Central exercise action increases the AMPK and mTOR response to leptin
about
Hypothalamic AMPK as a Regulator of Energy HomeostasisIL-6 and IL-10 anti-inflammatory activity links exercise to hypothalamic insulin and leptin sensitivity through IKKbeta and ER stress inhibitionVoluntary Exercise Improves Estrous Cyclicity in Prenatally Androgenized Female Mice Despite Programming Decreased Voluntary Exercise: Implications for Polycystic Ovary Syndrome (PCOS).Metabolic syndrome and insulin resistance: underlying causes and modification by exercise training.Knockdown of the fat mass and obesity gene disrupts cellular energy balance in a cell-type specific manner.Hypothalamic mTOR signaling mediates the orexigenic action of ghrelin.Exercise reduces appetite and traffics excess nutrients away from energetically efficient pathways of lipid deposition during the early stages of weight regainEffects of exercise on brain functions in diabetic animal models.The effects of exercise on the neuronal response to food cues.S6K1 in the central nervous system regulates energy expenditure via MC4R/CRH pathways in response to deprivation of an essential amino acid.Benefits of resistance exercise in lean women with fibromyalgia: involvement of IGF-1 and leptinAnorexigenic and orexigenic hormone modulation of mammalian target of rapamycin complex 1 activity and the regulation of hypothalamic agouti-related protein mRNA expression.Regular exercise attenuates the metabolic drive to regain weight after long-term weight loss.Hypothalamic activation is essential for endotoxemia-induced acute muscle wasting.mTORC1 signaling in energy balance and metabolic disease.Hypothalamic AMP-activated protein kinase as a mediator of whole body energy balance.Interleukin-6 enhances glucose-stimulated insulin secretion from pancreatic beta-cells: potential involvement of the PLC-IP3-dependent pathway.Combination therapy targeting Akt and mammalian target of rapamycin improves functional outcome after controlled cortical impact in mice.Retraction Note: Diacerhein attenuates the inflammatory response and improves survival in a model of severe sepsis.Inhibition of hypothalamic Foxo1 expression reduced food intake in diet-induced obesity rats.Acute exercise suppresses hypothalamic PTP1B protein level and improves insulin and leptin signaling in obese rats.Retraction: Central Exercise Action Increases the AMPK and mTOR Response to Leptin.Hypothalamic mTOR pathway mediates thyroid hormone-induced hyperphagia in hyperthyroidism.IL-6 regulates exercise and training-induced adaptations in subcutaneous adipose tissue in miceGhrelin, Lipid Metabolism, and Metabolic Syndrome
P2860
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P2860
Central exercise action increases the AMPK and mTOR response to leptin
description
2008 nî lūn-bûn
@nan
2008 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
name
Central exercise action increases the AMPK and mTOR response to leptin
@ast
Central exercise action increases the AMPK and mTOR response to leptin
@en
type
label
Central exercise action increases the AMPK and mTOR response to leptin
@ast
Central exercise action increases the AMPK and mTOR response to leptin
@en
prefLabel
Central exercise action increases the AMPK and mTOR response to leptin
@ast
Central exercise action increases the AMPK and mTOR response to leptin
@en
P2093
P2860
P1433
P1476
Central exercise action increases the AMPK and mTOR response to leptin
@en
P2093
Eduardo R Ropelle
José B C Carvalheira
Kleber G Franchini
Lício A Velloso
Marcelo B S Flores
Maria Fernanda A Fernandes
Mario J A Saad
Mirian Ueno
Rodrigo Marin
P2860
P356
10.1371/JOURNAL.PONE.0003856
P407
P577
2008-12-04T00:00:00Z