AKT kinase activity is required for lithium to modulate mood-related behaviors in mice.
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Lithium: the pharmacodynamic actions of the amazing ionReview of pharmacological treatment in mood disorders and future directions for drug development.Dopamine receptors - IUPHAR Review 13A selective HDAC 1/2 inhibitor modulates chromatin and gene expression in brain and alters mouse behavior in two mood-related testsAdvancing drug discovery for schizophrenia.Glycogen synthase kinase-3 in the etiology and treatment of mood disorders.Neuroprotective effects of lithium: implications for the treatment of Alzheimer's disease and related neurodegenerative disordersA new avenue for lithium: intervention in traumatic brain injury.Role of the axonal initial segment in psychiatric disorders: function, dysfunction, and interventionA high-throughput screen for Wnt/β-catenin signaling pathway modulators in human iPSC-derived neural progenitors.Role of Beta-arrestin 2 downstream of dopamine receptors in the Basal Ganglia.Beyond cAMP: The Regulation of Akt and GSK3 by Dopamine ReceptorsIdentifying a kinase network regulating FGF14:Nav1.6 complex assembly using split-luciferase complementation.Characterization of bipolar disorder patient-specific induced pluripotent stem cells from a family reveals neurodevelopmental and mRNA expression abnormalities.Lithium ions attenuate serum-deprivation-induced apoptosis in PC12 cells through regulation of the Akt/FoxO1 signaling pathways.Molecular Signatures of Psychosocial Stress and Cognition Are Modulated by Chronic Lithium Treatment.A composite peripheral blood gene expression measure as a potential diagnostic biomarker in bipolar disorder.Lithium increases proliferation of hippocampal neural stem/progenitor cells and rescues irradiation-induced cell cycle arrest in vitro.Lithium and neuroprotection: translational evidence and implications for the treatment of neuropsychiatric disorders.STAT3-mediated coincidence detection regulates noncanonical immediate early gene induction.Translation: screening for novel therapeutics with disease-relevant cell types derived from human stem cell models.Search for common targets of lithium and valproic acid identifies novel epigenetic effects of lithium on the rat leptin receptor gene.Neuroglialpharmacology: myelination as a shared mechanism of action of psychotropic treatments.GSK-3 and Wnt Signaling in Neurogenesis and Bipolar Disorder.Lithium's role in neural plasticity and its implications for mood disordersThe emerging role of dopamine-glutamate interaction and of the postsynaptic density in bipolar disorder pathophysiology: Implications for treatment.New Repeat Polymorphism in the AKT1 Gene Predicts Striatal Dopamine D2/D3 Receptor Availability and Stimulant-Induced Dopamine Release in the Healthy Human Brain.Genetic Deletion of Akt3 Induces an Endophenotype Reminiscent of Psychiatric Manifestations in Mice.Fine-Tuning of the RIG-I-Like Receptor/Interferon Regulatory Factor 3-Dependent Antiviral Innate Immune Response by the Glycogen Synthase Kinase 3/β-Catenin PathwayInhibition of GSK3 by lithium, from single molecules to signaling networks.A rare schizophrenia risk variant of CACNA1I disrupts CaV3.3 channel activity.Integrated transcriptome and methylome analysis in youth at high risk for bipolar disorder: a preliminary analysisLithium Reversibly Inhibits Schwann Cell Proliferation and Differentiation Without Inducing Myelin Loss.An emerging role for Wnt and GSK3 signaling pathways in schizophrenia.A Role for Phosphodiesterase 11A (PDE11A) in the Formation of Social Memories and the Stabilization of Mood.Lithium and bipolar disorder: Impacts from molecular to behavioural circadian rhythms.Ketamine up-regulates a cluster of intronic miRNAs within the serotonin receptor 2C gene by inhibiting glycogen synthase kinase-3.Development of [18F]Maleimide-Based Glycogen Synthase Kinase-3β Ligands for Positron Emission Tomography Imaging.Predicting adverse drug reaction profiles by integrating protein interaction networks with drug structures.Lithium-associated transcriptional regulation of CRMP1 in patient-derived olfactory neurons and symptom changes in bipolar disorder.
P2860
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P2860
AKT kinase activity is required for lithium to modulate mood-related behaviors in mice.
description
2011 nî lūn-bûn
@nan
2011 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մարտին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
AKT kinase activity is required for lithium to modulate mood-related behaviors in mice.
@ast
AKT kinase activity is required for lithium to modulate mood-related behaviors in mice.
@en
AKT kinase activity is required for lithium to modulate mood-related behaviors in mice.
@nl
type
label
AKT kinase activity is required for lithium to modulate mood-related behaviors in mice.
@ast
AKT kinase activity is required for lithium to modulate mood-related behaviors in mice.
@en
AKT kinase activity is required for lithium to modulate mood-related behaviors in mice.
@nl
prefLabel
AKT kinase activity is required for lithium to modulate mood-related behaviors in mice.
@ast
AKT kinase activity is required for lithium to modulate mood-related behaviors in mice.
@en
AKT kinase activity is required for lithium to modulate mood-related behaviors in mice.
@nl
P2093
P2860
P356
P1476
AKT kinase activity is required for lithium to modulate mood-related behaviors in mice
@en
P2093
Edward B Holson
Edward M Scolnick
Elizabeth L Clore
Erin Berry-Scott
Florence F Wagner
Josh K Ketterman
Keenan R Richards
Michael C Lewis
Misha Riley
P2860
P2888
P304
P356
10.1038/NPP.2011.24
P407
P577
2011-03-09T00:00:00Z
P5875
P6179
1053330359