oriP is essential for EBNA gene promoter activity in Epstein-Barr virus-immortalized lymphoblastoid cell lines.
about
Epstein-Barr virus nuclear antigen 3C putative repression domain mediates coactivation of the LMP1 promoter with EBNA-2.Epstein-Barr virus latent genesEpstein-Barr virus latency switch in human B-cells: a physico-chemical model.Zinc coordination is required for and regulates transcription activation by Epstein-Barr nuclear antigen 1Transcriptional activation signals found in the Epstein-Barr virus (EBV) latency C promoter are conserved in the latency C promoter sequences from baboon and Rhesus monkey EBV-like lymphocryptoviruses (cercopithicine herpesviruses 12 and 15).Murine gammaherpesvirus 68 encodes a functional regulator of complement activationThree distinct regions of the murine gammaherpesvirus 68 genome are transcriptionally active in latently infected mice.CTCF prevents the epigenetic drift of EBV latency promoter QpEpigenetic regulation of EBV persistence and oncogenesis.Chromatin organization of gammaherpesvirus latent genomes.The Epstein-Barr virus promoter initiating B-cell transformation is activated by RFX proteins and the B-cell-specific activator protein BSAP/Pax5Characterization of gammaherpesvirus 68 gene 50 transcriptionThe murine gammaherpesvirus 68 v-cyclin gene is an oncogene that promotes cell cycle progression in primary lymphocytes.DNA binding and modulation of gene expression by the latency-associated nuclear antigen of Kaposi's sarcoma-associated herpesvirusProtein-DNA binding and CpG methylation at nucleotide resolution of latency-associated promoters Qp, Cp, and LMP1p of Epstein-Barr virusEBV latency types adopt alternative chromatin conformations.Viral latency and its regulation: lessons from the gamma-herpesviruses.The spacing between adjacent binding sites in the family of repeats affects the functions of Epstein-Barr nuclear antigen 1 in transcription activation and stable plasmid maintenanceUnexpected instability of family of repeats (FR), the critical cis-acting sequence required for EBV latent infection, in EBV-BAC systems.Molecular virology of Epstein-Barr virus.EBNA3A association with RBP-Jkappa down-regulates c-myc and Epstein-Barr virus-transformed lymphoblast growth.Transcriptional activation by EBV nuclear antigen 1 is essential for the expression of EBV's transforming genes.trans-Repression of protein expression dependent on the Epstein-Barr virus promoter Wp during latency.Novel cell type-specific antiviral mechanism of interferon gamma action in macrophagesMetaphase chromosome tethering is necessary for the DNA synthesis and maintenance of oriP plasmids but is insufficient for transcription activation by Epstein-Barr nuclear antigen 1.Host-cell-determined methylation of specific Epstein-Barr virus promoters regulates the choice between distinct viral latency programsChromatin organization and virus gene expression.Epstein-Barr Nuclear Antigen 1 modulates replication of oriP-plasmids by impeding replication and transcription fork migration through the family of repeats.Identifying sites bound by Epstein-Barr virus nuclear antigen 1 (EBNA1) in the human genome: defining a position-weighted matrix to predict sites bound by EBNA1 in viral genomes.Epigenetic deregulation of the LMP1/LMP2 locus of Epstein-Barr virus by mutation of a single CTCF-cohesin binding site.Dynamic chromatin boundaries delineate a latency control region of Epstein-Barr virusVariable methylation of the Epstein-Barr virus Wp EBNA gene promoter in B-lymphoblastoid cell lines.Dynamic Epstein-Barr virus gene expression on the path to B-cell transformation.Methylation status of the Epstein-Barr virus (EBV) BamHI W latent cycle promoter and promoter activity: analysis with novel EBV-positive Burkitt and lymphoblastoid cell lines.Contributions of CTCF and DNA methyltransferases DNMT1 and DNMT3B to Epstein-Barr virus restricted latency.Genetic evidence that EBNA-1 is needed for efficient, stable latent infection by Epstein-Barr virusIdentification and initial characterization of the murine gammaherpesvirus 68 gene M3, encoding an abundantly secreted proteinUp to 100-fold increase of apparent gene expression in the presence of Epstein-Barr virus oriP sequences and EBNA1: implications of the nuclear import of plasmids.Methylation of transcription factor binding sites in the Epstein-Barr virus latent cycle promoter Wp coincides with promoter down-regulation during virus-induced B-cell transformation.Latent membrane protein 1 of Epstein-Barr virus inhibits as well as stimulates gene expression
P2860
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P2860
oriP is essential for EBNA gene promoter activity in Epstein-Barr virus-immortalized lymphoblastoid cell lines.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
oriP is essential for EBNA gen ...... zed lymphoblastoid cell lines.
@ast
oriP is essential for EBNA gen ...... zed lymphoblastoid cell lines.
@en
type
label
oriP is essential for EBNA gen ...... zed lymphoblastoid cell lines.
@ast
oriP is essential for EBNA gen ...... zed lymphoblastoid cell lines.
@en
prefLabel
oriP is essential for EBNA gen ...... zed lymphoblastoid cell lines.
@ast
oriP is essential for EBNA gen ...... zed lymphoblastoid cell lines.
@en
P2093
P2860
P1433
P1476
oriP is essential for EBNA gen ...... ized lymphoblastoid cell lines
@en
P2093
M T Puglielli
M Woisetschlaeger
P2860
P304
P407
P577
1996-09-01T00:00:00Z