Prying into the Prion Hypothesis for Parkinson's Disease. - Test2 - elhamkhani - TriplyDB

Prying into the Prion Hypothesis for Parkinson's Disease.

Prying into the Prion Hypothesis for Parkinson's Disease.

http://www.wikidata.org/entity/Q47665674

about

Mitochondria: A Common Target for Genetic Mutations and Environmental Toxicants in Parkinson's Disease.Hopping from One Cell to Another: Huntington's Disease Propagates.Dementia with Lewy bodies and Parkinson's disease-dementia: current concepts and controversies.Modeling Parkinson's Disease in Drosophila: What Have We Learned for Dominant Traits?Chronic Caffeine Treatment Protects Against α-Synucleinopathy by Reestablishing Autophagy Activity in the Mouse Striatum.Are dementia with Lewy bodies and Parkinson's disease dementia the same disease?Anti-Parkinson Potential of Silymarin: Mechanistic Insight and Therapeutic Standing.Hallmarks of Brain Aging: Adaptive and Pathological Modification by Metabolic States.Testing the Protein Propagation Hypothesis of Parkinson Disease.Amyloid assembly and disassembly LRRK2 kinase regulates α-synuclein propagation via RAB35 phosphorylation Modeling Parkinson's Disease Using Patient-specific Induced Pluripotent Stem Cells Membrane binding, internalization, and sorting of alpha-synuclein in the cell Microglia Response During Parkinson's Disease: Alpha-Synuclein Intervention

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P2860

Prying into the Prion Hypothesis for Parkinson's Disease.

http://www.wikidata.org/entity/Q47665674

description

2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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2017年學術文章

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name

Prying into the Prion Hypothesis for Parkinson's Disease.

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Prying into the Prion Hypothesis for Parkinson's Disease.

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Prying into the Prion Hypothesis for Parkinson's Disease.

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Prying into the Prion Hypothesis for Parkinson's Disease.

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Prying into the Prion Hypothesis for Parkinson's Disease.

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Prying into the Prion Hypothesis for Parkinson's Disease.

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JNEUROSCI.1788-16.2017

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Journal of Neuroscience

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Prying into the Prion Hypothesis for Parkinson's Disease.

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Ronald Melki

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Genetic perspective on the role of the autophagy-lysosome pathway in Parkinson disease

Environmental toxins and Parkinson's disease: what have we learned from pesticide-induced animal models?

The role of oxidative stress in Parkinson's disease

Transfer of host-derived α synuclein to grafted dopaminergic neurons in rat

Heparan sulfate proteoglycans mediate internalization and propagation of specific proteopathic seeds

A paravascular pathway facilitates CSF flow through the brain parenchyma and the clearance of interstitial solutes, including amyloid β.

α-Synuclein propagates from mouse brain to grafted dopaminergic neurons and seeds aggregation in cultured human cells.

Multicenter analysis of glucocerebrosidase mutations in Parkinson's disease.

Inclusion formation and neuronal cell death through neuron-to-neuron transmission of alpha-synuclein

Evidence for α-synuclein prions causing multiple system atrophy in humans with parkinsonism

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9808-9818

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scholarly article

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10.1523/JNEUROSCI.1788-16.2017

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Parkinson's disease

Prion protein family

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