Lethal effect of the Abelson murine leukemia virus transforming gene product.
about
The complete coding sequence of arg defines the Abelson subfamily of cytoplasmic tyrosine kinasesHuman c-fms proto-oncogene: comparative analysis with an abnormal alleleHuman immunodeficiency virus type 1 glycoprotein precursor retains a CD4-p56lck complex in the endoplasmic reticulumTransforming pathways activated by the v-Abl tyrosine kinaseThree different activated ras genes in mouse tumours; evidence for oncogene activation during progression of a mouse lymphomaThe cytostatic function of c-Abl is controlled by multiple nuclear localization signals and requires the p53 and Rb tumor suppressor gene productsProduction of high-titer helper-free retroviruses by transient transfectionBCR first exon sequences specifically activate the BCR/ABL tyrosine kinase oncogene of Philadelphia chromosome-positive human leukemias.A Swiss 3T3 variant cell line resistant to the effects of tumor promoters cannot be transformed by srcRescue of cells from ras oncogene-induced growth arrest by a second, complementing, oncogene.N-terminal mutations activate the leukemogenic potential of the myristoylated form of c-ablAbelson murine leukemia virus variants with increased oncogenic potential.Regulation of pp56lck during T-cell activation: functional implications for the src-like protein tyrosine kinasesOncogenic v-Abl tyrosine kinase can inhibit or stimulate growth, depending on the cell context.p53 deficiency increases transformation by v-Abl and rescues the ability of a C-terminally truncated v-Abl mutant to induce pre-B lymphoma in vivo.Complete c-mos (rat) nucleotide sequence: presence of conserved domains in c-mos proteinsEpstein-Barr virus LMP1 modulates the malignant potential of gastric carcinoma cells involving apoptosis.Insertional mutagenesis of the Abelson murine leukemia virus genome: identification of mutants with altered kinase activity and defective transformation ability.P80: a tumor-related protein found in many lymphomas of mice.BCR-ABL and v-abl oncogenes induce distinct patterns of thymic lymphoma involving different lymphocyte subsets.Mutated alpha subunit of the Gq protein induces malignant transformation in NIH 3T3 cellsOncogene cooperation in lymphocyte transformation: malignant conversion of E mu-myc transgenic pre-B cells in vitro is enhanced by v-H-ras or v-raf but not v-abl.The BCR-ABL oncogene transforms Rat-1 cells and cooperates with v-myc.Alternative forms of the BCR-ABL oncogene have quantitatively different potencies for stimulation of immature lymphoid cells.Recombinants within the tyrosine kinase region of v-abl and v-src identify a v-abl segment that confers lymphoid specificity.Neoplastic transformation induced by an activated lymphocyte-specific protein tyrosine kinase (pp56lck).Temperature-sensitive mutants of Abelson murine leukemia virus deficient in protein tyrosine kinase activity.DNA sequence, structure, and tyrosine kinase activity of the Drosophila melanogaster Abelson proto-oncogene homologCarboxyl-terminal determinants of Abelson protein important for lymphoma induction.The transforming domain alone of the latent membrane protein of Epstein-Barr virus is toxic to cells when expressed at high levelsOnly site-directed antibodies reactive with the highly conserved src-homologous region of the v-abl protein neutralize kinase activity.Protein stabilization explains the gag requirement for transformation of lymphoid cells by Abelson murine leukemia virusA membrane-associated, carbohydrate-modified form of the v-abl protein that cannot be phosphorylated in vivo or in vitroLoss of viral gene expression and retention of tumorigenicity by Abelson lymphoma cells.Normal cellular and transformation-associated abl proteins share common sites for protein kinase C phosphorylation.Recombinational junctions of variants of Moloney murine sarcoma virus: generation and divergence of a mammalian transforming geneSite-directed deletions of Abelson murine leukemia virus define 3' sequences essential for transformation and lethality.Progression of the transformed phenotype in clonal lines of Abelson virus-infected lymphocytesThe location of v-src in a retrovirus vector determines whether the virus is toxic or transforming.Long-term cultured B lymphocytes and their precursors reconstitute the B-lymphocyte lineage in vivo.
P2860
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P2860
Lethal effect of the Abelson murine leukemia virus transforming gene product.
description
1981 nî lūn-bûn
@nan
1981 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1981 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1981年の論文
@ja
1981年論文
@yue
1981年論文
@zh-hant
1981年論文
@zh-hk
1981年論文
@zh-mo
1981年論文
@zh-tw
1981年论文
@wuu
name
Lethal effect of the Abelson murine leukemia virus transforming gene product.
@ast
Lethal effect of the Abelson murine leukemia virus transforming gene product.
@en
type
label
Lethal effect of the Abelson murine leukemia virus transforming gene product.
@ast
Lethal effect of the Abelson murine leukemia virus transforming gene product.
@en
prefLabel
Lethal effect of the Abelson murine leukemia virus transforming gene product.
@ast
Lethal effect of the Abelson murine leukemia virus transforming gene product.
@en
P2093
P1433
P1476
Lethal effect of the Abelson murine leukemia virus transforming gene product.
@en
P2093
P304
P356
10.1016/0092-8674(81)90389-5
P407
P433
P577
1981-12-01T00:00:00Z