The complexity of phosphorylated H2AX foci formation and DNA repair assembly at DNA double-strand breaks.
about
Feedback between p21 and reactive oxygen production is necessary for cell senescenceUbiquitin-activating enzyme UBA1 is required for cellular response to DNA damageVaccinia-related kinase 1 (VRK1) is an upstream nucleosomal kinase required for the assembly of 53BP1 foci in response to ionizing radiation-induced DNA damageVRK1 chromatin kinase phosphorylates H2AX and is required for foci formation induced by DNA damageDynamics of DNA damage response proteins at DNA breaks: a focus on protein modificationsDNA damage signaling assessed in individual cells in relation to the cell cycle phase and induction of apoptosisUse of the γ-H2AX assay to monitor DNA damage and repair in translational cancer researchDNA double-strand breaks in heterochromatin elicit fast repair protein recruitment, histone H2AX phosphorylation and relocation to euchromatin.Types, causes, detection and repair of DNA fragmentation in animal and human sperm cellsImaging the DNA damage response with PET and SPECTAnalysis of individual molecular events of DNA damage response by flow- and image-assisted cytometryCdk1 uncouples CtIP-dependent resection and Rad51 filament formation during M-phase double-strand break repairActivation of DNA damage response signaling by condensed chromatin.Give me a break, but not in mitosis: the mitotic DNA damage response marks DNA double-strand breaks with early signaling eventsMolecular and cellular pharmacology of the novel noncamptothecin topoisomerase I inhibitor Genz-644282.Assessing 'radiosensitivity' with kinetic profiles of γ-H2AX, 53BP1 and BRCA1 foci.Schedule-dependent synergy of histone deacetylase inhibitors with DNA damaging agents in small cell lung cancerStreptococcus pneumoniae secretes hydrogen peroxide leading to DNA damage and apoptosis in lung cells.Nucleoporin NUP153 guards genome integrity by promoting nuclear import of 53BP1DNA Damage Response Assessments in Human Tumor Samples Provide Functional Biomarkers of Radiosensitivity.Chemical proteomics reveals a γH2AX-53BP1 interaction in the DNA damage responseATM Activation and H2AX Phosphorylation Induced by Genotoxic Agents Assessed by Flow- and Laser Scanning Cytometry.An autonomous chromatin/DNA-PK mechanism for localized DNA damage signaling in mammalian cells.Nuclear envelope rupture and repair during cancer cell migration.Analogs of the novel phytohormone, strigolactone, trigger apoptosis and synergize with PARP inhibitors by inducing DNA damage and inhibiting DNA repair.Chromatin response to DNA double-strand break damage.Reading, writing, and repair: the role of ubiquitin and the ubiquitin-like proteins in DNA damage signaling and repair.Chromatin regulation of DNA damage repair and genome integrity in the central nervous system.Part I-mechanism of adaptation: high nitric oxide adapted A549 cells show enhanced DNA damage response and activation of antiapoptotic pathways.Recruitment of cyclin G2 to promyelocytic leukemia nuclear bodies promotes dephosphorylation of γH2AX following treatment with ionizing radiation.Regulation of DNA damage following termination of Hedgehog (HH) survival signaling at the level of the GLI genes in human colon cancerDiscordance between phosphorylation and recruitment of 53BP1 in response to DNA double-strand breaks.The human WRN and BLM RecQ helicases differentially regulate cell proliferation and survival after chemotherapeutic DNA damage.Induction of DNA strand breaks by dental composite components compared to X-ray exposure in human gingival fibroblasts.DNA damage response induced by exposure of human lung adenocarcinoma cells to smoke from tobacco- and nicotine-free cigarettes.Ape1 guides DNA repair pathway choice that is associated with drug tolerance in glioblastomaThe Causal Relationship between DNA Damage Induction in Bovine Lymphocytes and the Fukushima Nuclear Power Plant Accident.Requirement for DNA ligase IV during embryonic neuronal development.Conversations between chromatin modifications and DNA double strand break repair: a commentary.The Histone Deacetylase Inhibitor Valproic Acid Exerts a Synergistic Cytotoxicity with the DNA-Damaging Drug Ellipticine in Neuroblastoma Cells.
P2860
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P2860
The complexity of phosphorylated H2AX foci formation and DNA repair assembly at DNA double-strand breaks.
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2010 nî lūn-bûn
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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2010年の論文
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2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
The complexity of phosphorylat ...... y at DNA double-strand breaks.
@ast
The complexity of phosphorylat ...... y at DNA double-strand breaks.
@en
The complexity of phosphorylat ...... y at DNA double-strand breaks.
@nl
type
label
The complexity of phosphorylat ...... y at DNA double-strand breaks.
@ast
The complexity of phosphorylat ...... y at DNA double-strand breaks.
@en
The complexity of phosphorylat ...... y at DNA double-strand breaks.
@nl
prefLabel
The complexity of phosphorylat ...... y at DNA double-strand breaks.
@ast
The complexity of phosphorylat ...... y at DNA double-strand breaks.
@en
The complexity of phosphorylat ...... y at DNA double-strand breaks.
@nl
P2093
P2860
P356
P1433
P1476
The complexity of phosphorylat ...... y at DNA double-strand breaks.
@en
P2093
Asako J Nakamura
V Ashutosh Rao
William M Bonner
P2860
P304
P356
10.4161/CC.9.2.10475
P50
P577
2010-01-29T00:00:00Z