11β-HSD1 is the major regulator of the tissue-specific effects of circulating glucocorticoid excess - Wikidata - wikimedia - TriplyDB

11β-HSD1 is the major regulator of the tissue-specific effects of circulating glucocorticoid excess

11β-HSD1 is the major regulator of the tissue-specific effects of circulating glucocorticoid excess

http://www.wikidata.org/entity/Q33789944

about

Developmental programming of hypothalamic neuronal circuits: impact on energy balance control Getting to the heart of intracellular glucocorticoid regeneration: 11β-HSD1 in the myocardium The Metabolic Implications of Glucocorticoids in a High-Fat Diet Setting and the Counter-Effects of Exercise Fructose, Glucocorticoids and Adipose Tissue: Implications for the Metabolic Syndrome.Plasma, salivary and urinary cortisol levels following physiological and stress doses of hydrocortisone in normal volunteers.Erythropoietin negatively regulates pituitary ACTH secretion Saturated fatty acids in human visceral adipose tissue are associated with increased 11- β-hydroxysteroid-dehydrogenase type 1 expression Association Analysis between the Polymorphisms of HSD11B1 and H6PD and Risk of Polycystic Ovary Syndrome in Chinese Population.Meta-analysis derived atopic dermatitis (MADAD) transcriptome defines a robust AD signature highlighting the involvement of atherosclerosis and lipid metabolism pathways Cortisol dysregulation in obesity-related metabolic disorders.The Role of Placental 11-Beta Hydroxysteroid Dehydrogenase Type 1 and Type 2 Methylation on Gene Expression and Infant Birth Weight.Glucocorticoid Excess Increases Hepatic 11β-HSD-1 Activity in Humans: Implications in Steroid-Induced Diabetes.Local corticosterone activation by 11β-hydroxysteroid dehydrogenase 1 in keratinocytes: the role in narrow-band UVB-induced dermatitis.Effect of Age, Season, Body Condition, and Endocrine Status on Serum Free Cortisol Fraction and Insulin Concentration in Horses.Investigating the role of cortisol and growth hormone in fatty liver development: fatty liver index in patients with pituitary adenomas.11β-Hydroxysteroid Dehydrogenase Type 1(11β-HSD1) mediates insulin resistance through JNK activation in adipocytes 11β-Hydroxysteroid dehydrogenase type 1 within muscle protects against the adverse effects of local inflammation.In ovo injection of betaine alleviates corticosterone-induced fatty liver in chickens through epigenetic modifications.Analysis of Purified Pancreatic Islet Beta and Alpha Cell Transcriptomes Reveals 11β-Hydroxysteroid Dehydrogenase (Hsd11b1) as a Novel Disallowed Gene.Cellular and genetic models of H6PDH and 11β-HSD1 function in skeletal muscle.Glucocorticoids and 11β-HSD1 are major regulators of intramyocellular protein metabolism.INVITED REVIEW: The usefulness of measuring glucocorticoids for assessing animal welfare.11β-HSD1 Modulates the Set Point of Brown Adipose Tissue Response to Glucocorticoids in Male Mice.Male 11β-HSD1 Knockout Mice Fed Trans-Fats and Fructose Are Not Protected From Metabolic Syndrome or Nonalcoholic Fatty Liver Disease.11β-Hydroxysteroid Dehydrogenase Type 1 Is Expressed in Neutrophils and Restrains an Inflammatory Response in Male Mice.Polymorphisms of the GR and HSD11B1 genes influence body mass index and weight gain during hormone replacement treatment in patients with Addison's disease.Compared to casein, bovine lactoferrin reduces plasma leptin and corticosterone and affects hypothalamic gene expression without altering weight gain or fat mass in high fat diet fed C57/BL6J mice.Gene expression changes in subcutaneous adipose tissue due to Cushing's disease.Hydroxysteroid dehydrogenase HSD1L is localised to the pituitary-gonadal axis of primates.Elevated Hypothalamic Glucocorticoid Levels Are Associated With Obesity and Hyperphagia in Male Mice.The Pathogenesis of Polycystic Ovary Syndrome (PCOS): The Hypothesis of PCOS as Functional Ovarian Hyperandrogenism Revisited.HPA axis dysregulation, NR3C1 polymorphisms and glucocorticoid receptor isoforms imbalance in metabolic syndrome.11β-Hydroxysteroid Dehydrogenase Type 1 Inhibition Attenuates the Adverse Effects of Glucocorticoids on Dermal Papilla Cells.Anti-inflammatory effects of levalbuterol-induced 11β-hydroxysteroid dehydrogenase type 1 activity in airway epithelial cells.11β-hydroxysteroid dehydrogenase type 1 has no effect on survival during experimental malaria but affects parasitemia in a parasite strain-specific manner.11β-HSD1 inhibition ameliorates diabetes-induced cardiomyocyte hypertrophy and cardiac fibrosis through modulation of EGFR activity.11β-Hydroxysteroid Dehydrogenases and Hypertension in the Metabolic Syndrome.Quantification of 11β-hydroxysteroid dehydrogenase 1 kinetics and pharmacodynamic effects of inhibitors in brain using mass spectrometry imaging and stable-isotope tracers in mice.11β-hydroxysteroid dehydrogenase-1 deficiency alters brain energy metabolism in acute systemic inflammation.Acute Sarcopenia Secondary to Hospitalisation - An Emerging Condition Affecting Older Adults.

P2860

Q26997752-5B550C9B-8EC7-4AF5-990C-88D730FBD7F4 Q28067162-C99C15D1-AA91-4086-ACD4-E4465E09106F Q28076700-370978AA-53A7-40A6-A31E-05866A238E00 Q33749151-3FE8ABCD-C55C-4D80-90ED-98726925DDB2 Q34794021-CE061638-3B0F-4758-881B-A967D1FF292E Q35306172-3EBA0204-C970-4EB9-BBE2-EFAE96EFD957 Q35583941-B383A812-76D7-4BD5-AA89-DCB28AB2B24D Q35801880-F302C8F1-2B51-41BF-B670-69A13FF10F72 Q35803992-DEC02F22-6A40-4CE0-9CDB-A3214ED996DB Q35896904-FDAA430B-DA75-4315-B3BC-5A482763EF3B Q36298779-E1ACAE41-8C91-4D1B-8F23-4CCFB1883DE8 Q36433592-47D71251-57FC-409F-90F1-00D095B805F6 Q36887524-F4738040-B9DC-4BDE-BEE8-0D0C9824E681 Q37020324-CEE8214B-8636-4E3B-A920-689C081EAEE5 Q37201576-A9DACEE2-BBB6-4296-A9EA-63C4D5665E13 Q37411798-229810B8-8D3B-49FD-BCC3-A01F24C2EC05 Q37417655-B54A4B4E-6FD3-4256-8D31-FA351EA0D629 Q37563307-DD28FE22-73EB-40E6-8CA4-19163F4D8A78 Q37742862-E1091137-84C6-484E-B4FC-97B795D5753B Q38658108-BA164561-947C-4803-B913-63E14738817C Q38780542-DBC65BFA-23F9-4C43-9599-F51F876BD86A Q38804458-3AF48000-67D1-428B-8F06-7BDB860F2F7A Q38860951-8406400A-E56A-42F8-BB91-65E7E2A501D1 Q39626951-957E0EBD-9250-44FE-8857-DB0F263E6893 Q39795478-25DD4DEE-2B1F-49F0-ACAE-7C40A5EA5879 Q40072950-12A4C41D-DFB5-4B2C-A471-D9DA61E88327 Q40230388-6F1FA6D7-1893-479E-9F75-A87B16F3DE6E Q40762458-231DFB01-D76E-4468-A6AD-F5F6F5A768E4 Q41396281-5E27E286-9C68-47E6-B533-C3168ABE8731 Q41619943-3B9EF95F-6A17-470A-BFDB-504DFF3B4212 Q41640666-AB52DA41-1562-4F04-AA1D-3EFF2AA4ACB8 Q42497430-B5B148FE-2631-454C-B86C-35A26B293942 Q42646933-58F8FBC8-8A9D-4CC7-87F5-D1B4072E22EA Q43101461-5E294D8D-A30F-4C55-A221-C943F402D538 Q43272640-2B420A4D-81CE-4F96-9247-4B3B474800BE Q47094783-D78520BE-8C24-4C9A-AF7B-C190BBF1D18E Q47098934-FB0421AC-AF97-41F0-8B99-4BB4DF8EC79E Q47297002-6223A63D-8488-4419-9428-8C0EEB96E7E0 Q47433431-70782A12-209B-4228-AC81-092226F2C0EC Q47698223-46FF90DE-4A2B-4531-B4E8-0037B2666B0C

P2860

11β-HSD1 is the major regulator of the tissue-specific effects of circulating glucocorticoid excess

http://www.wikidata.org/entity/Q33789944

description

2014 nî lūn-bûn

@nan

2014 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

11β-HSD1 is the major regulato ...... culating glucocorticoid excess

@ast

11β-HSD1 is the major regulato ...... culating glucocorticoid excess

@en

11β-HSD1 is the major regulato ...... culating glucocorticoid excess

@en-gb

type

label

11β-HSD1 is the major regulato ...... culating glucocorticoid excess

@ast

11β-HSD1 is the major regulato ...... culating glucocorticoid excess

@en

11β-HSD1 is the major regulato ...... culating glucocorticoid excess

@en-gb

prefLabel

11β-HSD1 is the major regulato ...... culating glucocorticoid excess

@ast

11β-HSD1 is the major regulato ...... culating glucocorticoid excess

@en

11β-HSD1 is the major regulato ...... culating glucocorticoid excess

@en-gb

P1433

Q33789944-B849F581-C44E-4C10-91C5-79FBBA9EE200

P1476

Q33789944-FF448E04-8659-4C50-B547-9E910AA654FF

P2093

Q33789944-0855B4F8-A93F-4DD7-BE0A-1271AE1F0411

Q33789944-0A8C95F1-130F-40EE-A5C5-BBFE4D3E94AD

Q33789944-0D7FC70C-608D-4F31-8A4C-9195174D2B85

Q33789944-18F83B37-441A-415A-B06F-05E07BC4EEEC

Q33789944-45AA1FF5-F7DC-40FF-AD2A-4C9B64ADEB1F

Q33789944-AE002236-AFEC-4D41-97D2-29CADE088BB3

Q33789944-DA8C5760-DACF-44AA-BC8C-1E104956839B

P2860

Q33789944-0199D370-41E0-43B4-9252-70DA975BE979

Q33789944-09667959-B10E-45E3-A5D0-8F302324C780

Q33789944-13A87958-EA22-4CDF-9EBF-84B11B95CA05

Q33789944-17A328E4-DA20-4B38-B23F-2BED7F5C4F27

Q33789944-18875404-30AF-48E9-BC9C-D82DB6CA5859

Q33789944-2351816C-1C30-4BA9-806E-A1DD01D236A7

Q33789944-251C7B76-5D1D-4B92-9D46-68204E374222

Q33789944-3274B6C0-8FD1-4BDD-9AFF-3C0EFE78BDEF

Q33789944-3969BFD6-27F1-4EAE-B5C6-2B3737C7E4DD

Q33789944-451556D7-97B3-4048-AF90-A4EB85DE224D

P304

Q33789944-40EEF5E3-2457-4B26-B7B1-323488439E3D

P31

Q33789944-C8CCCFA9-59D9-47EB-A1B3-CA421B7F0083

P356

Q33789944-C95C7A01-2B24-4C5C-A92D-4298F1591D7A

P407

Q33789944-0BEFA309-29CA-4629-940A-D0E3ECC4533B

P433

Q33789944-62B902C8-890E-4BB4-9EBC-11C7F25A1CAE

P478

Q33789944-9A9E8378-6F83-4D19-80E6-65B9C50DA804

P50

Q33789944-5DE884E1-CBE7-48F0-BD8C-0E5070C85DDA

Q33789944-A867633D-BE12-4866-A6E7-8E140754FA6D

P577

Q33789944-F2C988A9-DC52-4E81-AFCE-B535188CE9CD

P5875

Q33789944-34E0B715-A635-49C4-97E2-1D7A1FF12BE7

P698

Q33789944-5C0DE10C-BCDF-4280-BCA3-64C11DA4E936

P932

Q33789944-59E6DEDE-E5F7-4FDF-86EE-C20AF072E910

P356

PNAS.1323681111

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

11β-HSD1 is the major regulato ...... culating glucocorticoid excess

@en

P2093

Dean P Larner

http://www.w3.org/2001/XMLSchema#string

Emma L McCabe

http://www.w3.org/2001/XMLSchema#string

Gareth G Lavery

http://www.w3.org/2001/XMLSchema#string

Iwona J Bujalska

http://www.w3.org/2001/XMLSchema#string

Laura L Gathercole

http://www.w3.org/2001/XMLSchema#string

Stuart A Morgan

http://www.w3.org/2001/XMLSchema#string

Zaki K Hassan-Smith

http://www.w3.org/2001/XMLSchema#string

P2860

Dissociation of transactivation from transrepression by a selective glucocorticoid receptor agonist leads to separation of therapeutic effects from side effects

BOL-303242-X, a novel selective glucocorticoid receptor agonist, with full anti-inflammatory properties in human ocular cells

trans-Activation and repression properties of the novel nonsteroid glucocorticoid receptor ligand 2,5-dihydro-9-hydroxy-10-methoxy-2,2,4-trimethyl-5-(1-methylcyclohexen-3-y1)-1H-[1]benzopyrano[3,4-f]quinoline (A276575) and its four stereoisomers

Direct effect of glucocorticoids on lipolysis in adipocytes.

Metabolic syndrome without obesity: Hepatic overexpression of 11beta-hydroxysteroid dehydrogenase type 1 in transgenic mice

Endocrine and physiological changes in response to chronic corticosterone: a potential model of the metabolic syndrome in mouse.

The 11-beta-hydroxysteroid dehydrogenase type 1 inhibitor INCB13739 improves hyperglycemia in patients with type 2 diabetes inadequately controlled by metformin monotherapy.

A transgenic model of visceral obesity and the metabolic syndrome.

Transgenic amplification of glucocorticoid action in adipose tissue causes high blood pressure in mice.

Hexose-6-phosphate dehydrogenase contributes to skeletal muscle homeostasis independent of 11β-hydroxysteroid dehydrogenase type 1.

P304

E2482-91

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1073/PNAS.1323681111

http://www.w3.org/2001/XMLSchema#string

P407

P433

24

http://www.w3.org/2001/XMLSchema#string

P478

111

http://www.w3.org/2001/XMLSchema#string

P50

Jeremy W Tomlinson

P577

2014-06-02T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

262817184

http://www.w3.org/2001/XMLSchema#string

P698

24889609

http://www.w3.org/2001/XMLSchema#string

P932

4066483

http://www.w3.org/2001/XMLSchema#string