TGF-β regulates sclerostin expression via the ECR5 enhancer. - Wikidata - wikimedia - TriplyDB

TGF-β regulates sclerostin expression via the ECR5 enhancer.

TGF-β regulates sclerostin expression via the ECR5 enhancer.

http://www.wikidata.org/entity/Q35754103

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Load regulates bone formation and Sclerostin expression through a TGFβ-dependent mechanism Developments in sclerostin biology: regulation of gene expression, mechanisms of action, and physiological functions.Beneficial effect of resveratrol on phenotypic features and activity of osteoarthritic osteoblasts.Heavy Metal Ion Regulation of Gene Expression: MECHANISMS BY WHICH LEAD INHIBITS OSTEOBLASTIC BONE-FORMING ACTIVITY THROUGH MODULATION OF THE Wnt/β-CATENIN SIGNALING PATHWAY.The Wnt Inhibitor Sclerostin Is Up-regulated by Mechanical Unloading in Osteocytes in Vitro.Lipoprotein receptor-related protein 6 is required for parathyroid hormone-induced Sost suppression.Targeted deletion of Sost distal enhancer increases bone formation and bone mass Sclerostin Antibody Treatment Improves the Bone Phenotype of Crtap(-/-) Mice, a Model of Recessive Osteogenesis Imperfecta.Role and mechanism of action of sclerostin in bone.Analysis of SOST expression using large minigenes reveals the MEF2C binding site in the evolutionarily conserved region (ECR5) enhancer mediates forskolin, but not 1,25-dihydroxyvitamin D3 or TGFβ1 responsiveness.Indirect immobilized Jagged1 suppresses cell cycle progression and induces odonto/osteogenic differentiation in human dental pulp cells.A Selective Transforming Growth Factor-β Ligand Trap Attenuates Pulmonary Hypertension.Osteocyte-Intrinsic TGF-β Signaling Regulates Bone Quality through Perilacunar/Canalicular Remodeling.SOST, an LNGFR target, inhibits the osteogenic differentiation of rat ectomesenchymal stem cells.Sclerostin: an Emerging Target for the Treatment of Cancer-Induced Bone Disease.Notch Signaling Participates in TGF-β-Induced SOST Expression Under Intermittent Compressive Stress.Mechanical Force-induced TGFB1 Increases Expression of SOST/POSTN by hPDL Cells.Advanced Glycation End Product 3 (AGE3) Increases Apoptosis and the Expression of Sclerostin by Stimulating TGF-β Expression and Secretion in Osteocyte-Like MLO-Y4-A2 Cells.Do hypoxia and L-mimosine modulate sclerostin and dickkopf-1 production in human dental pulp-derived cells? Insights from monolayer, spheroid and tooth slice cultures.Systemic Activation of Activin A Signaling Causes Chronic Kidney Disease-Mineral Bone Disorder TGF-Beta Signaling in Bone with Chronic Kidney Disease

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P2860

TGF-β regulates sclerostin expression via the ECR5 enhancer.

http://www.wikidata.org/entity/Q35754103

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

TGF-β regulates sclerostin expression via the ECR5 enhancer.

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TGF-β regulates sclerostin expression via the ECR5 enhancer.

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type

label

TGF-β regulates sclerostin expression via the ECR5 enhancer.

@ast

TGF-β regulates sclerostin expression via the ECR5 enhancer.

@en

prefLabel

TGF-β regulates sclerostin expression via the ECR5 enhancer.

@ast

TGF-β regulates sclerostin expression via the ECR5 enhancer.

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J.BONE.2011.11.016

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TGF-β regulates sclerostin expression via the ECR5 enhancer.

@en

P2093

Damian C Genetos

http://www.w3.org/2001/XMLSchema#string

Deepa Murugesh

http://www.w3.org/2001/XMLSchema#string

Gabriela G Loots

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Hansjoerg Keller

http://www.w3.org/2001/XMLSchema#string

Nicole M Collette

http://www.w3.org/2001/XMLSchema#string

Olivier Leupin

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P2860

Prostaglandin E2 signals through PTGER2 to regulate sclerostin expression

Smad proteins function as co-modulators for MEF2 transcriptional regulatory proteins

Increased bone density in sclerosteosis is due to the deficiency of a novel secreted protein (SOST)

Bone overgrowth-associated mutations in the LRP4 gene impair sclerostin facilitator function

Osteocyte control of bone formation via sclerostin, a novel BMP antagonist

SOST is a ligand for LRP5/LRP6 and a Wnt signaling inhibitor

Endogenous TGF-beta signaling suppresses maturation of osteoblastic mesenchymal cells

The Wnt antagonist secreted frizzled-related protein-1 controls osteoblast and osteocyte apoptosis

Control of the SOST bone enhancer by PTH using MEF2 transcription factors

Bone dysplasia sclerosteosis results from loss of the SOST gene product, a novel cystine knot-containing protein

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10.1016/J.BONE.2011.11.016

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3

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22155511

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3278543

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