Shank synaptic scaffold proteins: keys to understanding the pathogenesis of autism and other synaptic disorders.
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Epigenetic Effect of Environmental Factors on Autism Spectrum DisordersDysfunctional cerebellar Purkinje cells contribute to autism-like behaviour in Shank2-deficient mice.Shank3 is localized in axons and presynaptic specializations of developing hippocampal neurons and involved in the modulation of NMDA receptor levels at axon terminalsSHANK proteins: roles at the synapse and in autism spectrum disorder.Investigation of SHANK3 in schizophrenia.The Shank3 Interaction Partner ProSAPiP1 Regulates Postsynaptic SPAR Levels and the Maturation of Dendritic Spines in Hippocampal Neurons.Selective Localization of Shanks to VGLUT1-Positive Excitatory Synapses in the Mouse Hippocampus.Zinc Stabilizes Shank3 at the Postsynaptic Density of Hippocampal Synapses.The Postsynaptic Density: There Is More than Meets the EyeElevated ERK/p90 ribosomal S6 kinase activity underlies audiogenic seizure susceptibility in fragile X mice.A critical role of spinal Shank2 proteins in NMDA-induced pain hypersensitivity.Sex differences in DNA methylation of the cord blood are related to sex-bias psychiatric diseases.Treating the Synapse in Major Psychiatric Disorders: The Role of Postsynaptic Density Network in Dopamine-Glutamate Interplay and Psychopharmacologic Drugs Molecular ActionsNeurophysiology and Regulation of the Balance Between Excitation and Inhibition in Neocortical Circuits.The Synapse as a Central Target for Neurodevelopmental Susceptibility to PesticidesMicroarray analysis of gene expression in the cyclooxygenase knockout mice - a connection to autism spectrum disorder.Aberrant cognitive phenotypes and altered hippocampal BDNF expression related to epigenetic modifications in mice lacking the post-synaptic scaffolding protein SHANK1: Implications for autism spectrum disorder.Deficits in the Proline-Rich Synapse-Associated Shank3 Protein in Multiple Neuropsychiatric Disorders.Epigenetic alterations induced by environmental stress associated with metabolic and neurodevelopmental disorders.CRISPR/Cas9-induced shank3b mutant zebrafish display autism-like behaviors.De novo 11q deletion including SHANK2 in a patient with global developmental delay.Animal Models for Coffin-Lowry Syndrome: RSK2 and Nervous System Dysfunction.Common Ribs of Inhibitory Synaptic Dysfunction in the Umbrella of Neurodevelopmental Disorders.Shank2 Deletion in Parvalbumin Neurons Leads to Moderate Hyperactivity, Enhanced Self-Grooming and Suppressed Seizure Susceptibility in Mice.Distinct Phenotypes of Mouse Models Reflect Neuropsychiatric Spectrum Disorders of Human Patients With VariantsGABA Neuronal Deletion of Exons 14-16 in Mice Suppresses Striatal Excitatory Synaptic Input and Induces Social and Locomotor Abnormalities
P2860
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P2860
Shank synaptic scaffold proteins: keys to understanding the pathogenesis of autism and other synaptic disorders.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Shank synaptic scaffold protei ...... and other synaptic disorders.
@en
type
label
Shank synaptic scaffold protei ...... and other synaptic disorders.
@en
prefLabel
Shank synaptic scaffold protei ...... and other synaptic disorders.
@en
P2093
P2860
P356
P1476
Shank synaptic scaffold protei ...... and other synaptic disorders.
@en
P2093
Adele Mossa
Cinzia Vicidomini
Ilaria Bigi
P2860
P304
P356
10.1111/JNC.13232
P407
P577
2015-07-06T00:00:00Z