The genetic basis of fluconazole resistance development in Candida albicans.
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Mechanisms of Candida biofilm drug resistanceCryptococcus gattii infectionsA tetraploid intermediate precedes aneuploid formation in yeasts exposed to fluconazoleArchitecture of a single membrane spanning cytochrome P450 suggests constraints that orient the catalytic domain relative to a bilayerMolecular mechanism of terbinafine resistance in Saccharomyces cerevisiaeThe transcription factor Mrr1p controls expression of the MDR1 efflux pump and mediates multidrug resistance in Candida albicansReverse genetics in Candida albicans predicts ARF cycling is essential for drug resistance and virulenceCandida albicans AGE3, the ortholog of the S. cerevisiae ARF-GAP-encoding gene GCS1, is required for hyphal growth and drug resistanceAzole drugs are imported by facilitated diffusion in Candida albicans and other pathogenic fungiThe transcription factor Ndt80 does not contribute to Mrr1-, Tac1-, and Upc2-mediated fluconazole resistance in Candida albicansComparative lipidomics in clinical isolates of Candida albicans reveal crosstalk between mitochondria, cell wall integrity and azole resistanceAntifungal Therapy: New Advances in the Understanding and Treatment of MycosisInhibition of efflux transporter-mediated fungicide resistance in Pyrenophora tritici-repentis by a derivative of 4'-hydroxyflavone and enhancement of fungicide activity.Rapid detection of ERG11 gene mutations in clinical Candida albicans isolates with reduced susceptibility to fluconazole by rolling circle amplification and DNA sequencing.An A643T mutation in the transcription factor Upc2p causes constitutive ERG11 upregulation and increased fluconazole resistance in Candida albicansIn vitro susceptibility of Malassezia pachydermatis isolates from canine skin with atopic dermatitis to ketoconazole and itraconazole in East Asia.In vitro antifungal activities of Euphorbia macroclada and fluconazole against pathogenic Candida species.Comparative lipidomics of azole sensitive and resistant clinical isolates of Candida albicans reveals unexpected diversity in molecular lipid imprints.Genome-wide expression profile analysis reveals coordinately regulated genes associated with stepwise acquisition of azole resistance in Candida albicans clinical isolates.Functional dissection of a Candida albicans zinc cluster transcription factor, the multidrug resistance regulator Mrr1.Development of fluconazole resistance in a series of Candida parapsilosis isolates from a persistent candidemia patient with prolonged antifungal therapyArv1 lipid transporter function is conserved between pathogenic and nonpathogenic fungiA Candida albicans petite mutant strain with uncoupled oxidative phosphorylation overexpresses MDR1 and has diminished susceptibility to fluconazole and voriconazoleEpidemiology and antifungal resistance in invasive candidiasis.Genome-wide expression and location analyses of the Candida albicans Tac1p regulon.Gain-of-function mutations in UPC2 are a frequent cause of ERG11 upregulation in azole-resistant clinical isolates of Candida albicans.Lipidomics and in vitro azole resistance in Candida albicans.In search of the holy grail of antifungal therapyA gain-of-function mutation in the transcription factor Upc2p causes upregulation of ergosterol biosynthesis genes and increased fluconazole resistance in a clinical Candida albicans isolate.Transcriptomics Analysis of Candida albicans Treated with Huanglian Jiedu Decoction Using RNA-seq.Survey of the Antibiofilm and Antimicrobial Effects of Zingiber officinale (in Vitro Study)Gain-of-function mutations in the transcription factor MRR1 are responsible for overexpression of the MDR1 efflux pump in fluconazole-resistant Candida dubliniensis strains.Gene Expression and Identification Related to Fluconazole Resistance of Candida glabrata Strains.Mutations in the multi-drug resistance regulator MRR1, followed by loss of heterozygosity, are the main cause of MDR1 overexpression in fluconazole-resistant Candida albicans strains.Identification of the Candida albicans Cap1p regulon.An MDR1 promoter allele with higher promoter activity is common in clinically isolated strains of Candida albicansEfficacy of PLD-118, a novel inhibitor of candida isoleucyl-tRNA synthetase, against experimental oropharyngeal and esophageal candidiasis caused by fluconazole-resistant C. albicans.Detection of species diversity in oral candida colonization and anti-fungal susceptibility among non-oral habit adult diabetic patients.Membrane sphingolipid-ergosterol interactions are important determinants of multidrug resistance in Candida albicans.Multidrug resistance in fungi: regulation of transporter-encoding gene expression.
P2860
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P2860
The genetic basis of fluconazole resistance development in Candida albicans.
description
2002 nî lūn-bûn
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2002 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
The genetic basis of fluconazole resistance development in Candida albicans.
@ast
The genetic basis of fluconazole resistance development in Candida albicans.
@en
The genetic basis of fluconazole resistance development in Candida albicans.
@nl
type
label
The genetic basis of fluconazole resistance development in Candida albicans.
@ast
The genetic basis of fluconazole resistance development in Candida albicans.
@en
The genetic basis of fluconazole resistance development in Candida albicans.
@nl
prefLabel
The genetic basis of fluconazole resistance development in Candida albicans.
@ast
The genetic basis of fluconazole resistance development in Candida albicans.
@en
The genetic basis of fluconazole resistance development in Candida albicans.
@nl
P1476
The genetic basis of fluconazole resistance development in Candida albicans.
@en
P2093
Joachim Morschhäuser
P304
P407
P577
2002-07-01T00:00:00Z