Cutting edge: The transcription factor eomesodermin enables CD8+ T cells to compete for the memory cell niche. - Wikidata - wikimedia - TriplyDB

Cutting edge: The transcription factor eomesodermin enables CD8+ T cells to compete for the memory cell niche.

Cutting edge: The transcription factor eomesodermin enables CD8+ T cells to compete for the memory cell niche.

http://www.wikidata.org/entity/Q34286922

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T-bet and Eomesodermin in NK Cell Development, Maturation, and Function Bone Marrow T Cells and the Integrated Functions of Recirculating and Tissue-Resident Memory T Cells TCR Signaling in T Cell Memory Molecular and cellular insights into T cell exhaustion Molecular characterization of HCMV-specific immune responses: Parallels between CD8(+) T cells, CD4(+) T cells, and NK cells Overcoming T cell exhaustion in infection and cancer Primer on tumor immunology and cancer immunotherapy The effects of TLR activation on T-cell development and differentiation Dendritic cell-based vaccination in cancer: therapeutic implications emerging from murine models microRNAs function in CD8+T cell biology Blimp-1 homolog Hobit identifies effector-type lymphocytes in humans The POSH/JIP-1 scaffold network regulates TCR-mediated JNK1 signals and effector function in CD8(+) T cells.CD8(+) T-cell effector function and transcriptional regulation during HIV pathogenesis Characterization of T-bet and eomes in peripheral human immune cells T-bet and Eomes are differentially linked to the exhausted phenotype of CD8+ T cells in HIV infection Generation and characterization of a tamoxifen-inducible Eomes(CreER) mouse line.Transcriptional control of effector and memory CD8+ T cell differentiation.IL-12 is required for mTOR regulation of memory CTLs during viral infection.CD40-activated B cells can efficiently prime antigen-specific naïve CD8+ T cells to generate effector but not memory T cells.Cutting edge: Lymphoproliferation caused by Fas deficiency is dependent on the transcription factor eomesodermin.CD4⁺ and CD8⁺ T cell-dependent antiviral immunity requires STIM1 and STIM2.mRNA Structural constraints on EBNA1 synthesis impact on in vivo antigen presentation and early priming of CD8+ T cells.T cells lacking HDAC11 have increased effector functions and mediate enhanced alloreactivity in a murine model.Cytokines and the inception of CD8 T cell responses.T-Bet and Eomes Regulate the Balance between the Effector/Central Memory T Cells versus Memory Stem Like T Cells.Clonal evolution of CD8+ T cell responses against latent viruses: relationship among phenotype, localization, and function.Specificity and dynamics of effector and memory CD8 T cell responses in human tick-borne encephalitis virus infection.T-bet is critical for the development of acute graft-versus-host disease through controlling T cell differentiation and function HLA-A02:01-restricted epitopes identified from the herpes simplex virus tegument protein VP11/12 preferentially recall polyfunctional effector memory CD8+ T cells from seropositive asymptomatic individuals and protect humanized HLA-A*02:01 transgeni Heteroclitic XBP1 peptides evoke tumor-specific memory cytotoxic T lymphocytes against breast cancer, colon cancer, and pancreatic cancer cells.Optomotor-blind in the development of the Drosophila HS and VS lobula plate tangential cells.Molecular regulation of effector and memory T cell differentiation Infection history determines the differentiation state of human CD8+ T cells T cell exhaustion during persistent viral infections.Type I interferons regulate eomesodermin expression and the development of unconventional memory CD8(+) T cells.IL-4 acts as a potent stimulator of IFN-γ expression in CD8+ T cells through STAT6-dependent and independent induction of Eomesodermin and T-bet.mTORC1 and mTORC2 selectively regulate CD8⁺ T cell differentiation.Protective antifungal memory CD8(+) T cells are maintained in the absence of CD4(+) T cell help and cognate antigen in mice.IL-4 Induced Innate CD8+ T Cells Control Persistent Viral Infection.Phenotypic and Functional Alterations in Circulating Memory CD8 T Cells with Time after Primary Infection.

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Cutting edge: The transcription factor eomesodermin enables CD8+ T cells to compete for the memory cell niche.

http://www.wikidata.org/entity/Q34286922

description

2010 nî lūn-bûn

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2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Cutting edge: The transcriptio ...... ete for the memory cell niche.

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Cutting edge: The transcriptio ...... ete for the memory cell niche.

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Cutting edge: The transcriptio ...... ete for the memory cell niche.

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Cutting edge: The transcriptio ...... ete for the memory cell niche.

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Cutting edge: The transcriptio ...... ete for the memory cell niche.

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Cutting edge: The transcriptio ...... ete for the memory cell niche.

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Cutting edge: The transcriptio ...... ete for the memory cell niche.

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Cutting edge: The transcriptio ...... ete for the memory cell niche.

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Cutting edge: The transcriptio ...... ete for the memory cell niche.

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JIMMUNOL.1002042

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Journal of Immunology

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Cutting edge: The transcriptio ...... pete for the memory cell niche

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Arnob Banerjee

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E John Wherry

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Erin C Mooney

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Scott M Gordon

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Steven L Reiner

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Tulia Lindsten

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P2860

Lineage relationship and protective immunity of memory CD8 T cell subsets

Control of effector CD8+ T cell function by the transcription factor Eomesodermin

Central memory and effector memory T cell subsets: function, generation, and maintenance

Effector and memory CD8+ T cell fate coupled by T-bet and eomesodermin.

Antigen-driven effector CD8 T cell function regulated by T-bet.

Inflammation directs memory precursor and short-lived effector CD8(+) T cell fates via the graded expression of T-bet transcription factor.

Requirement for T-bet in the aberrant differentiation of unhelped memory CD8+ T cells.

CD8 cell division maintaining cytotoxic memory occurs predominantly in the bone marrow.

Anomalous type 17 response to viral infection by CD8+ T cells lacking T-bet and eomesodermin.

The mTOR kinase determines effector versus memory CD8+ T cell fate by regulating the expression of transcription factors T-bet and Eomesodermin.

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4988-4992

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scholarly article

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10.4049/JIMMUNOL.1002042

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9

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185

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Michael A Paley

Andrew M Intlekofer

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2010-10-08T00:00:00Z

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47384386

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20935204

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2975552

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