Replication stress induces 53BP1-containing OPT domains in G1 cells. - Wikidata - wikimedia - TriplyDB

Replication stress induces 53BP1-containing OPT domains in G1 cells.

Replication stress induces 53BP1-containing OPT domains in G1 cells.

http://www.wikidata.org/entity/Q34854577

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Radiation-induced alterations in histone modification patterns and their potential impact on short-term radiation effects The proteasomal de-ubiquitinating enzyme POH1 promotes the double-strand DNA break response Ubiquitin-activating enzyme UBA1 is required for cellular response to DNA damage Maintaining Genome Stability in Defiance of Mitotic DNA Damage PR-Set7 and H4K20me1: at the crossroads of genome integrity, cell cycle, chromosome condensation, and transcription Role of DNA polymerase κ in the maintenance of genomic stability 53BP1: pro choice in DNA repair Replication stress: getting back on track Replication stress in Mammalian cells and its consequences for mitosis Initiation of genome instability and preneoplastic processes through loss of Fhit expression Mechanism of suppression of chromosomal instability by DNA polymerase POLQ Double strand break repair functions of histone H2AX microRNA-34a promotes DNA damage and mitotic catastrophe.Senataxin, defective in the neurodegenerative disorder ataxia with oculomotor apraxia 2, lies at the interface of transcription and the DNA damage response.Dephosphorylation enables the recruitment of 53BP1 to double-strand DNA breaks.ATR and a Chk1-Aurora B pathway coordinate postmitotic genome surveillance with cytokinetic abscission.A concomitant loss of dormant origins and FANCC exacerbates genome instability by impairing DNA replication fork progression.Human Adipose-Derived Stem Cells Expanded Under Ambient Oxygen Concentration Accumulate Oxidative DNA Lesions and Experience Procarcinogenic DNA Replication Stress Increased FOXM1 expression can stimulate DNA repair in normal hepatocytes in vivo but also increases nuclear foci associated with senescence.Human pluripotent stem cells have a novel mismatch repair-dependent damage response.The contribution of mitochondrial thymidylate synthesis in preventing the nuclear genome stress Rescue from replication stress during mitosis.Hybrid detectors improved time-lapse confocal microscopy of PML and 53BP1 nuclear body colocalization in DNA lesions.Spartan deficiency causes genomic instability and progeroid phenotypes.Carbon-ion beam irradiation kills X-ray-resistant p53-null cancer cells by inducing mitotic catastrophe Noncovalent interactions with SUMO and ubiquitin orchestrate distinct functions of the SLX4 complex in genome maintenance.Engineered proteins detect spontaneous DNA breakage in human and bacterial cells.Replication fork integrity and intra-S phase checkpoint suppress gene amplification.TPX2 impacts acetylation of histone H4 at lysine 16: implications for DNA damage response.Advancing small-molecule-based chemical biology with next-generation sequencing technologies.A Synthetic Lethal Screen Identifies DNA Repair Pathways that Sensitize Cancer Cells to Combined ATR Inhibition and Cisplatin Treatments.Rescue of replication failure by Fanconi anaemia proteins.Insights into phosphorylation-dependent mechanisms regulating USP1 protein stability during the cell cycle.Dysregulation of DNA polymerase κ recruitment to replication forks results in genomic instability.Nucleoporin NUP153 guards genome integrity by promoting nuclear import of 53BP1 BCL10 is recruited to sites of DNA damage to facilitate DNA double-strand break repair Chromatin dynamics in DNA double-strand break repair DNA polymerase zeta is required for proliferation of normal mammalian cells.SPOC1 modulates DNA repair by regulating key determinants of chromatin compaction and DNA damage response.Mammalian RAD51 paralogs protect nascent DNA at stalled forks and mediate replication restart.

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P2860

Replication stress induces 53BP1-containing OPT domains in G1 cells.

http://www.wikidata.org/entity/Q34854577

description

2011 nî lūn-bûn

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2011 թուականի Մարտին հրատարակուած գիտական յօդուած

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2011 թվականի մարտին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Replication stress induces 53BP1-containing OPT domains in G1 cells.

@ast

Replication stress induces 53BP1-containing OPT domains in G1 cells.

@en

Replication stress induces 53BP1-containing OPT domains in G1 cells.

@nl

type

label

Replication stress induces 53BP1-containing OPT domains in G1 cells.

@ast

Replication stress induces 53BP1-containing OPT domains in G1 cells.

@en

Replication stress induces 53BP1-containing OPT domains in G1 cells.

@nl

prefLabel

Replication stress induces 53BP1-containing OPT domains in G1 cells.

@ast

Replication stress induces 53BP1-containing OPT domains in G1 cells.

@en

Replication stress induces 53BP1-containing OPT domains in G1 cells.

@nl

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Journal of Cell Biology

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Replication stress induces 53BP1-containing OPT domains in G1 cells.

@en

P2093

Daniela S Dimitrova

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Jeanine A Harrigan

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Julia Coates

http://www.w3.org/2001/XMLSchema#string

Rimma Belotserkovskaya

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P2860

Model-based analysis of ChIP-Seq (MACS)

Regional and temporal specialization in the nucleus: a transcriptionally-active nuclear domain rich in PTF, Oct1 and PIKA antigens associates with specific chromosomes early in the cell cycle.

RNF168 binds and amplifies ubiquitin conjugates on damaged chromosomes to allow accumulation of repair proteins

ATM-dependent chromatin changes silence transcription in cis to DNA double-strand breaks

Poly(ADP-ribose)-dependent regulation of DNA repair by the chromatin remodeling enzyme ALC1

A role for DEAD box 1 at DNA double-strand breaks

Phospho-dependent interactions between NBS1 and MDC1 mediate chromatin retention of the MRN complex at sites of DNA damage

53BP1 loss rescues BRCA1 deficiency and is associated with triple-negative and BRCA-mutated breast cancers

Mammalian SUMO E3-ligases PIAS1 and PIAS4 promote responses to DNA double-strand breaks

The POU domains of the Oct1 and Oct2 transcription factors mediate specific interaction with TBP

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97-108

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scholarly article

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10.1083/JCB.201011083

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P433

1

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193

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Stephen P Jackson

Charles R Bradshaw

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2011-03-28T00:00:00Z

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50891771

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21444690

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3082192

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