Severe acute respiratory syndrome coronavirus protein 6 accelerates murine coronavirus infections.
about
The role of severe acute respiratory syndrome (SARS)-coronavirus accessory proteins in virus pathogenesisCoronavirus gene 7 counteracts host defenses and modulates virus virulenceSevere acute respiratory syndrome coronavirus as an agent of emerging and reemerging infectionPathogenicity of severe acute respiratory coronavirus deletion mutants in hACE-2 transgenic mice.The N-terminal region of severe acute respiratory syndrome coronavirus protein 6 induces membrane rearrangement and enhances virus replication.Mechanisms of severe acute respiratory syndrome pathogenesis and innate immunomodulation.The SARS coronavirus papain like protease can inhibit IRF3 at a post activation step that requires deubiquitination activity.Severe acute respiratory syndrome coronavirus accessory protein 6 is a virion-associated protein and is released from 6 protein-expressing cells.Severe Acute Respiratory Syndrome Coronavirus ORF7a Inhibits Bone Marrow Stromal Antigen 2 Virion Tethering through a Novel Mechanism of Glycosylation Interference.A review of genetic methods and models for analysis of coronavirus-induced severe pneumonitis.Severe acute respiratory syndrome coronavirus protein 6 accelerates murine hepatitis virus infections by more than one mechanism.Release of severe acute respiratory syndrome coronavirus nuclear import block enhances host transcription in human lung cells.SARS coronavirus accessory proteins.Severe acute respiratory syndrome coronavirus protein 6 is required for optimal replication.Alphacoronavirus protein 7 modulates host innate immune response.SARS-coronavirus protein 6 conformations required to impede protein import into the nucleus.Enhancement of murine coronavirus replication by severe acute respiratory syndrome coronavirus protein 6 requires the N-terminal hydrophobic region but not C-terminal sorting motifs.Severe acute respiratory syndrome coronavirus ORF6 antagonizes STAT1 function by sequestering nuclear import factors on the rough endoplasmic reticulum/Golgi membrane.
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Severe acute respiratory syndrome coronavirus protein 6 accelerates murine coronavirus infections.
description
2006 nî lūn-bûn
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2006年の論文
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2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Severe acute respiratory syndr ...... murine coronavirus infections.
@ast
Severe acute respiratory syndr ...... murine coronavirus infections.
@en
type
label
Severe acute respiratory syndr ...... murine coronavirus infections.
@ast
Severe acute respiratory syndr ...... murine coronavirus infections.
@en
prefLabel
Severe acute respiratory syndr ...... murine coronavirus infections.
@ast
Severe acute respiratory syndr ...... murine coronavirus infections.
@en
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Severe acute respiratory syndr ...... murine coronavirus infections.
@en
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Chandra Tangudu
Heidi Olivares
Jason Netland
Thomas Gallagher
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10.1128/JVI.01515-06
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2006-11-15T00:00:00Z