Stat5a/b contribute to interleukin 7-induced B-cell precursor expansion, but abl- and bcr/abl-induced transformation are independent of stat5
about
TYK2 is a key regulator of the surveillance of B lymphoid tumorsPAK-dependent STAT5 serine phosphorylation is required for BCR-ABL-induced leukemogenesisSignal transducer and activator of transcription (Stat) 5 controls the proliferation and differentiation of mammary alveolar epitheliumInterleukin-7, a new cytokine targeting the mouse hypothalamic arcuate nucleus: role in body weight and food intake regulation.Socs-1 inhibits TEL-JAK2-mediated transformation of hematopoietic cells through inhibition of JAK2 kinase activity and induction of proteasome-mediated degradation.Clarifying the role of Stat5 in lymphoid development and Abelson-induced transformation.BCR-ABL but not JAK2 V617F inhibits erythropoiesis through the Ras signal by inducing p21CIP1/WAF1.Stat5a/b are essential for normal lymphoid development and differentiation.Unique effects of KIT D816V in BaF3 cells: induction of cluster formation, histamine synthesis, and early mast cell differentiation antigens.Enhanced T cell lymphoma in NOD.Stat5b transgenic mice is caused by hyperactivation of Stat5b in CD8+ thymocytes.Stat proteins and oncogenesis.Murine leukemia retrovirus integration induces the formation of transcription factor complexes on palindromic sequences in the signal transducer and activator of transcription factor 5a gene during the development of pre-B lymphomagenesisThe extreme carboxyl terminus of v-Abl is required for lymphoid cell transformation by Abelson virus.Ebf1 or Pax5 haploinsufficiency synergizes with STAT5 activation to initiate acute lymphoblastic leukemia.A truncation mutant of Csf3r cooperates with PML-RARα to induce acute myeloid leukemia in mice.Rac GTPases as key regulators of p210-BCR-ABL-dependent leukemogenesis.STAT3 positively regulates an early step in B-cell development.Essential role for Stat5a/b in myeloproliferative neoplasms induced by BCR-ABL1 and JAK2(V617F) in mice.Diverging fates of cells of origin in acute and chronic leukaemia.FoxO tumor suppressors and BCR-ABL-induced leukemia: a matter of evasion of apoptosis.Loss of beta-catenin impairs the renewal of normal and CML stem cells in vivoInterpretation of cytokine signaling through the transcription factors STAT5A and STAT5BOncogenic signaling: new insights and controversies from chronic myeloid leukemia.Aberrant cytokine signaling in leukemia.Re-evaluating the role of BCR/ABL in chronic myelogenous leukemiaResearch using Mesenchymal Stem/Stromal Cells: quality metric towards developing a reference material.New insights into the roles of Stat5a/b and Stat3 in T cell development and differentiation.Molecular biology of bcr-abl1-positive chronic myeloid leukemia.Following the cytokine signaling pathway to leukemogenesis: a chronologyTargeting signal transducer and activator of transcription signaling pathway in leukemias.Cutting edge: inherent and acquired resistance to radiation-induced apoptosis in B cells: a pivotal role for STAT3.Acute myeloid leukemia fusion proteins deregulate genes involved in stem cell maintenance and DNA repairGene expression profile of the hippocampus of rats subjected to chronic immobilization stress.Conditional IFNAR1 ablation reveals distinct requirements of Type I IFN signaling for NK cell maturation and tumor surveillanceA developmentally controlled competitive STAT5-PU.1 DNA binding mechanism regulates activity of the Ig κ E3' enhancer.Activated STAT5 proteins induce activation of the PI 3-kinase/Akt and Ras/MAPK pathways via the Gab2 scaffolding adapter.Type I diabetes-associated tolerogenic properties of interleukin-2.Maximal STAT5-induced proliferation and self-renewal at intermediate STAT5 activity levels.Stat5 is indispensable for the maintenance of bcr/abl-positive leukaemia.Role of STAT5 in controlling cell survival and immunoglobulin gene recombination during pro-B cell development.
P2860
Q24559982-63CFC2CC-004A-4615-9426-3323FEB1B404Q28302461-437AA4D2-DBE5-4B35-B370-F2AD30CE2C56Q28513906-2815225D-CCCA-4D15-9F1C-D2BACB02B947Q33550810-F3810B89-4C37-44BA-8670-1530C87A36B3Q33559005-89BE9750-850D-4951-949C-25A8B26F2E2AQ33874580-442ECCCD-1E22-4569-A2F6-590CD0D09985Q34181572-C2E04E5E-4516-45C6-9565-FCAC9E10D1BFQ34253065-B094D55D-520F-4F5B-A5B4-12AD0E65CC81Q34293285-869BF5DF-20D5-4D20-8B70-A437E0AF4F86Q34606927-5D089ABF-C1A6-45FF-8F1C-77E8E99638AAQ34627064-F7882D85-064D-4223-BCBF-43DFC4BA001BQ34760669-554C38CE-91AF-4883-B69E-755C0867A56FQ34859191-3828D05E-48EB-4841-B2BC-BC663B784A26Q35213566-C93D1AB9-E446-4932-9BFA-167D1327F938Q35564931-965AB6BB-8210-41C8-933A-4A8AF00FCCB2Q35730978-C1C84779-6BDE-43C6-9DD5-CAB537EA3A3DQ35849035-B2F866F9-806C-4E0D-B061-A5DAC4F0F467Q35885308-29359D3C-2D39-4BB3-AABC-390906219E42Q36038002-B106903E-A3F6-4874-82F1-4F5998550096Q36334327-FFEF9A4F-2D7A-45E4-B3D3-485FB896AD4AQ36488720-93EFF4FA-557D-445A-B6FA-97FFC1B46406Q36672274-57CED21D-0B16-4AFB-AC1A-509740C86D5DQ36758024-D1A72048-85A6-4D82-87D8-13EA2062F4DAQ36970192-486CA931-C98E-47F2-ADF4-4103E9F1843BQ36997520-62D1203D-1C2D-40F7-B98C-1699A8D2CF15Q37090955-0E1744D8-601B-4242-822B-BD3006AD1595Q37133882-2B6A5222-88A3-4543-B55C-8363F84BC234Q37283128-111BDFB1-E260-43C4-9D56-ED6A40365179Q37316601-59B0B2A3-DE6F-4E4F-91C7-5576ECBAFB89Q37347522-A0D4B319-886D-41E8-93AA-B38A235C814CQ37406213-018A9916-48F5-4CD2-B856-229D50436C67Q38347126-07858CD2-2B5D-47F1-8960-7342833D128BQ38491174-20302588-FA13-4E8C-9605-A38262CC9C4EQ39241252-0709163D-3235-4046-BA24-71E02BEE1242Q39405967-954F5992-5FC7-4D02-A4D7-9BCAD324B27AQ40434680-AD1F7BEB-D3B7-45A5-A841-EF72F3356A58Q41852651-9713E906-D11B-4BA2-85EA-6D8B71DBA26AQ41896132-F3100CC4-F1A1-47A8-91E6-604FE009D8A1Q42089534-C410F360-CDF4-442F-B331-156F8BB080D4Q42175565-AFB31FDB-18B4-4D68-98D1-4175F0996900
P2860
Stat5a/b contribute to interleukin 7-induced B-cell precursor expansion, but abl- and bcr/abl-induced transformation are independent of stat5
description
2000 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2000
@ast
im September 2000 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2000/09/15)
@sk
vědecký článek publikovaný v roce 2000
@cs
wetenschappelijk artikel (gepubliceerd op 2000/09/15)
@nl
наукова стаття, опублікована у вересні 2000
@uk
مقالة علمية (نشرت في 15-9-2000)
@ar
name
Stat5a/b contribute to interle ...... ation are independent of stat5
@ast
Stat5a/b contribute to interle ...... ation are independent of stat5
@en
Stat5a/b contribute to interle ...... ation are independent of stat5
@nl
type
label
Stat5a/b contribute to interle ...... ation are independent of stat5
@ast
Stat5a/b contribute to interle ...... ation are independent of stat5
@en
Stat5a/b contribute to interle ...... ation are independent of stat5
@nl
prefLabel
Stat5a/b contribute to interle ...... ation are independent of stat5
@ast
Stat5a/b contribute to interle ...... ation are independent of stat5
@en
Stat5a/b contribute to interle ...... ation are independent of stat5
@nl
P2093
P921
P1433
P1476
Stat5a/b contribute to interle ...... ation are independent of stat5
@en
P2093
J. N. Ihle
K. D. Bunting
K. Rothammer
M. F. Roussel
M. P. Brown
R. Moriggl
R. Piekorz
P304
P407
P577
2000-09-15T00:00:00Z