Lack of a laterality phenotype in Pkd1 knock-out embryos correlates with absence of polycystin-1 in nodal cilia
about
A polycystin-2 (TRPP2) dimerization domain essential for the function of heteromeric polycystin complexesA polycystin-centric view of cyst formation and disease: the polycystins revisitedSalient features of the ciliated organ of asymmetryTbx6 regulates left/right patterning in mouse embryos through effects on nodal cilia and perinodal signalingMouse models of ciliopathies: the state of the art.C2cd3 is required for cilia formation and Hedgehog signaling in mouse.Cilia and developmental signalingSitus inversus in Dpcd/Poll-/-, Nme7-/- , and Pkd1l1-/- micePkd1l1 establishes left-right asymmetry and physically interacts with Pkd2.The centrosomal protein pericentrin identified at the basal body complex of the connecting cilium in mouse photoreceptorsSensing a sensor: identifying the mechanosensory function of primary ciliaLeft-right asymmetry: cilia stir up new surprises in the node.Cilia, calcium and the basis of left-right asymmetry.The versatile nature of the calcium-permeable cation channel TRPP2Cilium, centrosome and cell cycle regulation in polycystic kidney disease.Altered trafficking and stability of polycystins underlie polycystic kidney disease.Polycystin-1 maturation requires polycystin-2 in a dose-dependent manner.Cell biology of polycystin-2.The subcellular localization of TRPP2 modulates its function.Polycystic kidney disease and renal injury repair: common pathways, fluid flow, and the function of polycystin-1.Syntaxin 5 regulates the endoplasmic reticulum channel-release properties of polycystin-2.Function and regulation of TRPP2 at the plasma membrane.Emerging evidence of a link between the polycystins and the mTOR pathways.Cystic kidney disease: the role of Wnt signaling.Polycystins and renovascular mechanosensory transduction.The primary cilium calcium channels and their role in flow sensing.Polycystin-1 but not polycystin-2 deficiency causes upregulation of the mTOR pathway and can be synergistically targeted with rapamycin and metformin.
P2860
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P2860
Lack of a laterality phenotype in Pkd1 knock-out embryos correlates with absence of polycystin-1 in nodal cilia
description
2005 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2005
@ast
im Oktober 2005 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2005/10/01)
@sk
vědecký článek publikovaný v roce 2005
@cs
wetenschappelijk artikel (gepubliceerd op 2005/10/01)
@nl
наукова стаття, опублікована в жовтні 2005
@uk
مقالة علمية (نشرت في أكتوبر 2005)
@ar
name
Lack of a laterality phenotype ...... of polycystin-1 in nodal cilia
@ast
Lack of a laterality phenotype ...... of polycystin-1 in nodal cilia
@en
Lack of a laterality phenotype ...... of polycystin-1 in nodal cilia
@nl
type
label
Lack of a laterality phenotype ...... of polycystin-1 in nodal cilia
@ast
Lack of a laterality phenotype ...... of polycystin-1 in nodal cilia
@en
Lack of a laterality phenotype ...... of polycystin-1 in nodal cilia
@nl
prefLabel
Lack of a laterality phenotype ...... of polycystin-1 in nodal cilia
@ast
Lack of a laterality phenotype ...... of polycystin-1 in nodal cilia
@en
Lack of a laterality phenotype ...... of polycystin-1 in nodal cilia
@nl
P2093
P1433
P1476
Lack of a laterality phenotype ...... of polycystin-1 in nodal cilia
@en
P2093
Anja Fischer
Axel Schweickert
Christina Karcher
Eva Bitzer
Martin Blum
Shigeo Horie
P304
P356
10.1111/J.1432-0436.2005.00048.X
P577
2005-10-01T00:00:00Z