Topoisomerase poisons activate the transcription factor NF-kappaB in ACH-2 and CEM cells.
about
The inhibitory effects of camptothecin, a topoisomerase I inhibitor, on collagen synthesis in fibroblasts from patients with systemic sclerosisSignals from within: the DNA-damage-induced NF-kappaB responseOxidative stress, inflammation, and cancer: how are they linked?Cell survival from chemotherapy depends on NF-kappaB transcriptional up-regulation of coenzyme Q biosynthesis.ATM: a mediator of multiple responses to genotoxic stress.Transcriptomic and proteomic profiling of maize embryos exposed to camptothecinTopoisomerase I inhibition with topotecan: pharmacologic and clinical issues.NPRL-Z-1, as a new topoisomerase II poison, induces cell apoptosis and ROS generation in human renal carcinoma cells.Identification of a type I Ca2+/Mg2+-dependent endonuclease induced in maize cells exposed to camptothecinProcessing of topoisomerase I cleavable complexes into DNA damage by transcription.Nuclear initiated NF-κB signaling: NEMO and ATM take center stage.Identification of a novel pro-apopotic function of NF-kappaB in the DNA damage response.Discovery of novel immunostimulants by dendritic-cell-based functional screening.ATM and the catalytic subunit of DNA-dependent protein kinase activate NF-kappaB through a common MEK/extracellular signal-regulated kinase/p90(rsk) signaling pathway in response to distinct forms of DNA damage.DNA damage-dependent NF-κB activation: NEMO turns nuclear signaling inside out.Cellular-signaling pathways unveil the carcinogenic potential of chemicals.NF-kappaB activation by camptothecin. A linkage between nuclear DNA damage and cytoplasmic signaling events.SUMO and the robustness of cancer.The transcriptional inhibitors, actinomycin D and alpha-amanitin, activate the HIV-1 promoter and favor phosphorylation of the RNA polymerase II C-terminal domain.Oxidative stress and lipid peroxidation products in cancer progression and therapy.Nuclear and non-nuclear targets of genotoxic agents in the induction of gene expression. Shared principles in yeast, rodents, man and plants.Mechanism of cytosine arabinoside-mediated apoptosis: role of Rel A (p65) dephosphorylation.The ATM protein is required for sustained activation of NF-kappaB following DNA damage.Imaging of nuclear factor κB activation induced by ionizing radiation in human embryonic kidney (HEK) cells.Survivin antagonizes chemotherapy-induced cell death of colorectal cancer cells.
P2860
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P2860
Topoisomerase poisons activate the transcription factor NF-kappaB in ACH-2 and CEM cells.
description
1996 nî lūn-bûn
@nan
1996 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1996 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
name
Topoisomerase poisons activate the transcription factor NF-kappaB in ACH-2 and CEM cells.
@ast
Topoisomerase poisons activate the transcription factor NF-kappaB in ACH-2 and CEM cells.
@en
Topoisomerase poisons activate the transcription factor NF-kappaB in ACH-2 and CEM cells.
@nl
type
label
Topoisomerase poisons activate the transcription factor NF-kappaB in ACH-2 and CEM cells.
@ast
Topoisomerase poisons activate the transcription factor NF-kappaB in ACH-2 and CEM cells.
@en
Topoisomerase poisons activate the transcription factor NF-kappaB in ACH-2 and CEM cells.
@nl
prefLabel
Topoisomerase poisons activate the transcription factor NF-kappaB in ACH-2 and CEM cells.
@ast
Topoisomerase poisons activate the transcription factor NF-kappaB in ACH-2 and CEM cells.
@en
Topoisomerase poisons activate the transcription factor NF-kappaB in ACH-2 and CEM cells.
@nl
P2860
P356
P1476
Topoisomerase poisons activate the transcription factor NF-kappaB in ACH-2 and CEM cells.
@en
P2093
P2860
P304
P356
10.1093/NAR/24.21.4242
P407
P577
1996-11-01T00:00:00Z