Human metapneumovirus M2-2 protein inhibits innate cellular signaling by targeting MAVS
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Recent vaccine development for human metapneumovirusSARS-coronavirus open reading frame-9b suppresses innate immunity by targeting mitochondria and the MAVS/TRAF3/TRAF6 signalosome.Human metapneumovirus infection induces significant changes in small noncoding RNA expression in airway epithelial cells.Viral and host factors determine innate immune responses in airway epithelial cells from children with wheeze and atopy.Human metapneumovirus M2-2 protein inhibits innate immune response in monocyte-derived dendritic cellsMolecular comparisons of full length metapneumovirus (MPV) genomes, including newly determined French AMPV-C and -D isolates, further supports possible subclassification within the MPV Genus.Effect of in vitro syncytium formation on the severity of human metapneumovirus disease in a murine modelMitochondrial antiviral-signalling protein plays an essential role in host immunity against human metapneumovirusRespiratory Syncytial Virus Utilizes a tRNA Fragment to Suppress Antiviral Responses Through a Novel Targeting Mechanism.Human metapneumovirus small hydrophobic (SH) protein downregulates type I IFN pathway signaling by affecting STAT1 expression and phosphorylationMyD88 controls human metapneumovirus-induced pulmonary immune responses and disease pathogenesis.Human metapneumovirus antagonism of innate immune responses.Host-Viral Interactions: Role of Pattern Recognition Receptors (PRRs) in Human Pneumovirus InfectionsFunctional motifs responsible for human metapneumovirus M2-2-mediated innate immune evasion.Modulation of Host Immunity by the Human Metapneumovirus.Aberrant T cell immunity triggered by human Respiratory Syncytial Virus and human Metapneumovirus infection.Human Metapneumovirus M2-2 Protein Acts as a Negative Regulator of Alpha Interferon Production by Plasmacytoid Dendritic Cells.Highly Pathogenic Porcine Reproductive and Respiratory Syndrome Virus Nsp4 Cleaves VISA to Impair Antiviral Responses Mediated by RIG-I-like Receptors.Detection of Nuclear Protein Profile Changes by Human Metapneumovirus M2-2 Protein Using Quantitative Differential Proteomics.Human metapneumovirus - what we know now.Human Metapneumovirus: Mechanisms and Molecular Targets Used by the Virus to Avoid the Immune SystemInterferon-Mediated Response to Human Metapneumovirus Infection
P2860
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P2860
Human metapneumovirus M2-2 protein inhibits innate cellular signaling by targeting MAVS
description
2012 nî lūn-bûn
@nan
2012 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Human metapneumovirus M2-2 protein inhibits innate cellular signaling by targeting MAVS
@ast
Human metapneumovirus M2-2 protein inhibits innate cellular signaling by targeting MAVS
@en
Human metapneumovirus M2-2 protein inhibits innate cellular signaling by targeting MAVS
@nl
type
label
Human metapneumovirus M2-2 protein inhibits innate cellular signaling by targeting MAVS
@ast
Human metapneumovirus M2-2 protein inhibits innate cellular signaling by targeting MAVS
@en
Human metapneumovirus M2-2 protein inhibits innate cellular signaling by targeting MAVS
@nl
prefLabel
Human metapneumovirus M2-2 protein inhibits innate cellular signaling by targeting MAVS
@ast
Human metapneumovirus M2-2 protein inhibits innate cellular signaling by targeting MAVS
@en
Human metapneumovirus M2-2 protein inhibits innate cellular signaling by targeting MAVS
@nl
P2093
P2860
P3181
P356
P1433
P1476
Human metapneumovirus M2-2 protein inhibits innate cellular signaling by targeting MAVS
@en
P2093
Chien-Te K Tseng
Deborah J Prusak
Deepthi Kolli
Junping Ren
Qingrong Wang
Thomas G Wood
Xiaoyong Bao
P2860
P304
13049-13061
P3181
P356
10.1128/JVI.01248-12
P407
P577
2012-09-26T00:00:00Z