Synergistic role of Sprouty2 inactivation and c-Met up-regulation in mouse and human hepatocarcinogenesis.
about
JNK and STAT3 signaling pathways converge on Akt-mediated phosphorylation of EZH2 in bronchial epithelial cells induced by arsenicNEDD4 controls the expression of GUCD1, a protein upregulated in proliferating liver cells.The developing story of Sprouty and cancer.c-Met represents a potential therapeutic target for personalized treatment in hepatocellular carcinomaRegulation of cellular levels of Sprouty2 protein by prolyl hydroxylase domain and von Hippel-Lindau proteinsDown-regulation of CMTM8 induces epithelial-to-mesenchymal transition-like changes via c-MET/extracellular signal-regulated kinase (ERK) signaling.Hypoxia inducible factors regulate the transcription of the sprouty2 gene and expression of the sprouty2 proteinCo-activation of AKT and c-Met triggers rapid hepatocellular carcinoma development via the mTORC1/FASN pathway in mice.[(11)C]acetate PET Imaging is not Always Associated with Increased Lipogenesis in Hepatocellular Carcinoma in MiceHydrodynamic transfection for generation of novel mouse models for liver cancer research.Fibroblast growth factor signaling in liver carcinogenesis.Sprouty2 Protein Regulates Hypoxia-inducible Factor-α (HIFα) Protein Levels and Transcription of HIFα-responsive Genes.Inactivation of Spry2 accelerates AKT-driven hepatocarcinogenesis via activation of MAPK and PKM2 pathways.EGFR activation is a potential determinant of primary resistance of hepatocellular carcinoma cells to sorafenib.Functional crosstalk between AKT/mTOR and Ras/MAPK pathways in hepatocarcinogenesis: implications for the treatment of human liver cancer.Pathogenetic and Prognostic Significance of Inactivation of RASSF Proteins in Human Hepatocellular Carcinoma.Liver proliferation: the GUCD1/NEDD4-1 connection.An in vivo transfection system for inducible gene expression and gene silencing in murine hepatocytes.Glucose Catabolism in Liver Tumors Induced by c-MYC Can Be Sustained by Various PKM1/PKM2 Ratios and Pyruvate Kinase Activities.Loss of Pten synergizes with c-Met to promote hepatocellular carcinoma development via mTORC2 pathway.SPRY2 is a novel MET interactor that regulates metastatic potential and differentiation in rhabdomyosarcoma.Oncogenic potential of N-terminal deletion and S45Y mutant β-catenin in promoting hepatocellular carcinoma development in mice
P2860
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P2860
Synergistic role of Sprouty2 inactivation and c-Met up-regulation in mouse and human hepatocarcinogenesis.
description
2010 nî lūn-bûn
@nan
2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Synergistic role of Sprouty2 i ...... nd human hepatocarcinogenesis.
@ast
Synergistic role of Sprouty2 i ...... nd human hepatocarcinogenesis.
@en
Synergistic role of Sprouty2 i ...... nd human hepatocarcinogenesis.
@nl
type
label
Synergistic role of Sprouty2 i ...... nd human hepatocarcinogenesis.
@ast
Synergistic role of Sprouty2 i ...... nd human hepatocarcinogenesis.
@en
Synergistic role of Sprouty2 i ...... nd human hepatocarcinogenesis.
@nl
prefLabel
Synergistic role of Sprouty2 i ...... nd human hepatocarcinogenesis.
@ast
Synergistic role of Sprouty2 i ...... nd human hepatocarcinogenesis.
@en
Synergistic role of Sprouty2 i ...... nd human hepatocarcinogenesis.
@nl
P2093
P2860
P356
P1433
P1476
Synergistic role of Sprouty2 i ...... nd human hepatocarcinogenesis.
@en
P2093
Diego F Calvisi
Frank Dombrowski
Matthias Evert
Susie A Lee
Valentina De Murtas
P2860
P304
P356
10.1002/HEP.23681
P407
P577
2010-08-01T00:00:00Z