Novel function of Rad27 (FEN-1) in restricting short-sequence recombination.
about
Activation of an alternative, rec12 (spo11)-independent pathway of fission yeast meiotic recombination in the absence of a DNA flap endonucleaseArginine residues 47 and 70 of human flap endonuclease-1 are involved in DNA substrate interactions and cleavage site determinationWRN helicase and FEN-1 form a complex upon replication arrest and together process branchmigrating DNA structures associated with the replication forkStimulation of flap endonuclease-1 by the Bloom's syndrome proteinInferring transcriptional compensation interactions in yeast via stepwise structure equation modelingOkazaki fragment processing: modulation of the strand displacement activity of DNA polymerase delta by the concerted action of replication protein A, proliferating cell nuclear antigen, and flap endonuclease-1.Okazaki fragment maturation in yeast. I. Distribution of functions between FEN1 AND DNA2.Multiple pathways promote short-sequence recombination in Saccharomyces cerevisiaeKinetics of endogenous mouse FEN1 in base excision repair.The DNA-protein interaction modes of FEN-1 with gap substrates and their implication in preventing duplication mutations.Complementary non-radioactive assays for investigation of human flap endonuclease 1 activity.The Rad27 (Fen-1) nuclease inhibits Ty1 mobility in Saccharomyces cerevisiae.Nuclease-deficient FEN-1 blocks Rad51/BRCA1-mediated repair and causes trinucleotide repeat instabilityFunctional analyses of human DNA repair proteins important for aging and genomic stability using yeast geneticsA mutant allele of the transcription factor IIH helicase gene, RAD3, promotes loss of heterozygosity in response to a DNA replication defect in Saccharomyces cerevisiae.The role of DNA exonucleases in protecting genome stability and their impact on ageing.RAD59 is required for efficient repair of simultaneous double-strand breaks resulting in translocations in Saccharomyces cerevisiaeSaccharomyces cerevisiae flap endonuclease 1 uses flap equilibration to maintain triplet repeat stabilityHuman replication factor C stimulates flap endonuclease 1Fen-1 facilitates homologous recombination by removing divergent sequences at DNA break ends.Analysis of human flap endonuclease 1 mutants reveals a mechanism to prevent triplet repeat expansion.Human Bloom protein stimulates flap endonuclease 1 activity by resolving DNA secondary structure.Interaction interface of human flap endonuclease-1 with its DNA substrates.Roles for the Rad27 Flap Endonuclease in Mitochondrial Mutagenesis and Double-Strand Break Repair in Saccharomyces cerevisiae.
P2860
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P2860
Novel function of Rad27 (FEN-1) in restricting short-sequence recombination.
description
2001 nî lūn-bûn
@nan
2001 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2001年の論文
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2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
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name
Novel function of Rad27 (FEN-1) in restricting short-sequence recombination.
@ast
Novel function of Rad27 (FEN-1) in restricting short-sequence recombination.
@en
type
label
Novel function of Rad27 (FEN-1) in restricting short-sequence recombination.
@ast
Novel function of Rad27 (FEN-1) in restricting short-sequence recombination.
@en
prefLabel
Novel function of Rad27 (FEN-1) in restricting short-sequence recombination.
@ast
Novel function of Rad27 (FEN-1) in restricting short-sequence recombination.
@en
P2093
P2860
P1476
Novel function of Rad27 (FEN-1) in restricting short-sequence recombination.
@en
P2093
P2860
P304
P356
10.1128/MCB.21.7.2349-2358.2001
P407
P577
2001-04-01T00:00:00Z