Site-specific DICER and DROSHA RNA products control the DNA-damage response - Wikidata - wikimedia - TriplyDB

Site-specific DICER and DROSHA RNA products control the DNA-damage response

Site-specific DICER and DROSHA RNA products control the DNA-damage response

http://www.wikidata.org/entity/Q28269332

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Lamarckian evolution explains human brain evolution and psychiatric disorders The role of dicer in DNA damage repair Choreographing the Double Strand Break Response: Ubiquitin and SUMO Control of Nuclear Architecture Production of small RNAs by mammalian Dicer Small RNAs: essential regulators of gene expression and defenses against environmental stresses in plants Activation of the DNA Damage Response by RNA Viruses Non-Coding RNA: Sequence-Specific Guide for Chromatin Modification and DNA Damage Signaling Regulation of mammalian transcription and splicing by Nuclear RNAi The expanding world of small RNAs in plants MicroRNAs, cancer and ionizing radiation: Where are we?Noncoding RNAs in DNA repair and genome integrity MicroRNA-independent roles of the RNase III enzymes Drosha and Dicer Single molecule fluorescence approaches shed light on intracellular RNAs Dogma derailed: the many influences of RNA on the genome MicroRNA in radiotherapy: miRage or miRador?Emerging roles of the nucleolus in regulating the DNA damage response: the noncanonical DNA repair enzyme APE1/Ref-1 as a paradigmatical example The roles of microRNAs and siRNAs in mammalian spermatogenesis Serial interactome capture of the human cell nucleus Cellular functions of the microprocessor Systematic screen identifies miRNAs that target RAD51 and RAD51D to enhance chemosensitivity Endogenous Mouse Dicer Is an Exclusively Cytoplasmic Protein Intergenically Spliced Chimeric RNAs in Cancer.PCAT-1, a long noncoding RNA, regulates BRCA2 and controls homologous recombination in cancer.Ago2 facilitates Rad51 recruitment and DNA double-strand break repair by homologous recombination.Chromatin modifications during repair of environmental exposure-induced DNA damage: a potential mechanism for stable epigenetic alterations.DGCR8 Mediates Repair of UV-Induced DNA Damage Independently of RNA Processing.Repair of DNA double-strand breaks by templated nucleotide sequence insertions derived from distant regions of the genome.New perspectives on the diversification of the RNA interference system: insights from comparative genomics and small RNA sequencing.The DNA damage response: the omics era and its impact.Genome-wide analysis of genetic and epigenetic control of programmed DNA deletion.Double-strand break repair deficiency in NONO knockout murine embryonic fibroblasts and compensation by spontaneous upregulation of the PSPC1 paralog.Somatic mutations in DROSHA and DICER1 impair microRNA biogenesis through distinct mechanisms in Wilms tumours microRNA expression and biogenesis in cellular response to ionizing radiation RNA polymerase II CTD modifications: how many tales from a single tail.RNA interference in the nucleus: roles for small RNAs in transcription, epigenetics and beyond.Irreparable telomeric DNA damage and persistent DDR signalling as a shared causative mechanism of cellular senescence and ageing.Exome sequencing of pleuropulmonary blastoma reveals frequent biallelic loss of TP53 and two hits in DICER1 resulting in retention of 5p-derived miRNA hairpin loop sequences.An attempt to detect siRNA-mediated genomic DNA modification by artificially induced mismatch siRNA in Arabidopsis.DICER-dependent biogenesis of let-7 miRNAs affects human cell response to DNA damage via targeting p21/p27.Histone H3K56 acetylation is required for quelling-induced small RNA production through its role in homologous recombination.

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Site-specific DICER and DROSHA RNA products control the DNA-damage response

http://www.wikidata.org/entity/Q28269332

description

2012 nî lūn-bûn

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2012 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի օգոստոսին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Site-specific DICER and DROSHA RNA products control the DNA-damage response

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Site-specific DICER and DROSHA RNA products control the DNA-damage response

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Site-specific DICER and DROSHA RNA products control the DNA-damage response

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type

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Site-specific DICER and DROSHA RNA products control the DNA-damage response

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Site-specific DICER and DROSHA RNA products control the DNA-damage response

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Site-specific DICER and DROSHA RNA products control the DNA-damage response

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prefLabel

Site-specific DICER and DROSHA RNA products control the DNA-damage response

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Site-specific DICER and DROSHA RNA products control the DNA-damage response

@en

Site-specific DICER and DROSHA RNA products control the DNA-damage response

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Site-specific DICER and DROSHA RNA products control the DNA-damage response

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Alka Saxena

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Michiel de Hoon

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Sofia Francia

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The DNA-damage response in human biology and disease

Induced ncRNAs allosterically modify RNA-binding proteins in cis to inhibit transcription

Biogenesis of small RNAs in animals

Long non-coding RNAs: insights into functions

The widespread regulation of microRNA biogenesis, function and decay

MicroRNA expression in zebrafish embryonic development

Polycomb proteins targeted by a short repeat RNA to the mouse X chromosome

Long noncoding RNAs: functional surprises from the RNA world

The colorectal microRNAome

Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication

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10.1038/NATURE11179

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488

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Flavia Michelini

Fabrizio d'Adda di Fagagna

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2012-08-09T00:00:00Z

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227857347

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