Activation of macrophages for destruction of Francisella tularensis: identification of cytokines, effector cells, and effector molecules.
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TularemiaNeutralization of gamma interferon and tumor necrosis factor alpha blocks in vivo synthesis of nitrogen oxides from L-arginine and protection against Francisella tularensis infection in Mycobacterium bovis BCG-treated miceThe involvement of IL-17A in the murine response to sub-lethal inhalational infection with Francisella tularensisMicroarray analysis of human monocytes infected with Francisella tularensis identifies new targets of host response subversionEffective, broad spectrum control of virulent bacterial infections using cationic DNA liposome complexes combined with bacterial antigensMonophosphoryl Lipid A Enhances Efficacy of a Francisella tularensis LVS-Catanionic Nanoparticle Subunit Vaccine against F. tularensis Schu S4 Challenge by Augmenting both Humoral and Cellular Immunity.New protein fold revealed by a 1.65 A resolution crystal structure of Francisella tularensis pathogenicity island protein IglCRole of TLR signaling in Francisella tularensis-LPS-induced, antibody-mediated protection against Francisella tularensis challenge.Establishment of three Francisella infections in zebrafish embryos at different temperatures.Endotoxin-induced tumor necrosis factor alpha synthesis in murine embryo fibroblasts.Cytokine response in Balb/c mice infected with Francisella tularensis LVS and the Pohang isolateMucosal immunotherapy for protection from pneumonic infection with Francisella tularensisRestricted cytosolic growth of Francisella tularensis subsp. tularensis by IFN-gamma activation of macrophagesBactericidal activities of antibiotics against intracellular Francisella tularensis.Use of aminoglycosides in treatment of infections due to intracellular bacteriaRapid local expression of interleukin-12, tumor necrosis factor alpha, and gamma interferon after cutaneous Francisella tularensis infection in tularemia-immune mice.Effects of opsonization and gamma interferon on growth of Brucella melitensis 16M in mouse peritoneal macrophages in vitroInfluence of the bcg locus on natural resistance to primary infection with the facultative intracellular bacterium Francisella tularensis in miceFrancisella tularensis LVS induction of prostaglandin biosynthesis by infected macrophages requires specific host phospholipases and lipid phosphatasesGeneration of a convalescent model of virulent Francisella tularensis infection for assessment of host requirements for survival of tularemiaHost-pathogen interactions and immune evasion strategies in Francisella tularensis pathogenicityImportance of nitric oxide synthase in the control of infection by Bacillus anthracis.Association between virulence of Yersinia pestis and suppression of gamma interferon and tumor necrosis factor alpha.Neutrophils are critical for host defense against primary infection with the facultative intracellular bacterium Francisella tularensis in mice and participate in defense against reinfectionTransfer of immunity against lethal murine Francisella infection by specific antibody depends on host gamma interferon and T cells.CD4+ and CD8+ T-cell-dependent and -independent host defense mechanisms can operate to control and resolve primary and secondary Francisella tularensis LVS infection in miceIKKβ in myeloid cells controls the host response to lethal and sublethal Francisella tularensis LVS infection.The Francisella tularensis LVS ΔpdpC mutant exhibits a unique phenotype during intracellular infectionType A Francisella tularensis acid phosphatases contribute to pathogenesis.Administration of a synthetic TLR4 agonist protects mice from pneumonic tularemia.The mechanism of the inhibitory effect of polyamines on the induction of nitric oxide synthase: role of aldehyde metabolites.The francisella intracellular life cycle: toward molecular mechanisms of intracellular survival and proliferation.The subversion of the immune system by francisella tularensis.Tryptophan prototrophy contributes to Francisella tularensis evasion of gamma interferon-mediated host defense.Vaccines against tularemia.Role of pathogenicity determinant protein C (PdpC) in determining the virulence of the Francisella tularensis subspecies tularensis SCHU.A Francisella tularensis locus required for spermine responsiveness is necessary for virulence.Cytokine expression in the liver during the early phase of murine tularemia.Endogenous interleukin-12 is involved in resistance to Brucella abortus infection.Growth of Francisella tularensis LVS in macrophages: the acidic intracellular compartment provides essential iron required for growth
P2860
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P2860
Activation of macrophages for destruction of Francisella tularensis: identification of cytokines, effector cells, and effector molecules.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Activation of macrophages for ...... cells, and effector molecules.
@en
type
label
Activation of macrophages for ...... cells, and effector molecules.
@en
prefLabel
Activation of macrophages for ...... cells, and effector molecules.
@en
P2093
P2860
P1476
Activation of macrophages for ...... cells, and effector molecules.
@en
P2093
P2860
P304
P407
P577
1992-03-01T00:00:00Z