Consequences of a compromised intrauterine environment on islet function.
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The Elevated Susceptibility to Diabetes in India: An Evolutionary PerspectiveEnvironmental Contaminants and Pancreatic Beta-CellsComplications during pregnancy and fetal development: implications for the occurrence of chronic kidney disease.Postnatal pancreatic islet β cell function and insulin sensitivity at different stages of lifetime in rats born with intrauterine growth retardation.Maternal obesity in ewes results in reduced fetal pancreatic β-cell numbers in late gestation and decreased circulating insulin concentration at term.Increased adrenergic signaling is responsible for decreased glucose-stimulated insulin secretion in the chronically hyperinsulinemic ovine fetusCharacterization of glucose-insulin responsiveness and impact of fetal number and sex difference on insulin response in the sheep fetus.Ethnic differences in neonatal body composition in a multi-ethnic population and the impact of parental factors: a population-based cohort study.Fetal adrenal demedullation lowers circulating norepinephrine and attenuates growth restriction but not reduction of endocrine cell mass in an ovine model of intrauterine growth restrictionCatecholamines mediate multiple fetal adaptations during placental insufficiency that contribute to intrauterine growth restriction: lessons from hyperthermic sheep.The intrauterine growth restriction phenotype: fetal adaptations and potential implications for later life insulin resistance and diabetes.Insulin-like growth factor and fibroblast growth factor expression profiles in growth-restricted fetal sheep pancreas.Chronic pulsatile hyperglycemia reduces insulin secretion and increases accumulation of reactive oxygen species in fetal sheep islets.Increased fetal insulin concentrations for one week fail to improve insulin secretion or β-cell mass in fetal sheep with chronically reduced glucose supplyReductions in insulin concentrations and β-cell mass precede growth restriction in sheep fetuses with placental insufficiencyMaternal nutrient restriction predisposes ventricular remodeling in adult sheep offspring.Elevated plasma norepinephrine inhibits insulin secretion, but adrenergic blockade reveals enhanced β-cell responsiveness in an ovine model of placental insufficiency at 0.7 of gestationEnhanced insulin secretion responsiveness and islet adrenergic desensitization after chronic norepinephrine suppression is discontinued in fetal sheep.Early-Life Nutritional Programming of Type 2 Diabetes: Experimental and Quasi-Experimental Evidence.Are twins growth restricted?Pancreatic islet hepatocyte growth factor and vascular endothelial growth factor A signaling in growth restricted fetuses.Challenges in nourishing the intrauterine growth-restricted foetus - Lessons learned from studies in the intrauterine growth-restricted foetal sheep.RNA Sequencing Exposes Adaptive and Immune Responses to Intrauterine Growth Restriction in Fetal Sheep Islets.Dysregulation of calcium channels decreases parasecretion in pancreatic β-cells in rats born small for gestational age.Maternal fat mass at mid-pregnancy and birth weight in Brazilian women.Effects of Intrauterine Growth Restriction During Late Pregnancy on the Development of the Ovine Fetal Thymus and the T-Lymphocyte Subpopulation.Chronically Increased Amino Acids Improve Insulin Secretion, Pancreatic Vascularity, and Islet Size in Growth-Restricted Fetal Sheep.Fetal adaptations in insulin secretion result from high catecholamines during placental insufficiency.β2-Adrenergic receptor desensitization in perirenal adipose tissue in fetuses and lambs with placental insufficiency-induced intrauterine growth restriction.Arginine nutrition and fetal brown adipose tissue development in nutrient-restricted sheep.Pre- and postnatal methyl deficiency in the rat differentially alters glucose homeostasis.Newborn umbilical cord blood DNA methylation and gene expression levels exhibit limited association with birth weight.Developmental programming: Interaction between prenatal BPA and postnatal overfeeding on cardiac tissue gene expression in female sheep.Neonatal milk supplementation in lambs has persistent effects on growth and metabolic function that differ by sex and gestational age.Ponderal index at birth associates with later risk of gestational diabetes mellitus.Programming of glucose-insulin homoeostasis: long-term consequences of pre-natal versus early post-natal nutrition insults. Evidence from a sheep model.The impact of IUGR on pancreatic islet development and β-cell function.Enhanced insulin secretion and insulin sensitivity in young lambs with placental insufficiency-induced intrauterine growth restriction.Brief neonatal nutritional supplementation has sex-specific effects on glucose tolerance and insulin regulating genes in juvenile lambs.Postnatal development of metabolic flexibility and enhanced oxidative capacity after prenatal undernutrition.
P2860
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P2860
Consequences of a compromised intrauterine environment on islet function.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh
2010年學術文章
@zh-hant
name
Consequences of a compromised intrauterine environment on islet function.
@ast
Consequences of a compromised intrauterine environment on islet function.
@en
type
label
Consequences of a compromised intrauterine environment on islet function.
@ast
Consequences of a compromised intrauterine environment on islet function.
@en
prefLabel
Consequences of a compromised intrauterine environment on islet function.
@ast
Consequences of a compromised intrauterine environment on islet function.
@en
P2093
P2860
P356
P1476
Consequences of a compromised intrauterine environment on islet function.
@en
P2093
Alice S Green
Paul J Rozance
Sean W Limesand
P2860
P304
P356
10.1677/JOE-09-0399
P407
P577
2010-03-11T00:00:00Z