Advances in understanding of pathogenesis of aHUS and HELLP. - Wikidata - wikimedia - TriplyDB

Advances in understanding of pathogenesis of aHUS and HELLP.

Advances in understanding of pathogenesis of aHUS and HELLP.

http://www.wikidata.org/entity/Q33380684

about

Plasma complement components and activation fragments: associations with age-related macular degeneration genotypes and phenotypes The complement system and adverse pregnancy outcomes Mutations in complement regulatory proteins predispose to preeclampsia: a genetic analysis of the PROMISSE cohort Early clinical indicators of transplant-associated thrombotic microangiopathy in pediatric neuroblastoma patients undergoing auto-SCT.Use of eculizumab for atypical haemolytic uraemic syndrome and C3 glomerulopathies.Renal arteriolar C4d deposition: a novel characteristic of hematopoietic stem cell transplantation-associated thrombotic microangiopathy.A curious case of haemolysis, seizure and acute renal failure associated with pregnancy.Complement regulators in human disease: lessons from modern genetics.Overactivation of Complement Alternative Pathway in Postpartum Atypical Hemolytic Uremic Syndrome Patients with Renal Involvement.Direct evidence of complement activation in HELLP syndrome: A link to atypical hemolytic uremic syndrome.Dysregulated complement activation as a common pathway of injury in preeclampsia and other pregnancy complications.Autoantibody-mediated complement C3a receptor activation contributes to the pathogenesis of preeclampsia.Defective complement inhibitory function predisposes to renal disease.HELLP Syndrome at 17 Weeks Gestation: A Rare and Catastrophic Phenomenon.Complement, age-related macular degeneration and a vision of the future The management of the surgical complications of HELLP syndrome Recent insights into the pathobiology of innate immune deficiencies Complement activation: an emerging player in the pathogenesis of cardiovascular disease Complement in multiple sclerosis: its role in disease and potential as a biomarker.Early elevations of the complement activation fragment C3a and adverse pregnancy outcomes.Age-related macular degeneration: genetic and clinical findings.HELLP syndrome: understanding and management of a pregnancy-specific disease.Immunology of hepatic diseases during pregnancy.Complementopathies.Low levels of protein Z are associated with HELLP syndrome and its severity.The genetics of the alternative pathway of complement in the pathogenesis of HELLP syndrome.Pregnancy-induced atypical haemolytic uremic syndrome: A new era with eculizumab.

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P2860

Advances in understanding of pathogenesis of aHUS and HELLP.

http://www.wikidata.org/entity/Q33380684

description

2008 nî lūn-bûn

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2008 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի օգոստոսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

@zh-tw

2008年论文

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name

Advances in understanding of pathogenesis of aHUS and HELLP.

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Advances in understanding of pathogenesis of aHUS and HELLP.

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type

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Advances in understanding of pathogenesis of aHUS and HELLP.

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Advances in understanding of pathogenesis of aHUS and HELLP.

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Advances in understanding of pathogenesis of aHUS and HELLP.

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Advances in understanding of pathogenesis of aHUS and HELLP.

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J.1365-2141.2008.07324.X

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British Journal of Haematology

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Advances in understanding of pathogenesis of aHUS and HELLP.

@en

P2093

Anna Richards

http://www.w3.org/2001/XMLSchema#string

Celia J Fang

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John P Atkinson

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M Kathryn Liszewski

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P2860

Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia

Mutations in factor H reduce binding affinity to C3b and heparin and surface attachment to endothelial cells in hemolytic uremic syndrome.

Diagnosis, controversies, and management of the syndrome of hemolysis, elevated liver enzymes, and low platelet count.

Imitators of severe preeclampsia.

A novel non-synonymous polymorphism (p.Arg240His) in C4b-binding protein is associated with atypical hemolytic uremic syndrome and leads to impaired alternative pathway cofactor activity.

Comparison of plasma exchange with plasma infusion in the treatment of thrombotic thrombocytopenic purpura. Canadian Apheresis Study Group.

Acute renal failure in pregnancies complicated by hemolysis, elevated liver enzymes, and low platelets.

Vascular endothelial growth factor ligands and receptors that regulate human cytotrophoblast survival are dysregulated in severe preeclampsia and hemolysis, elevated liver enzymes, and low platelets syndrome.

Alternative complement pathway activation fragment Bb in early pregnancy as a predictor of preeclampsia.

Binding of complement factor H to endothelial cells is mediated by the carboxy-terminal glycosaminoglycan binding site.

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10.1111/J.1365-2141.2008.07324.X

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