about
BAALC, the human member of a novel mammalian neuroectoderm gene lineage, is implicated in hematopoiesis and acute leukemiaLeucine-zipper dimerization motif encoded by the AF17 gene fused to ALL-1 (MLL) in acute leukemiaNew insight into the molecular mechanisms of MLL-associated leukemiat(11;22)(q23;q11.2) In acute myeloid leukemia of infant twins fuses MLL with hCDCrel, a cell division cycle gene in the genomic region of deletion in DiGeorge and velocardiofacial syndromesIdentification and characterization of the ARP1 gene, a target for the human acute leukemia ALL1 genePanhandle PCR strategy to amplify MLL genomic breakpoints in treatment-related leukemiasIdentification of a gene at 11q23 encoding a guanine nucleotide exchange factor: evidence for its fusion with MLL in acute myeloid leukemiaStructural Basis for WDR5 Interaction (Win) Motif Recognition in Human SET1 Family Histone MethyltransferasesMLL2, the second human homolog of the Drosophila trithorax gene, maps to 19q13.1 and is amplified in solid tumor cell linesHuman LPP gene is fused to MLL in a secondary acute leukemia with a t(3;11) (q28;q23)Functional contribution of EEN to leukemogenic transformation by MLL-EEN fusion proteinA SALL4/MLL/HOXA9 pathway in murine and human myeloid leukemogenesisDisordered epigenetic regulation in MLL-related leukemia.Advances in cancer cytogenetics.The management of patients with leukaemia: the role of cytogenetics in this molecular era.Immortalization and leukemic transformation of a myelomonocytic precursor by retrovirally transduced HRX-ENL.Tumorigenesis in mice with a fusion of the leukaemia oncogene Mll and the bacterial lacZ gene.The oncogenic capacity of HRX-ENL requires the transcriptional transactivation activity of ENL and the DNA binding motifs of HRX.Mobile elements and viral integrations prompt considerations for bacterial DNA integration as a novel carcinogenMLL-SEPTIN6 fusion recurs in novel translocation of chromosomes 3, X, and 11 in infant acute myelomonocytic leukaemia and in t(X;11) in infant acute myeloid leukaemia, and MLL genomic breakpoint in complex MLL-SEPTIN6 rearrangement is a DNA topoisomInvolvement of the ALL-1 gene in a solid tumor.Common themes in the pathogenesis of acute myeloid leukemia.Molecular mechanisms of leukemogenesis mediated by MLL fusion proteins.The structure of the human ALL-1/MLL/HRX gene.The partial tandem duplication of ALL1 (MLL) is consistently generated by Alu-mediated homologous recombination in acute myeloid leukemia.DB2: a probabilistic approach for accurate detection of tandem duplication breakpoints using paired-end reads.Genetic and molecular analysis of region 88E9;88F2 in Drosophila melanogaster, including the ear gene related to human factors involved in lineage-specific leukemias.The MLL recombinome of acute leukemias in 2013.Genetic profile of acute myeloid leukemia.Identification of intragenic deletions and duplication in the FLCN gene in Birt-Hogg-Dubé syndrome.Two decades of leukemia oncoprotein epistasis: the MLL1 paradigm for epigenetic deregulation in leukemia.Complex patterns of chromosome 11 aberrations in myeloid malignancies target CBL, MLL, DDB1 and LMO2.Panhandle PCR for cDNA: a rapid method for isolation of MLL fusion transcripts involving unknown partner genes.Malignant hematopoietic cell lines: in vitro models for the study of MLL gene alterations.Clinical relevance of mutations and gene-expression changes in adult acute myeloid leukemia with normal cytogenetics: are we ready for a prognostically prioritized molecular classification?Long-term disease-free survivors with cytogenetically normal acute myeloid leukemia and MLL partial tandem duplication: a Cancer and Leukemia Group B study.The MLL partial tandem duplication: evidence for recessive gain-of-function in acute myeloid leukemia identifies a novel patient subgroup for molecular-targeted therapyThe partial tandem duplication of ALL1 in acute myeloid leukemia with normal cytogenetics or trisomy 11 is restricted to one chromosome.The MLL partial tandem duplication in acute myeloid leukaemia.DNA hypermethylation and epigenetic silencing of the tumor suppressor gene, SLC5A8, in acute myeloid leukemia with the MLL partial tandem duplication.
P2860
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P2860
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
ALL-1 partial duplication in acute leukemia.
@ast
ALL-1 partial duplication in acute leukemia.
@en
type
label
ALL-1 partial duplication in acute leukemia.
@ast
ALL-1 partial duplication in acute leukemia.
@en
prefLabel
ALL-1 partial duplication in acute leukemia.
@ast
ALL-1 partial duplication in acute leukemia.
@en
P2093
P2860
P356
P1476
ALL-1 partial duplication in acute leukemia.
@en
P2093
M A Caligiuri
M P Strout
S A Schichman
P2860
P304
P356
10.1073/PNAS.91.13.6236
P407
P577
1994-06-01T00:00:00Z