about
Radiation-induced alterations in histone modification patterns and their potential impact on short-term radiation effectsInterplay between Ubiquitin, SUMO, and Poly(ADP-Ribose) in the Cellular Response to Genotoxic StressATM-Dependent Phosphorylation of All Three Members of the MRN Complex: From Sensor to AdaptorFine-tuning the ubiquitin code at DNA double-strand breaks: deubiquitinating enzymes at workCrosstalk between ubiquitin and other post-translational modifications on chromatin during double-strand break repairDma/RNF8 proteins are evolutionarily conserved E3 ubiquitin ligases that target septinsSam68 Is Required for DNA Damage Responses via Regulating Poly(ADP-ribosyl)ationThe versatile functions of ATM kinase.Ribosomal protein S27-like is a physiological regulator of p53 that suppresses genomic instability and tumorigenesisSynergistic interaction of Rnf8 and p53 in the protection against genomic instability and tumorigenesis.ATM signalling and cancer.PTEN interacts with histone H1 and controls chromatin condensation.Regulation of the DNA damage response by ubiquitin conjugation.TPX2 impacts acetylation of histone H4 at lysine 16: implications for DNA damage response.Ubiquitin-H2AX fusions render 53BP1 recruitment to DNA damage sites independent of RNF8 or RNF168Quantitative proteomics profiling of the poly(ADP-ribose)-related response to genotoxic stress.Small molecule inhibition of the CHFR-PARP1 interaction as novel approach to overcome intrinsic taxane resistance in cancerCHFR is important for the first wave of ubiquitination at DNA damage sitesCHFR is important for the survival of male premeiotic germ cellsSam68/KHDRBS1 is critical for colon tumorigenesis by regulating genotoxic stress-induced NF-κB activation.Regulation of the DNA damage response on male meiotic sex chromosomesThe FHA and BRCT domains recognize ADP-ribosylation during DNA damage response.CHFR: a key checkpoint component implicated in a wide range of cancers.Fusing telomeres with RNF8.Epigenetics in radiation-induced fibrosis.Spatiotemporal regulation of posttranslational modifications in the DNA damage response.Cross-talk between the H3K36me3 and H4K16ac histone epigenetic marks in DNA double-strand break repair.KMT Set7/9 affects genotoxic stress response via the Mdm2 axis.Rad18 and Rnf8 facilitate homologous recombination by two distinct mechanisms, promoting Rad51 focus formation and suppressing the toxic effect of nonhomologous end joining.DMAP1 is an essential regulator of ATM activity and function.The telomere deprotection response is functionally distinct from the genomic DNA damage response.The role of poly ADP-ribosylation in the first wave of DNA damage response.Ataxia telangiectasia syndrome: moonlighting ATM.Destabilization of linker histone H1.2 is essential for ATM activation and DNA damage repair.PARP2 mediates branched poly ADP-ribosylation in response to DNA damage
P2860
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P2860
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Chfr and RNF8 synergistically regulate ATM activation.
@ast
Chfr and RNF8 synergistically regulate ATM activation.
@en
type
label
Chfr and RNF8 synergistically regulate ATM activation.
@ast
Chfr and RNF8 synergistically regulate ATM activation.
@en
prefLabel
Chfr and RNF8 synergistically regulate ATM activation.
@ast
Chfr and RNF8 synergistically regulate ATM activation.
@en
P2093
P2860
P356
P1476
Chfr and RNF8 synergistically regulate ATM activation.
@en
P2093
David O Ferguson
Mats Ljungman
Michelle T Paulsen
Xiaochun Yu
Yibin Chen
P2860
P2888
P304
P356
10.1038/NSMB.2078
P577
2011-06-26T00:00:00Z