ooth muscle cell proliferation

@nl

prefLabel

Novel blocker of NFAT activati ...... ooth muscle cell proliferation

@en

Novel blocker of NFAT activati ...... ooth muscle cell proliferation

@nl

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AJPCELL.00590.2005

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American Journal of Physiology - Cell Physiology

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Novel blocker of NFAT activati ...... ooth muscle cell proliferation

@en

P2093

Dag Wide-Swensson

http://www.w3.org/2001/XMLSchema#string

Ina Nordström

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Jenny Nilsson

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Lisa M Nilsson

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Marie-Louise Lydrup

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Per Hellstrand

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Yung-Wu Chen

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Zheng-Wu Sun

http://www.w3.org/2001/XMLSchema#string

P2860

Calcineurin-GATA-6 pathway is involved in smooth muscle-specific transcription.

Generic signals and specific outcomes: signaling through Ca2+, calcineurin, and NF-AT

Transcription factors of the NFAT family: regulation and function

NFATc3 and NFATc4 are required for cardiac development and mitochondrial function

Molecular regulation of vascular smooth muscle cell differentiation in development and disease

Calcineurin/NFAT coupling participates in pathological, but not physiological, cardiac hypertrophy.

Calcineurin signaling and NFAT activation in cardiovascular and skeletal muscle development.

Myocardin and ternary complex factors compete for SRF to control smooth muscle gene expression.

Serum response factor: toggling between disparate programs of gene expression.

Alteration in temporal kinetics of Ca2+ signaling and control of growth and proliferation.

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C1167-78

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scholarly article

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10.1152/AJPCELL.00590.2005

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3

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292

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Jeffery D. Molkentin

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2006-11-01T00:00:00Z

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17079331

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