Alteration in calcium handling at the subcellular level in mdx myotubes.
about
Hypernitrosylated ryanodine receptor calcium release channels are leaky in dystrophic muscleInvestigation of Debio 025, a cyclophilin inhibitor, in the dystrophic mdx mouse, a model for Duchenne muscular dystrophyRescue of dystrophic skeletal muscle by PGC-1α involves a fast to slow fiber type shift in the mdx mouseCalcium Dyshomeostasis in Tubular Aggregate MyopathyGenetic evidence in the mouse solidifies the calcium hypothesis of myofiber death in muscular dystrophyBeat-to-beat oscillations of mitochondrial [Ca2+] in cardiac cellsPrimary Murine Myotubes as a Model for Investigating Muscular DystrophyPhysical coupling supports the local Ca2+ transfer between sarcoplasmic reticulum subdomains and the mitochondria in heart muscle.Dystrophic Cardiomyopathy-Potential Role of Calcium in Pathogenesis, Treatment and Novel TherapiesMicroarchitecture is severely compromised but motor protein function is preserved in dystrophic mdx skeletal muscle.Convergent regulation of skeletal muscle Ca2+ channels by dystrophin, the actin cytoskeleton, and cAMP-dependent protein kinase.Orai1 mediates exacerbated Ca(2+) entry in dystrophic skeletal muscleDefects in mitochondrial ATP synthesis in dystrophin-deficient mdx skeletal muscles may be caused by complex I insufficiency.Mitochondrial dysfunctions during progression of dystrophic cardiomyopathyMutation of delta-sarcoglycan is associated with Ca(2+) -dependent vascular remodeling in the Syrian hamster.Increased resting intracellular calcium modulates NF-κB-dependent inducible nitric-oxide synthase gene expression in dystrophic mdx skeletal myotubesEnhanced Na+/H+ exchange activity contributes to the pathogenesis of muscular dystrophy via involvement of P2 receptors.Structural and functional evaluation of branched myofibers lacking intermediate filamentsCharacterization and Functional Analysis of Extracellular Vesicles and Muscle-Abundant miRNAs (miR-1, miR-133a, and miR-206) in C2C12 Myocytes and mdx MiceIn vivo monitoring of Ca(2+) uptake into mitochondria of mouse skeletal muscle during contractionA novel mechanism of myocyte degeneration involving the Ca2+-permeable growth factor-regulated channel.Stressed out: the skeletal muscle ryanodine receptor as a target of stressIdentification of disease specific pathways using in vivo SILAC proteomics in dystrophin deficient mdx mouse.Altered calcium pump and secondary deficiency of gamma-sarcoglycan and microspan in sarcoplasmic reticulum membranes isolated from delta-sarcoglycan knockout mice.Cancer cachexia causes skeletal muscle damage via transient receptor potential vanilloid 2-independent mechanisms, unlike muscular dystrophyInteractions between sarco-endoplasmic reticulum and mitochondria in cardiac and skeletal muscle - pivotal roles in Ca²⁺ and reactive oxygen species signaling.Loss of positive allosteric interactions between neuronal nitric oxide synthase and phosphofructokinase contributes to defects in glycolysis and increased fatigability in muscular dystrophy.Malformed mdx myofibers have normal cytoskeletal architecture yet altered EC coupling and stress-induced Ca2+ signaling.Sarcoplasmic reticulum-mitochondrial through-space coupling in skeletal muscle.Alterations in mitochondrial function as a harbinger of cardiomyopathy: lessons from the dystrophic heart.Mitochondria mediate cell membrane repair and contribute to Duchenne muscular dystrophy.Cardiac and respiratory dysfunction in Duchenne muscular dystrophy and the role of second messengers.Dysregulation of calcium homeostasis in muscular dystrophies.Mitochondrial Ca(2+) uptake in skeletal muscle health and disease.Metabolic regulation of Ca2+ release in permeabilized mammalian skeletal muscle fibres.Transfer and tunneling of Ca2+ from sarcoplasmic reticulum to mitochondria in skeletal muscle.Bcl-2 overexpression prevents calcium overload and subsequent apoptosis in dystrophic myotubes.Drastic reduction of sarcalumenin in Dp427 (dystrophin of 427 kDa)-deficient fibres indicates that abnormal calcium handling plays a key role in muscular dystrophy.Depolarization-induced contraction and SR function in mechanically skinned muscle fibers from dystrophic mdx mice.Application of complementary luminescent and fluorescent imaging techniques to visualize nuclear and cytoplasmic Ca²⁺ signalling during the in vivo differentiation of slow muscle cells in zebrafish embryos under normal and dystrophic conditions.
P2860
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P2860
Alteration in calcium handling at the subcellular level in mdx myotubes.
description
2000 nî lūn-bûn
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2000 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի հոտեմբերին հրատարակված գիտական հոդված
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2000年の論文
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2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Alteration in calcium handling at the subcellular level in mdx myotubes.
@ast
Alteration in calcium handling at the subcellular level in mdx myotubes.
@en
type
label
Alteration in calcium handling at the subcellular level in mdx myotubes.
@ast
Alteration in calcium handling at the subcellular level in mdx myotubes.
@en
prefLabel
Alteration in calcium handling at the subcellular level in mdx myotubes.
@ast
Alteration in calcium handling at the subcellular level in mdx myotubes.
@en
P2093
P356
P1476
Alteration in calcium handling at the subcellular level in mdx myotubes
@en
P2093
P304
P356
10.1074/JBC.M006337200
P407
P577
2000-10-11T00:00:00Z