Death and survival signals determine active/inactive conformations of pro-apoptotic BAX, BAD, and BID molecules. - Wikidata - wikimedia - TriplyDB

Death and survival signals determine active/inactive conformations of pro-apoptotic BAX, BAD, and BID molecules.

Death and survival signals determine active/inactive conformations of pro-apoptotic BAX, BAD, and BID molecules.

http://www.wikidata.org/entity/Q34170796

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Transforming growth factor beta 1 induces apoptosis through cleavage of BAD in a Smad3-dependent mechanism in FaO hepatoma cells Bax shuttling after neonatal hypoxia-ischemia: hyperoxia effects.Fas-associated protein with death domain (FADD)-independent recruitment of c-FLIPL to death receptor 5.Host Responses and Regulation by NFκB Signaling in the Liver and Liver Epithelial Cells Infected with A Novel Tick-borne Bunyavirus.Emerging Bcl-2 inhibitors for the treatment of cancer.Bcl-2 family members as prognostic indicators in AML.Neurotoxicity of a Biopesticide Analog on Zebrafish Larvae at Nanomolar Concentrations.Vasopressin inhibits apoptosis in renal collecting duct cells.Functional hepatocyte culture and its application to cell therapies.Embedded together: the life and death consequences of interaction of the Bcl-2 family with membranes.Regulation of tumor suppressor EAF2 polyubiquitination by ELL1 and SIAH2 in prostate cancer cells Inhibition of Bim enhances replication of varicella-zoster virus and delays plaque formation in virus-infected cells.Methylseleninic acid sensitizes prostate cancer cells to TRAIL-mediated apoptosis.Proteasome inhibitor MG132 upregulates death receptor 5 and cooperates with Apo2L/TRAIL to induce apoptosis in Bax-proficient and -deficient cells.Polyamine depletion prevents camptothecin-induced apoptosis by inhibiting the release of cytochrome c.Granzyme B induces BID-mediated cytochrome c release and mitochondrial permeability transition.Pro-survival function of Akt/protein kinase B in prostate cancer cells. Relationship with TRAIL resistance.Constitutively active Akt is an important regulator of TRAIL sensitivity in prostate cancer.Knockdown of SLC39A7 suppresses cell proliferation, migration and invasion in cervical cancer.MOMP, cell suicide as a BCL-2 family business.Neuroimmune regulation of ventilator-induced lung injury.

P2860

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P2860

Death and survival signals determine active/inactive conformations of pro-apoptotic BAX, BAD, and BID molecules.

http://www.wikidata.org/entity/Q34170796

description

1999 nî lūn-bûn

@nan

1999 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

1999 թվականի հունվարին հրատարակված գիտական հոդված

@hy

1999年の論文

@ja

1999年論文

@yue

1999年論文

@zh-hant

1999年論文

@zh-hk

1999年論文

@zh-mo

1999年論文

@zh-tw

1999年论文

@wuu

name

Death and survival signals det ...... c BAX, BAD, and BID molecules.

@ast

Death and survival signals det ...... c BAX, BAD, and BID molecules.

@en

Death and survival signals det ...... c BAX, BAD, and BID molecules.

@nl

type

label

Death and survival signals det ...... c BAX, BAD, and BID molecules.

@ast

Death and survival signals det ...... c BAX, BAD, and BID molecules.

@en

Death and survival signals det ...... c BAX, BAD, and BID molecules.

@nl

prefLabel

Death and survival signals det ...... c BAX, BAD, and BID molecules.

@ast

Death and survival signals det ...... c BAX, BAD, and BID molecules.

@en

Death and survival signals det ...... c BAX, BAD, and BID molecules.

@nl

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P356

SQB.1999.64.343

P1433

Cold Spring Harbor Laboratory. Symposia on Quantitative Biology

P1476

Death and survival signals det ...... c BAX, BAD, and BID molecules.

@en

P2093

Gross A

http://www.w3.org/2001/XMLSchema#string

Harada H

http://www.w3.org/2001/XMLSchema#string

Korsmeyer SJ

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Wang K

http://www.w3.org/2001/XMLSchema#string

Wei M

http://www.w3.org/2001/XMLSchema#string

Yin XM

http://www.w3.org/2001/XMLSchema#string

Zha J

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P2860

Dimerization properties of human BAD. Identification of a BH-3 domain and analysis of its binding to mutant BCL-2 and BCL-XL proteins

Inhibition of Bax channel-forming activity by Bcl-2

Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis

Molecular ordering of the Fas-apoptotic pathway: the Fas/APO-1 protease Mch5 is a CrmA-inhibitable protease that activates multiple Ced-3/ICE-like cysteine proteases

Cytosol-to-membrane redistribution of Bax and Bcl-X(L) during apoptosis

Cell damage-induced conformational changes of the pro-apoptotic protein Bak in vivo precede the onset of apoptosis

Movement of Bax from the cytosol to mitochondria during apoptosis

Solution structure of BID, an intracellular amplifier of apoptotic signaling

Solution structure of the proapoptotic molecule BID: a structural basis for apoptotic agonists and antagonists

P304

343-350

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scholarly article

P356

10.1101/SQB.1999.64.343

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P478

64

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P50

Sandra S. Zinkel

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1999-01-01T00:00:00Z

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P698

11232306

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P921

apoptotic process