Efficiency alleles of the Pctr1 modifier locus for plasmacytoma susceptibility
about
Preclinical validation of interleukin 6 as a therapeutic target in multiple myelomaMndal, a new interferon-inducible family member, is highly polymorphic, suppresses cell growth, and may modify plasmacytoma susceptibilityPitx2a expression alters actin-myosin cytoskeleton and migration of HeLa cells through Rho GTPase signalingIL-6 transgenic mouse model for extraosseous plasmacytoma.p16 INK4a gene promoter variation and differential binding of a repressor, the ras-responsive zinc-finger transcription factor, RREB.Novel targeted deregulation of c-Myc cooperates with Bcl-X(L) to cause plasma cell neoplasms in mice.Frap, FKBP12 rapamycin-associated protein, is a candidate gene for the plasmacytoma resistance locus Pctr2 and can act as a tumor suppressor geneObligate roles for p16(Ink4a) and p19(Arf)-p53 in the suppression of murine pancreatic neoplasia.Understanding the human condition: experimental strategies in mammalian genetics.Identification of susceptibility loci in a mouse model of KRASG12D-driven pancreatic cancer.Paradoxical expression of INK4c in proliferative multiple myeloma tumors: bi-allelic deletion vs increased expression.Cell-cycle control of plasma cell differentiation and tumorigenesis.Neoplastic development in plasma cells.Susceptibility to neoplastic and non-neoplastic pulmonary diseases in mice: genetic similarities.Target cell frequency is a genetically determined risk factor in radiation leukaemogenesis.p16(Ink4a) interferes with Abelson virus transformation by enhancing apoptosis.A novel rapid-onset high-penetrance plasmacytoma mouse model driven by deregulation of cMYC cooperating with KRAS12V in BALB/c mice.Increased gene dosage of Ink4a/Arf results in cancer resistance and normal aging.TORC1 and class I HDAC inhibitors synergize to suppress mature B cell neoplasms.Waldenström macroglobulinemia: clinical and immunological aspects, natural history, cell of origin, and emerging mouse modelsCanine Mammary Carcinomas: A Comparative Analysis of Altered Gene Expression.Loss of p16Ink4a with retention of p19Arf predisposes mice to tumorigenesis.A new locus for resistance to gamma-radiation-induced thymic lymphoma identified using inter-specific consomic and inter-specific recombinant congenic strains of mice.Early Generated B-1-Derived B Cells Have the Capacity To Progress To Become Mantle Cell Lymphoma-like Neoplasia in Aged Mice.
P2860
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P2860
Efficiency alleles of the Pctr1 modifier locus for plasmacytoma susceptibility
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Efficiency alleles of the Pctr1 modifier locus for plasmacytoma susceptibility
@ast
Efficiency alleles of the Pctr1 modifier locus for plasmacytoma susceptibility
@en
type
label
Efficiency alleles of the Pctr1 modifier locus for plasmacytoma susceptibility
@ast
Efficiency alleles of the Pctr1 modifier locus for plasmacytoma susceptibility
@en
prefLabel
Efficiency alleles of the Pctr1 modifier locus for plasmacytoma susceptibility
@ast
Efficiency alleles of the Pctr1 modifier locus for plasmacytoma susceptibility
@en
P2093
P2860
P1476
Efficiency alleles of the Pctr1 modifier locus for plasmacytoma susceptibility
@en
P2093
E S Ramsay
L Taddesse-Heath
R A DePinho
V Bliskovski
P2860
P304
P356
10.1128/MCB.21.1.310-318.2001
P407
P577
2001-01-01T00:00:00Z