Dicer-dependent pathways regulate chondrocyte proliferation and differentiation - Test2 - elhamkhani - TriplyDB

Dicer-dependent pathways regulate chondrocyte proliferation and differentiation

Dicer-dependent pathways regulate chondrocyte proliferation and differentiation

http://www.wikidata.org/entity/Q36882995

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MicroRNA-221 regulates chondrogenic differentiation through promoting proteosomal degradation of slug by targeting Mdm2 A microRNA signature for a BMP2-induced osteoblast lineage commitment program miR-199a, a bone morphogenic protein 2-responsive MicroRNA, regulates chondrogenesis via direct targeting to Smad1 Roles of microRNAs in prenatal chondrogenesis, postnatal chondrogenesis and cartilage-related diseases Dicer1 depletion in male germ cells leads to infertility due to cumulative meiotic and spermiogenic defects Roles of microRNA and signaling pathway in osteoarthritis pathogenesis Current Status and Strategy of microRNA Research for Cartilage Development and Osteoarthritis Pathogenesis MicroRNAs regulate bone development and regeneration Bone marrow stroma-derived miRNAs as regulators, biomarkers and therapeutic targets of bone metastasis Matrix-embedded cells control osteoclast formation BMP canonical Smad signaling through Smad1 and Smad5 is required for endochondral bone formation miR-1247 functions by targeting cartilage transcription factor SOX9 The neural crest-enriched microRNA miR-452 regulates epithelial-mesenchymal signaling in the first pharyngeal arch Deletion of Dicer in somatic cells of the female reproductive tract causes sterility.Regulation of the IGFBP-5 and MMP-13 genes by the microRNAs miR-140 and miR-27a in human osteoarthritic chondrocytes.MicroRNA-145 regulates chondrogenic differentiation of mesenchymal stem cells by targeting Sox9.Podocyte-selective deletion of dicer induces proteinuria and glomerulosclerosis.Concerted stimuli regulating osteo-chondral differentiation from stem cells: phenotype acquisition regulated by microRNAs.MicroRNA-140 is expressed in differentiated human articular chondrocytes and modulates interleukin-1 responses Loss of cardiac microRNA-mediated regulation leads to dilated cardiomyopathy and heart failure MicroRNAs and micromanaging the skeleton in disease, development and evolution Altered expression of circulating microRNA in plasma of patients with primary osteoarthritis and in silico analysis of their pathways Dicer inactivation in osteoprogenitor cells compromises fetal survival and bone formation, while excision in differentiated osteoblasts increases bone mass in the adult mouse.MicroRNA-29b Contributes to Collagens Imbalance in Human Osteoarthritic and Dedifferentiated Articular Chondrocytes.p16INK4a and its regulator miR-24 link senescence and chondrocyte terminal differentiation-associated matrix remodeling in osteoarthritis.miR-146a facilitates osteoarthritis by regulating cartilage homeostasis via targeting Camk2d and Ppp3r2 Upregulation of miR-23b enhances the autologous therapeutic potential for degenerative arthritis by targeting PRKACB in synovial fluid-derived mesenchymal stem cells from patients.MicroRNAs and Autophagy: Fine Players in the Control of Chondrocyte Homeostatic Activities in Osteoarthritis.Targeted deletion of Capn4 in cells of the chondrocyte lineage impairs chondrocyte proliferation and differentiation MicroRNA-140 plays dual roles in both cartilage development and homeostasis.MicroRNAs dynamically remodel gastrointestinal smooth muscle cells Dicer ablation in oligodendrocytes provokes neuronal impairment in mice MicroRNA-125b confers the resistance of breast cancer cells to paclitaxel through suppression of pro-apoptotic Bcl-2 antagonist killer 1 (Bak1) expression.Dicer activity in neural crest cells is essential for craniofacial organogenesis and pharyngeal arch artery morphogenesis.Type II collagen expression is regulated by tissue-specific miR-675 in human articular chondrocytes.MicroRNA-1 regulates chondrocyte phenotype by repressing histone deacetylase 4 during growth plate development.miRNAs in circulation: mirroring bone conditions?MiR-365: a mechanosensitive microRNA stimulates chondrocyte differentiation through targeting histone deacetylase 4.The expression and function of microRNAs in chondrogenesis and osteoarthritis.Next-generation sequencing identifies equine cartilage and subchondral bone miRNAs and suggests their involvement in osteochondrosis physiopathology.

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Dicer-dependent pathways regulate chondrocyte proliferation and differentiation

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2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Dicer-dependent pathways regulate chondrocyte proliferation and differentiation

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Dicer-dependent pathways regulate chondrocyte proliferation and differentiation

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Dicer-dependent pathways regulate chondrocyte proliferation and differentiation

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Dicer-dependent pathways regulate chondrocyte proliferation and differentiation

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P2093

Andrew P McMahon

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Bradley S Cobb

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Ernestina Schipani

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Henry M Kronenberg

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Jun Lu

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Stephen J Rodda

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P2860

The RNaseIII enzyme Dicer is required for morphogenesis but not patterning of the vertebrate limb.

mRNA degradation by miRNAs and GW182 requires both CCR4:NOT deadenylase and DCP1:DCP2 decapping complexes

Erralpha and Gabpa/b specify PGC-1alpha-dependent oxidative phosphorylation gene expression that is altered in diabetic muscle

The role of microRNA-1 and microRNA-133 in skeletal muscle proliferation and differentiation

Requirement of bic/microRNA-155 for normal immune function

Disrupting the pairing between let-7 and Hmga2 enhances oncogenic transformation

Cerebellar neurodegeneration in the absence of microRNAs

The tumor suppressor microRNA let-7 represses the HMGA2 oncogene.

Prediction of mammalian microRNA targets

Model-based analysis of oligonucleotide arrays: expression index computation and outlier detection

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1949-1954

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6

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105

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Matthias Merkenschlager

Tatsuya Kobayashi

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2008-01-31T00:00:00Z

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5612838

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18238902

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2538863

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