Analysis of DNA breaks, DNA damage response, and apoptosis produced by high NaCl. - Test2 - elhamkhani - TriplyDB

Analysis of DNA breaks, DNA damage response, and apoptosis produced by high NaCl.

Analysis of DNA breaks, DNA damage response, and apoptosis produced by high NaCl.

http://www.wikidata.org/entity/Q37018485

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The combinatorial nature of osmosensing in fishes Increased cancer risk of augmentation cystoplasty: possible role for hyperosmolal microenvironment on DNA damage recognition Mediator of DNA damage checkpoint 1 (MDC1) contributes to high NaCl-induced activation of the osmoprotective transcription factor TonEBP/OREBP.Recent developments in the use of γ-H2AX as a quantitative DNA double-strand break biomarker.Mre11 is expressed in mammalian mitochondria where it binds to mitochondrial DNA.High salt and DNA double-strand breaks.DNA double-strand breaks induced by high NaCl occur predominantly in gene deserts Osmotic challenge drives rapid and reversible chromatin condensation in chondrocytes Low Dose BCG Infection as a Model for Macrophage Activation Maintaining Cell Viability.NF-κB-dependent upregulation of (pro)renin receptor mediates high-NaCl-induced apoptosis in mouse inner medullary collecting duct cells.

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Analysis of DNA breaks, DNA damage response, and apoptosis produced by high NaCl.

http://www.wikidata.org/entity/Q37018485

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on October 2008

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Analysis of DNA breaks, DNA damage response, and apoptosis produced by high NaCl.

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Analysis of DNA breaks, DNA damage response, and apoptosis produced by high NaCl.

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type

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Analysis of DNA breaks, DNA damage response, and apoptosis produced by high NaCl.

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Analysis of DNA breaks, DNA damage response, and apoptosis produced by high NaCl.

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prefLabel

Analysis of DNA breaks, DNA damage response, and apoptosis produced by high NaCl.

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Analysis of DNA breaks, DNA damage response, and apoptosis produced by high NaCl.

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AJPRENAL.90424.2008

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American Journal of Physiology - Renal Physiology

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Analysis of DNA breaks, DNA damage response, and apoptosis produced by high NaCl.

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Maurice B Burg

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Chk1 is an essential kinase that is regulated by Atr and required for the G(2)/M DNA damage checkpoint

Initiation of DNA fragmentation during apoptosis induces phosphorylation of H2AX histone at serine 139

A critical role for histone H2AX in recruitment of repair factors to nuclear foci after DNA damage

Regulation of DNA double-strand break repair pathway choice

ATR-Chk2 signaling in p53 activation and DNA damage response during cisplatin-induced apoptosis

Focusing on foci: H2AX and the recruitment of DNA-damage response factors

The comet assay: what can it really tell us?

Laser-scanning cytometry: A new instrumentation with many applications.

Detection of caspases activation by fluorochrome-labeled inhibitors: Multiparameter analysis by laser scanning cytometry.

Caspase inhibitors.

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F1678-88

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scholarly article

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10.1152/AJPRENAL.90424.2008

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6

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Natalia I Dmitrieva

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2008-10-01T00:00:00Z

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apoptotic process

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