Characterization of glucose-insulin responsiveness and impact of fetal number and sex difference on insulin response in the sheep fetus.
about
A physiological increase in insulin suppresses muscle-specific ubiquitin ligase gene activation in fetal sheep with sustained hypoglycemiaThe pancreas is altered by in utero androgen exposure: implications for clinical conditions such as polycystic ovary syndrome (PCOS).Increased adrenergic signaling is responsible for decreased glucose-stimulated insulin secretion in the chronically hyperinsulinemic ovine fetusChronic pulsatile hyperglycemia reduces insulin secretion and increases accumulation of reactive oxygen species in fetal sheep islets.Increased amino acid supply potentiates glucose-stimulated insulin secretion but does not increase β-cell mass in fetal sheep.Reductions in insulin concentrations and β-cell mass precede growth restriction in sheep fetuses with placental insufficiencyAdrenal Demedullation and Oxygen Supplementation Independently Increase Glucose-Stimulated Insulin Concentrations in Fetal Sheep With Intrauterine Growth RestrictionElevated plasma norepinephrine inhibits insulin secretion, but adrenergic blockade reveals enhanced β-cell responsiveness in an ovine model of placental insufficiency at 0.7 of gestationEnhanced insulin secretion responsiveness and islet adrenergic desensitization after chronic norepinephrine suppression is discontinued in fetal sheep.Attenuation of ductus arteriosus intimal thickening in preterm sheep twins compared with singletons.RNA Sequencing Exposes Adaptive and Immune Responses to Intrauterine Growth Restriction in Fetal Sheep Islets.Differential effects of chronic pulsatile versus chronic constant maternal hyperglycemia on fetal pancreatic β-cells.Chronically Increased Amino Acids Improve Insulin Secretion, Pancreatic Vascularity, and Islet Size in Growth-Restricted Fetal Sheep.Islet adaptations in fetal sheep persist following chronic exposure to high norepinephrine.Postnatal effects of intrauterine treatment of the growth-restricted ovine fetus with intra-amniotic insulin-like growth factor-1.Size at birth and adult fat mass in twin sheep are determined in early gestation.Chronic anemic hypoxemia attenuates glucose-stimulated insulin secretion in fetal sheep.Hypoxaemia-induced catecholamine secretion from adrenal chromaffin cells inhibits glucose-stimulated hyperinsulinaemia in fetal sheep.
P2860
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P2860
Characterization of glucose-insulin responsiveness and impact of fetal number and sex difference on insulin response in the sheep fetus.
description
2011 nî lūn-bûn
@nan
2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Characterization of glucose-in ...... n response in the sheep fetus.
@ast
Characterization of glucose-in ...... n response in the sheep fetus.
@en
Characterization of glucose-in ...... n response in the sheep fetus.
@nl
type
label
Characterization of glucose-in ...... n response in the sheep fetus.
@ast
Characterization of glucose-in ...... n response in the sheep fetus.
@en
Characterization of glucose-in ...... n response in the sheep fetus.
@nl
prefLabel
Characterization of glucose-in ...... n response in the sheep fetus.
@ast
Characterization of glucose-in ...... n response in the sheep fetus.
@en
Characterization of glucose-in ...... n response in the sheep fetus.
@nl
P2093
P2860
P1476
Characterization of glucose-in ...... n response in the sheep fetus.
@en
P2093
Alice S Green
Antoni R Macko
Dustin T Yates
Paul J Rozance
Sean W Limesand
William W Hay
Xiaochuan Chen
P2860
P304
P356
10.1152/AJPENDO.00572.2010
P577
2011-02-22T00:00:00Z