DNA polymerase θ (POLQ), double-strand break repair, and cancer
about
Remodeling and Control of Homologous Recombination by DNA Helicases and Translocases that Target Recombinases and SynapsisDrugging the Cancers Addicted to DNA Repair.Drosophila DNA polymerase theta utilizes both helicase-like and polymerase domains during microhomology-mediated end joining and interstrand crosslink repairMultiple mechanisms contribute to double-strand break repair at rereplication forks in Drosophila follicle cells.A prosurvival DNA damage-induced cytoplasmic interferon response is mediated by end resection factors and is limited by Trex1.DNA Repair in Drosophila: Mutagens, Models, and Missing Genes.Dual loss of human POLQ and LIG4 abolishes random integration.Modification of 3' Terminal Ends of DNA and RNA Using DNA Polymerase θ Terminal Transferase Activity.Expression and Structural Analyses of Human DNA Polymerase θ (POLQ).Polθ helicase: drive or reverse.Studies of lncRNAs in DNA double strand break repair: what is new?Secondary structure forming sequences drive SD-MMEJ repair of DNA double-strand breaks.Family A and B DNA Polymerases in Cancer: Opportunities for Therapeutic Interventions.Mutational signatures of non-homologous and polymerase theta-mediated end-joining in embryonic stem cells.DNA polymerase beta participates in DNA End-joining.The helicase domain of Polθ counteracts RPA to promote alt-NHEJ.The A-Rule and Deletion Formation During Abasic and Oxidized Abasic Site Bypass by DNA Polymerase θ.p53 orchestrates DNA replication restart homeostasis by suppressing mutagenic RAD52 and POLθ pathways.DNA double-strand break response factors influence end-joining features of IgH class switch and general translocation junctions.Translesion and Repair DNA Polymerases: Diverse Structure and Mechanism.SIRF: Quantitative in situ analysis of protein interactions at DNA replication forks.DNA polymerase ν gene expression influences fludarabine resistance in chronic lymphocytic leukemia independently from p53 status.Persistent 3'-phosphate termini and increased cytotoxicity of radiomimetic DNA double-strand breaks in cells lacking polynucleotide kinase/phosphatase despite presence of an alternative 3'-phosphatase.Polymerase θ-helicase efficiently unwinds DNA and RNA-DNA hybrids
P2860
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P2860
DNA polymerase θ (POLQ), double-strand break repair, and cancer
description
2016 nî lūn-bûn
@nan
2016 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2016 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
name
DNA polymerase θ (POLQ), double-strand break repair, and cancer
@ast
DNA polymerase θ (POLQ), double-strand break repair, and cancer
@en
DNA polymerase θ (POLQ), double-strand break repair, and cancer
@nl
type
label
DNA polymerase θ (POLQ), double-strand break repair, and cancer
@ast
DNA polymerase θ (POLQ), double-strand break repair, and cancer
@en
DNA polymerase θ (POLQ), double-strand break repair, and cancer
@nl
prefLabel
DNA polymerase θ (POLQ), double-strand break repair, and cancer
@ast
DNA polymerase θ (POLQ), double-strand break repair, and cancer
@en
DNA polymerase θ (POLQ), double-strand break repair, and cancer
@nl
P2860
P3181
P1433
P1476
DNA polymerase θ (POLQ), double-strand break repair, and cancer
@en
P2093
Richard D. Wood
P2860
P3181
P356
10.1016/J.DNAREP.2016.05.003
P407
P577
2016-05-14T00:00:00Z