Antibody facilitation of multiple sclerosis-like lesions in a nonhuman primate. - Wikidata - wikimedia - TriplyDB

Antibody facilitation of multiple sclerosis-like lesions in a nonhuman primate.

Antibody facilitation of multiple sclerosis-like lesions in a nonhuman primate.

http://www.wikidata.org/entity/Q35769109

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Antibodies directed against rubella virus induce demyelination in aggregating rat brain cell cultures Human nerve growth factor protects common marmosets against autoimmune encephalomyelitis by switching the balance of T helper cell type 1 and 2 cytokines within the central nervous system Amelioration of experimental autoimmune encephalomyelitis in C57BL/6 mice by photobiomodulation induced by 670 nm light Neurobehavioral development of common marmoset monkeys.Molecular characterization of antibody specificities against myelin/oligodendrocyte glycoprotein in autoimmune demyelination Myelin Oligodendrocyte Glycoprotein: Deciphering a Target in Inflammatory Demyelinating Diseases.Therapeutic strategies in multiple sclerosis. I. Immunotherapy Surprising pleiotropy of nerve growth factor in the treatment of experimental autoimmune encephalomyelitis.T- and B-cell responses to myelin oligodendrocyte glycoprotein in experimental autoimmune encephalomyelitis and multiple sclerosis.Antibodies to myelin oligodendrocyte glycoprotein in bilateral and recurrent optic neuritis.Antibodies to native myelin oligodendrocyte glycoprotein are serologic markers of early inflammation in multiple sclerosis.Elevated intrathecal myelin oligodendrocyte glycoprotein antibodies in multiple sclerosis.Limited sufficiency of antigen presentation by dendritic cells in models of central nervous system autoimmunity.Preclinical assessment of stem cell therapies for neurological diseases.Effects of early IL-17A neutralization on disease induction in a primate model of experimental autoimmune encephalomyelitis.Gene therapy in nonhuman primate models of human autoimmune disease.Myelin/oligodendrocyte glycoprotein-deficient (MOG-deficient) mice reveal lack of immune tolerance to MOG in wild-type mice.Update on the autoimmune pathology of multiple sclerosis: B-cells as disease-drivers and therapeutic targets Identification of a pathogenic antibody response to native myelin oligodendrocyte glycoprotein in multiple sclerosis.T cell subsets and their signature cytokines in autoimmune and inflammatory diseases.B cell antigen presentation is sufficient to drive neuroinflammation in an animal model of multiple sclerosis.B cells in multiple sclerosis.Oligodendrocyte apoptosis and primary demyelination induced by local TNF/p55TNF receptor signaling in the central nervous system of transgenic mice: models for multiple sclerosis with primary oligodendrogliopathy.Human monoclonal antibodies reactive to oligodendrocytes promote remyelination in a model of multiple sclerosis.Antibodies from inflamed central nervous system tissue recognize myelin oligodendrocyte glycoprotein.B cells and autoantibodies in the pathogenesis of multiple sclerosis and related inflammatory demyelinating diseases Autoantigen conformation influences both B- and T-cell responses and encephalitogenicity The Charcot Lecture | beating MS: a story of B cells, with twists and turns Self-antigen tetramers discriminate between myelin autoantibodies to native or denatured protein.IgG glycan hydrolysis by EndoS inhibits experimental autoimmune encephalomyelitis Treatment with MOG-DNA vaccines induces CD4+CD25+FoxP3+ regulatory T cells and up-regulates genes with neuroprotective functions in experimental autoimmune encephalomyelitis.Antigen-specific therapies in multiple sclerosis: going beyond proteins and peptides.A critical role for lymphotoxin in experimental allergic encephalomyelitis.Critical role of activation induced cytidine deaminase in experimental autoimmune encephalomyelitis.Pathogenic and regulatory roles for B cells in experimental autoimmune encephalomyelitis.B cells limit epitope spreading and reduce severity of EAE induced with PLP peptide in BALB/c mice Systems biology approaches for the study of multiple sclerosis.Identification of gene expression patterns crucially involved in experimental autoimmune encephalomyelitis and multiple sclerosis.MHC haplotype-dependent regulation of MOG-induced EAE in rats.MHC class II-dependent B cell APC function is required for induction of CNS autoimmunity independent of myelin-specific antibodies.

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P2860

Antibody facilitation of multiple sclerosis-like lesions in a nonhuman primate.

http://www.wikidata.org/entity/Q35769109

description

1995 nî lūn-bûn

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1995年の論文

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1995年論文

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1995年論文

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1995年論文

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1995年論文

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1995年論文

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1995年论文

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1995年论文

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1995年论文

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name

Antibody facilitation of multiple sclerosis-like lesions in a nonhuman primate.

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Antibody facilitation of multiple sclerosis-like lesions in a nonhuman primate.

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type

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Antibody facilitation of multiple sclerosis-like lesions in a nonhuman primate.

@ast

Antibody facilitation of multiple sclerosis-like lesions in a nonhuman primate.

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prefLabel

Antibody facilitation of multiple sclerosis-like lesions in a nonhuman primate.

@ast

Antibody facilitation of multiple sclerosis-like lesions in a nonhuman primate.

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Journal of Clinical Investigation

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Antibody facilitation of multiple sclerosis-like lesions in a nonhuman primate

@en

P2093

C P Genain

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M Adelman

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M B Lees

http://www.w3.org/2001/XMLSchema#string

M H Nguyen

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N L Letvin

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R L Davis

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R Pearl

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S L Hauser

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P2860

Characterization and expression of the cDNA coding for the human myelin/oligodendrocyte glycoprotein

Differential ultrastructural localization of myelin basic protein, myelin/oligodendroglial glycoprotein, and 2',3'-cyclic nucleotide 3'-phosphodiesterase in the CNS of adult rats

Myelin/oligodendrocyte glycoprotein is a unique member of the immunoglobulin superfamily.

Severe periphlebitis, peripheral retinal ischemia, and preretinal neovascularization in patients with multiple sclerosis.

Reactivity to myelin antigens in multiple sclerosis. Peripheral blood lymphocytes respond predominantly to myelin oligodendrocyte glycoprotein.

In healthy primates, circulating autoreactive T cells mediate autoimmune disease.

Prevention of autoimmune demyelination in non-human primates by a cAMP-specific phosphodiesterase inhibitor.

The antidepressant rolipram suppresses cytokine production and prevents autoimmune encephalomyelitis.

Experimental allergic encephalomyelitis in cynomolgus monkeys. Quantitation of T cell responses in peripheral blood.

Transfer of allergic encephalomyelitis in rats by means of lymph node cells.

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2966-2974

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10.1172/JCI118368

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6

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96

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Christopher Linington

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1995-12-01T00:00:00Z

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8675668

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multiple sclerosis

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186008

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