Complex relationship between mismatch repair proteins and MBD4 during immunoglobulin class switch recombination - Test2 - elhamkhani - TriplyDB

Complex relationship between mismatch repair proteins and MBD4 during immunoglobulin class switch recombination

Complex relationship between mismatch repair proteins and MBD4 during immunoglobulin class switch recombination

http://www.wikidata.org/entity/Q35036377

about

Involvement of MBD4 inactivation in mismatch repair-deficient tumorigenesis Inter-individual variation in DNA repair capacity: a need for multi-pathway functional assays to promote translational DNA repair research Serine protease inhibition attenuates rIL-12-induced GZMA activity and proinflammatory events by modulating the Th2 profile from estrogen-treated mice.Hydroxymethylated cytosines are associated with elevated C to G transversion rates.Antibody diversification caused by disrupted mismatch repair and promiscuous DNA polymerases MBD4 Facilitates Immunoglobulin Class Switch Recombination.The Mbd4 DNA glycosylase protects mice from inflammation-driven colon cancer and tissue injury.Production of truncated MBD4 protein by frameshift mutation in DNA mismatch repair-deficient cells enhances 5-fluorouracil sensitivity that is independent of hMLH1 status.

P2860

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P2860

Complex relationship between mismatch repair proteins and MBD4 during immunoglobulin class switch recombination

http://www.wikidata.org/entity/Q35036377

description

2013 nî lūn-bûn

@nan

2013 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2013年の論文

@ja

2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

name

Complex relationship between m ...... lin class switch recombination

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Complex relationship between m ...... lin class switch recombination

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type

label

Complex relationship between m ...... lin class switch recombination

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Complex relationship between m ...... lin class switch recombination

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prefLabel

Complex relationship between m ...... lin class switch recombination

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Complex relationship between m ...... lin class switch recombination

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JOURNAL.PONE.0078370

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Complex relationship between m ...... lin class switch recombination

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Alfonso Bellacosa

http://www.w3.org/2001/XMLSchema#string

Amy L Kenter

http://www.w3.org/2001/XMLSchema#string

Fernando Grigera

http://www.w3.org/2001/XMLSchema#string

P2860

Genomic structure and chromosomal mapping of the murine and human Mbd1, Mbd2, Mbd3, and Mbd4 genes

MED1, a novel human methyl-CpG-binding endonuclease, interacts with DNA mismatch repair protein MLH1

The base excision repair enzyme MED1 mediates DNA damage response to antitumor drugs and is associated with mismatch repair system integrity

The FANCJ/MutLalpha interaction is required for correction of the cross-link response in FA-J cells

Excision of thymine and 5-hydroxymethyluracil by the MBD4 DNA glycosylase domain: structural basis and implications for active DNA demethylation

AID targeting is dependent on RNA polymerase II pausing.

IgH class switching and translocations use a robust non-classical end-joining pathway

Involvement of mouse Mlh1 in DNA mismatch repair and meiotic crossing over

Somatic Apc mutations are selected upon their capacity to inactivate the beta-catenin downregulating activity

Specific expression of activation-induced cytidine deaminase (AID), a novel member of the RNA-editing deaminase family in germinal center B cells

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scholarly article

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10.1371/JOURNAL.PONE.0078370

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