A critical role for lymphotoxin in experimental allergic encephalomyelitis.
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The intriguing biology of the tumour necrosis factor/tumour necrosis factor receptor superfamily: players, rules and the gamesAnti-cytokine therapy in chronic destructive arthritisRole of Tumor Necrosis Factor Superfamily in Neuroinflammation and AutoimmunityICOS co-stimulatory receptor is essential for T-cell activation and functionInvolvement of tissue plasminogen activator in onset and effector phases of experimental allergic encephalomyelitisChallenging cytokine redundancy: inflammatory cell movement and clinical course of experimental autoimmune encephalomyelitis are normal in lymphotoxin-deficient, but not tumor necrosis factor-deficient, miceThe B7 family member B7-H3 preferentially down-regulates T helper type 1-mediated immune responsesAbsence of monocyte chemoattractant protein 1 in mice leads to decreased local macrophage recruitment and antigen-specific T helper cell type 1 immune response in experimental autoimmune encephalomyelitisCell-selective knockout and 3D confocal image analysis reveals separate roles for astrocyte-and endothelial-derived CCL2 in neuroinflammation.Bringing CLARITY to gray matter atrophy.On the role of tumor necrosis factor and receptors in models of multiorgan failure, rheumatoid arthritis, multiple sclerosis and inflammatory bowel disease.Unlinking tumor necrosis factor biology from the major histocompatibility complex: lessons from human genetics and animal modelsThe role of TNFalpha and lymphotoxin in demyelinating disease.SHP-1 deficiency and increased inflammatory gene expression in PBMCs of multiple sclerosis patients.Tumour necrosis factor and anti-tumour necrosis factor approach to inflammatory demyelinating diseases of the central nervous system.Role of lymphotoxin alpha in T-cell responses during an acute viral infection.Glial expression of tumor necrosis factor in transgenic animals: how do these models reflect the "normal situation"?Multiple roles for tumor necrosis factor-alpha and lymphotoxin alpha/beta in immunity and autoimmunity.Memory B cells from a subset of treatment-naïve relapsing-remitting multiple sclerosis patients elicit CD4(+) T-cell proliferation and IFN-γ production in response to myelin basic protein and myelin oligodendrocyte glycoprotein.CD4 T cells: fates, functions, and faultsAntagonistic TNF receptor one-specific antibody (ATROSAB): receptor binding and in vitro bioactivityA tumor necrosis factor receptor 1-dependent conversation between central nervous system-specific T cells and the central nervous system is required for inflammatory infiltration of the spinal cord.The effect of glatiramer acetate therapy on functional properties of B cells from patients with relapsing-remitting multiple sclerosis.Depletion of CD4(+)CD25(+) T cells exacerbates experimental autoimmune encephalomyelitis induced by mouse, but not rat, antigensA role for surface lymphotoxin in experimental autoimmune encephalomyelitis independent of LIGHT.Experimental autoimmune encephalomyelitis (EAE) in CCR2(-/-) mice: susceptibility in multiple strainsOligodendrocyte apoptosis and primary demyelination induced by local TNF/p55TNF receptor signaling in the central nervous system of transgenic mice: models for multiple sclerosis with primary oligodendrogliopathy.Cortical atrophy in experimental autoimmune encephalomyelitis: in vivo imagingEstrogen treatment prevents gray matter atrophy in experimental autoimmune encephalomyelitis.Essential role of lymph nodes in contact hypersensitivity revealed in lymphotoxin-alpha-deficient mice.The CCL2 synthesis inhibitor bindarit targets cells of the neurovascular unit, and suppresses experimental autoimmune encephalomyelitis.Exosomes from HIV-1-infected Cells Stimulate Production of Pro-inflammatory Cytokines through Trans-activating Response (TAR) RNANovel 3D analysis of Claudin-5 reveals significant endothelial heterogeneity among CNS microvessels.Systems-based medicine approaches to understand and treat complex diseases. The example of multiple sclerosis.Glial reactions and degeneration of myelinated processes in spinal cord gray matter in chronic experimental autoimmune encephalomyelitis.Newborn screening for autism: in search of candidate biomarkers.Enhancing the ability of experimental autoimmune encephalomyelitis to serve as a more rigorous model of multiple sclerosis through refinement of the experimental designRegulation of Th17 cell differentiation and EAE induction by MAP3K NIK.Purkinje cell loss in experimental autoimmune encephalomyelitis.Reactive astrocytes form scar-like perivascular barriers to leukocytes during adaptive immune inflammation of the CNS.
P2860
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P2860
A critical role for lymphotoxin in experimental allergic encephalomyelitis.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
1997年學術文章
@zh
1997年學術文章
@zh-hant
name
A critical role for lymphotoxin in experimental allergic encephalomyelitis.
@ast
A critical role for lymphotoxin in experimental allergic encephalomyelitis.
@en
type
label
A critical role for lymphotoxin in experimental allergic encephalomyelitis.
@ast
A critical role for lymphotoxin in experimental allergic encephalomyelitis.
@en
prefLabel
A critical role for lymphotoxin in experimental allergic encephalomyelitis.
@ast
A critical role for lymphotoxin in experimental allergic encephalomyelitis.
@en
P2093
P2860
P356
P1476
A critical role for lymphotoxin in experimental allergic encephalomyelitis.
@en
P2093
C M Bergman
N H Ruddle
P Hjelmström
P2860
P304
P356
10.1084/JEM.186.8.1233
P407
P577
1997-10-01T00:00:00Z